Posttranslational mechanisms contribute to the suppression of specific cyclin:CDK complexes by all-trans retinoic acid in human bronchial epithelial cells

Cancer Research

Volumn 59, Issue 15, 1999, Pages 3838-3844

  Sueoka, Naoko ^a   Lee, Ho Young ^a   Walsh, Garrett L ^a   Hong, Waun Ki ^a   Kurie, Jonathan M ^a  

^a UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACTIN; CYCLIN D1; CYCLIN DEPENDENT KINASE; CYCLIN DEPENDENT KINASE 2; CYCLIN DEPENDENT KINASE 4; CYCLIN DEPENDENT KINASE INHIBITOR; CYCLIN E; RETINOIC ACID; TRANSCRIPTION FACTOR E2F; UNCLASSIFIED DRUG;

ARTICLE; BRONCHUS CANCER; BRONCHUS MUCOSA; CANCER INHIBITION; CONTROLLED STUDY; ENZYME BINDING; ENZYME SPECIFICITY; GROWTH INHIBITION; HUMAN; HUMAN CELL; HUMAN TISSUE; PRIORITY JOURNAL; PROTEIN PROCESSING; TUMOR SUPPRESSOR GENE;

ANTICARCINOGENIC AGENTS; BRONCHI; CARCINOMA, NON-SMALL-CELL LUNG; CELL CYCLE PROTEINS; CYCLIN D1; CYCLIN E; CYCLIN-DEPENDENT KINASE INHIBITOR P27; CYCLIN-DEPENDENT KINASES; EPITHELIAL CELLS; GENES, REPORTER; HUMANS; LUNG NEOPLASMS; MICROTUBULE-ASSOCIATED PROTEINS; PHOSPHORYLATION; PROTEIN PROCESSING, POST-TRANSLATIONAL; RETINOBLASTOMA PROTEIN; SIGNAL TRANSDUCTION; TRETINOIN; TUMOR CELLS, CULTURED; TUMOR SUPPRESSOR PROTEINS;

EID: 0033179266     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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