-
1
-
-
0028872649
-
Specificity of receptor tyrosine kinase signalling: Transient versus sustained extracellular signal-regulated kinase activation
-
Marshall, C. J. Specificity of receptor tyrosine kinase signalling: transient versus sustained extracellular signal-regulated kinase activation. Cell, 80: 179-185, 1995.
-
(1995)
Cell
, vol.80
, pp. 179-185
-
-
Marshall, C.J.1
-
2
-
-
0026483357
-
Function of Ras as a molecular switch in signal transduction
-
Satoh, T., Nakafuku, M., and Kaziro, Y. Function of Ras as a molecular switch in signal transduction. J. Biol. Chem., 267: 24149-24152, 1992.
-
(1992)
J. Biol. Chem.
, vol.267
, pp. 24149-24152
-
-
Satoh, T.1
Nakafuku, M.2
Kaziro, Y.3
-
3
-
-
0021970413
-
Requirement for Ras proto-oncogene function during serum-stimulated growth of NIH 3T3 cells
-
Mulcahy, L. S., Smith, M. R., and Stacey, D. W. Requirement for Ras proto-oncogene function during serum-stimulated growth of NIH 3T3 cells. Nature (Lond.), 313: 241-243, 1985.
-
(1985)
Nature (Lond.)
, vol.313
, pp. 241-243
-
-
Mulcahy, L.S.1
Smith, M.R.2
Stacey, D.W.3
-
4
-
-
0026336517
-
Dominant inhibitory Ras mutants demonstrate the requirement for Ras activity in the action of tyrosine kinase oncogenes
-
Stacey, D. W., Roudebush, M., Day, R., Mosser, S. D., Gibbs, J. B., and Feig, L. A. Dominant inhibitory Ras mutants demonstrate the requirement for Ras activity in the action of tyrosine kinase oncogenes. Oncogene, 6: 2297-2304, 1991.
-
(1991)
Oncogene
, vol.6
, pp. 2297-2304
-
-
Stacey, D.W.1
Roudebush, M.2
Day, R.3
Mosser, S.D.4
Gibbs, J.B.5
Feig, L.A.6
-
5
-
-
0026578026
-
Growth factor signaling: Where is the specificity?
-
Chao, M. V. Growth factor signaling: where is the specificity? Cell, 68: 995-997, 1992.
-
(1992)
Cell
, vol.68
, pp. 995-997
-
-
Chao, M.V.1
-
6
-
-
0030944985
-
Oncogenic Ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a
-
Serrano, M., Lin, A. W., McCurrach, M. E., Beach, D., and Lowe, S. W. Oncogenic Ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a. Cell, 88: 593-602, 1997.
-
(1997)
Cell
, vol.88
, pp. 593-602
-
-
Serrano, M.1
Lin, A.W.2
McCurrach, M.E.3
Beach, D.4
Lowe, S.W.5
-
7
-
-
0027170451
-
Mitogen-activated protein kinase p42mapk and p44mapk are required for fibroblast proliferation
-
Pages, G., Lenormand, P., L'Allemain, G., Chambard, J-C., Meloche, S., and Pouyssegur, J. Mitogen-activated protein kinase p42mapk and p44mapk are required for fibroblast proliferation. Proc. Natl. Acad. Sci. USA, 90: 8319-8323, 1993.
-
(1993)
Proc. Natl. Acad. Sci. USA
, vol.90
, pp. 8319-8323
-
-
Pages, G.1
Lenormand, P.2
L'Allemain, G.3
Chambard, J.-C.4
Meloche, S.5
Pouyssegur, J.6
-
8
-
-
0030933277
-
Oncoprotein networks
-
Hunter, T. Oncoprotein networks. Cell, 88: 333-346, 1997.
-
(1997)
Cell
, vol.88
, pp. 333-346
-
-
Hunter, T.1
-
9
-
-
0028228616
-
Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH3T3 cells
-
Cowley, S., Paterson, H., Kemp, P., and Marshall, C. J. Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH3T3 cells. Cell, 77: 841-852, 1994.
-
(1994)
Cell
, vol.77
, pp. 841-852
-
-
Cowley, S.1
Paterson, H.2
Kemp, P.3
Marshall, C.J.4
-
10
-
-
0026534571
-
Ras is essential for nerve growth factor- and phorbol ester-induced tyrosine phosphorylation of MAP kinases
-
Thomas, S. M., DeMarco, M., D'Arcangelo, G., Halegoua, S., and Brugge, J. S. Ras is essential for nerve growth factor- and phorbol ester-induced tyrosine phosphorylation of MAP kinases. Cell, 68: 1031-1040, 1992.
-
(1992)
Cell
, vol.68
, pp. 1031-1040
-
-
Thomas, S.M.1
DeMarco, M.2
D'Arcangelo, G.3
Halegoua, S.4
Brugge, J.S.5
-
11
-
-
0026523559
-
Ras mediates nerve growth factor receptor modulation of three signal-transducing protein kinases: MAP kinase, Raf-1 and RSK
-
Wood, K. W., Samecki, C., Roberts, T. M., and Blenis, J. Ras mediates nerve growth factor receptor modulation of three signal-transducing protein kinases: MAP kinase, Raf-1 and RSK. Cell, 68: 1041-1050, 1992.
-
(1992)
Cell
, vol.68
, pp. 1041-1050
-
-
Wood, K.W.1
Samecki, C.2
Roberts, T.M.3
Blenis, J.4
-
12
-
-
0029892754
-
Fibroblast growth factor-2 suppression of tumor necrosis factor α-mediated apoptosis requires ras and the activation of mitogen-activated protein kinase
-
Gardner, A. M., and Johnson, G. L. Fibroblast growth factor-2 suppression of tumor necrosis factor α-mediated apoptosis requires ras and the activation of mitogen-activated protein kinase. J. Biol. Chem., 271: 14560-14566, 1996.
-
(1996)
J. Biol. Chem.
, vol.271
, pp. 14560-14566
-
-
Gardner, A.M.1
Johnson, G.L.2
-
13
-
-
0026611053
-
Involvement of p21 ras in activation of extracellular signal-regulated kinase 2
-
de Vries-Smits, A. M. M., Burgering, B. M. T., Leevers, S. J., Marshall, C. J., and Bos, J. L. Involvement of p21 ras in activation of extracellular signal-regulated kinase 2. Nature (Lond.), 357: 602-604, 1992.
-
(1992)
Nature (Lond.)
, vol.357
, pp. 602-604
-
-
De Vries-Smits, A.M.M.1
Burgering, B.M.T.2
Leevers, S.J.3
Marshall, C.J.4
Bos, J.L.5
-
14
-
-
0027489748
-
Epidermal growth factor induces phosphorylation of extracellular signal-regulated kinase 2 via multiple pathways
-
Burgering, B. M. T., de Vries-Smits, A. M. M., Medema, R. H., van Weeren, P. C., Tertoolen, L. G. J., and Bos, J. L. Epidermal growth factor induces phosphorylation of extracellular signal-regulated kinase 2 via multiple pathways. Mol. Cell. Biol., 13: 7248-7256, 1993.
-
(1993)
Mol. Cell. Biol.
, vol.13
, pp. 7248-7256
-
-
Burgering, B.M.T.1
De Vries-Smits, A.M.M.2
Medema, R.H.3
Van Weeren, P.C.4
Tertoolen, L.G.J.5
Bos, J.L.6
-
15
-
-
0029788015
-
Altered transforming growth factor β signaling in epithelial cells when Ras activation is blocked
-
Hartsough, M. T., Frey, R. S., Zipfel, P. A., Buard, A., Cook, S. J., McCormick, F., and Mulder, K. M. Altered transforming growth factor β signaling in epithelial cells when Ras activation is blocked. J. Biol. Chem., 271: 22368-22375, 1996.
-
(1996)
J. Biol. Chem.
, vol.271
, pp. 22368-22375
-
-
Hartsough, M.T.1
Frey, R.S.2
Zipfel, P.A.3
Buard, A.4
Cook, S.J.5
McCormick, F.6
Mulder, K.M.7
-
16
-
-
0029890896
-
Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformation
-
Khosravi-Far, R., White, M. A., Westwick, J. K., Solski, P. A., Chrzanowska-Wodnicka, M., Van Aelst, L., Wigler, M. H., and Der, C. J. Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformation. Mol. Cell. Biol., 16: 3923-3933, 1996.
-
(1996)
Mol. Cell. Biol.
, vol.16
, pp. 3923-3933
-
-
Khosravi-Far, R.1
White, M.A.2
Westwick, J.K.3
Solski, P.A.4
Chrzanowska-Wodnicka, M.5
Van Aelst, L.6
Wigler, M.H.7
Der, C.J.8
-
17
-
-
0030022174
-
Ral-GTPases mediate a distinct downstream signaling pathway from Ras that facilitates cellular transformation
-
Urano, T., Emkey, R., and Feig, L. A. Ral-GTPases mediate a distinct downstream signaling pathway from Ras that facilitates cellular transformation. EMBO J., 15: 810-816, 1996.
-
(1996)
EMBO J.
, vol.15
, pp. 810-816
-
-
Urano, T.1
Emkey, R.2
Feig, L.A.3
-
18
-
-
0028074316
-
Phosphatidylinositol-3-OH kinase as a direct target of Ras
-
Rodriguez-Viciana, P., Warne, P. H., Dhand, R., Vanhaesebroeck, B., Gout, I., Fry, M., Waterfield, M. D., and Downward, J. Phosphatidylinositol-3-OH kinase as a direct target of Ras. Nature (Lond.), 370: 527-532, 1994.
-
(1994)
Nature (Lond.)
, vol.370
, pp. 527-532
-
-
Rodriguez-Viciana, P.1
Warne, P.H.2
Dhand, R.3
Vanhaesebroeck, B.4
Gout, I.5
Fry, M.6
Waterfield, M.D.7
Downward, J.8
-
19
-
-
0032541650
-
Increasing complexity of Ras signaling
-
Campbell, S. L., Khosravi-Far, R., Rossman, K. L., Clark, G. J., and Der, C. J. Increasing complexity of Ras signaling. Oncogene, 17: 1395-1413, 1998.
-
(1998)
Oncogene
, vol.17
, pp. 1395-1413
-
-
Campbell, S.L.1
Khosravi-Far, R.2
Rossman, K.L.3
Clark, G.J.4
Der, C.J.5
-
20
-
-
0028982948
-
Distinct pathways of Gi- and Gq-mediated mitogen-activated protein kinase activation
-
Hawes, B. E., van Biesen, T., Koch, W. J., Luttrell, L. M., and Lefkowitz, R. J. Distinct pathways of Gi- and Gq-mediated mitogen-activated protein kinase activation. J. Biol. Chem., 270: 17148-17153, 1995.
-
(1995)
J. Biol. Chem.
, vol.270
, pp. 17148-17153
-
-
Hawes, B.E.1
Van Biesen, T.2
Koch, W.J.3
Luttrell, L.M.4
Lefkowitz, R.J.5
-
21
-
-
0029789967
-
Protein kinase C delta activates the MEK-ERK pathway in a manner independent of Ras and dependent on Raf
-
Ueda, Y., Hirai, S-I., Osada, S-I., Suzuki, A., Mizuno, K., and Ohno, S. Protein kinase C delta activates the MEK-ERK pathway in a manner independent of Ras and dependent on Raf. J. Biol. Chem., 271; 23512-23519, 1996.
-
(1996)
J. Biol. Chem.
, vol.271
, pp. 23512-23519
-
-
Ueda, Y.1
Hirai, S.-I.2
Osada, S.-I.3
Suzuki, A.4
Mizuno, K.5
Ohno, S.6
-
22
-
-
0027359827
-
WAF1, a potential mediator of p53 tumor suppression
-
El-Deiry, W. S., Tokino, T., Velculescu, V. E., Levy, D. B., Parsons, R., Trent, J. M., Lin, D., Mercer, W. E., Kinzler, K. W., and Vogelstein, B. WAF1, a potential mediator of p53 tumor suppression. Cell, 75: 817-825, 1993.
-
(1993)
Cell
, vol.75
, pp. 817-825
-
-
El-Deiry, W.S.1
Tokino, T.2
Velculescu, V.E.3
Levy, D.B.4
Parsons, R.5
Trent, J.M.6
Lin, D.7
Mercer, W.E.8
Kinzler, K.W.9
Vogelstein, B.10
-
23
-
-
0028986639
-
p53-independent expression of p21Cip1 in muscle and other terminally differentiating cells
-
Washington DC
-
Parker, S. B., Elchele, G., Zhang, P., Rawls, A., Sands, A. T., Bradley, A., Olson, E. N., Harper, J. W., and Elledge, S. J. p53-independent expression of p21Cip1 in muscle and other terminally differentiating cells. Science (Washington DC), 267: 1024-1027, 1995.
-
(1995)
Science
, vol.267
, pp. 1024-1027
-
-
Parker, S.B.1
Elchele, G.2
Zhang, P.3
Rawls, A.4
Sands, A.T.5
Bradley, A.6
Olson, E.N.7
Harper, J.W.8
Elledge, S.J.9
-
24
-
-
0027496935
-
The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases
-
Harper, J. W., Adami, G. R., Wei, N., Keyomarsi, K., and Elledge, S. J. The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases. Cell, 75: 805-816, 1993.
-
(1993)
Cell
, vol.75
, pp. 805-816
-
-
Harper, J.W.1
Adami, G.R.2
Wei, N.3
Keyomarsi, K.4
Elledge, S.J.5
-
25
-
-
0030973878
-
New functional activities for the p21 family of CDK inhibitors
-
LaBaer, J., Garrett, M. D., Stevenson, L. F., Slingerland, J. M., Sandhu, C., Chou, H. S., Fattaey, A., and Harlow, E. New functional activities for the p21 family of CDK inhibitors. Genes Dev., 11: 847-862, 1997.
-
(1997)
Genes Dev.
, vol.11
, pp. 847-862
-
-
LaBaer, J.1
Garrett, M.D.2
Stevenson, L.F.3
Slingerland, J.M.4
Sandhu, C.5
Chou, H.S.6
Fattaey, A.7
Harlow, E.8
-
26
-
-
0033559264
-
The p21Cip1 and p27Kip1 CDK "inhibitors" are essential activators of cyclin D-dependent kinases in murine fibroblasts
-
Cheng, M., Olivier, P., Diehl, J. A., Fera, M., Roussel, M. F., Roberts, J. M., and Sherr, C. J. The p21Cip1 and p27Kip1 CDK "inhibitors" are essential activators of cyclin D-dependent kinases in murine fibroblasts. EMBO J., 18: 1571-1583, 1999.
-
(1999)
EMBO J.
, vol.18
, pp. 1571-1583
-
-
Cheng, M.1
Olivier, P.2
Diehl, J.A.3
Fera, M.4
Roussel, M.F.5
Roberts, J.M.6
Sherr, C.J.7
-
27
-
-
0028963841
-
p53-dependent and independent expression of p21 during cell growth, differentiation, and DNA damage
-
Macleod, K. F., Sherry, N., Hannon, G., Beach, D., Tokino, T., Kinzler, K., Vogelstein, B., and Jacks, T. p53-dependent and independent expression of p21 during cell growth, differentiation, and DNA damage. Genes Dev., 9: 935-944, 1995.
-
(1995)
Genes Dev.
, vol.9
, pp. 935-944
-
-
Macleod, K.F.1
Sherry, N.2
Hannon, G.3
Beach, D.4
Tokino, T.5
Kinzler, K.6
Vogelstein, B.7
Jacks, T.8
-
28
-
-
0030758522
-
Nerve growth factor induces transcription of the p21WAF1/CIP1 and cyclin D1 genes in PC12 cells by activating the Sp1 transcription factor
-
Yan, G-Z., and Ziff, E. B. Nerve growth factor induces transcription of the p21WAF1/CIP1 and cyclin D1 genes in PC12 cells by activating the Sp1 transcription factor. J. Neurosci., 17: 6122-6132, 1997.
-
(1997)
J. Neurosci.
, vol.17
, pp. 6122-6132
-
-
Yan, G.-Z.1
Ziff, E.B.2
-
29
-
-
0031028381
-
Cell cycle arrest mediated by the MEK/mitogen-activated protein kinase pathway. Proc
-
Pumiglia, K. M., and Decker, S. J. Cell cycle arrest mediated by the MEK/mitogen-activated protein kinase pathway. Proc. Natl. Acad. Sci. USA, 94: 448-452, 1997.
-
(1997)
Natl. Acad. Sci. USA
, vol.94
, pp. 448-452
-
-
Pumiglia, K.M.1
Decker, S.J.2
-
30
-
-
0028980376
-
Prolonged induction of p21Cip1/Waf1/CDK2/PCNA complex by epidermal growth factor receptor activation mediates ligand-induced A431 cell growth inhibition
-
Fan, Z., Lu, Y., Wu, X., DeBlasio, A., Koff, A., and Mendelsohn, J. Prolonged induction of p21Cip1/Waf1/CDK2/PCNA complex by epidermal growth factor receptor activation mediates ligand-induced A431 cell growth inhibition. J. Cell Biol., 131: 235-242, 1995.
-
(1995)
J. Cell Biol.
, vol.131
, pp. 235-242
-
-
Fan, Z.1
Lu, Y.2
Wu, X.3
DeBlasio, A.4
Koff, A.5
Mendelsohn, J.6
-
31
-
-
0028829465
-
Functional analysis of the transforming growth factor β responsive elements in the WAF1/Cip1/p21 promoter
-
Datto, M. B., Yu, Y., and Wang, X-F. Functional analysis of the transforming growth factor β responsive elements in the WAF1/Cip1/p21 promoter. J. Biol. Chem., 270: 28623-28628, 1995.
-
(1995)
J. Biol. Chem.
, vol.270
, pp. 28623-28628
-
-
Datto, M.B.1
Yu, Y.2
Wang, X.-F.3
-
32
-
-
0028236846
-
Induction of WAF1/CIP1 by a p53-independent pathway
-
Michieli, P., Chedid, M., Lin, D., Pierce, J. H., Mercer, W. E., and Givol, D. Induction of WAF1/CIP1 by a p53-independent pathway. Cancer Res., 54: 3391-3395, 1994.
-
(1994)
Cancer Res.
, vol.54
, pp. 3391-3395
-
-
Michieli, P.1
Chedid, M.2
Lin, D.3
Pierce, J.H.4
Mercer, W.E.5
Givol, D.6
-
33
-
-
0032554765
-
FGF signaling activates STAT1 and p21 and inhibits the estrogen response and proliferation of MCF-7 cells
-
Johnson, M. R., Valentine, C., Basilico, C., and Mansukhani, A. FGF signaling activates STAT1 and p21 and inhibits the estrogen response and proliferation of MCF-7 cells. Oncogene, 16: 2647-2656, 1998.
-
(1998)
Oncogene
, vol.16
, pp. 2647-2656
-
-
Johnson, M.R.1
Valentine, C.2
Basilico, C.3
Mansukhani, A.4
-
34
-
-
0030701842
-
The cyclin-dependent kinase inhibitor p21Cip1 mediates the growth inhibitory effect of phorbol esters in human venous endothelial cells
-
Zezula, J., Sexl, V., Hutter, C., Karel, A., Schütz, W., and Freissmuth, M. The cyclin-dependent kinase inhibitor p21Cip1 mediates the growth inhibitory effect of phorbol esters in human venous endothelial cells. J. Biol. Chem., 272: 29967-29974, 1997.
-
(1997)
J. Biol. Chem.
, vol.272
, pp. 29967-29974
-
-
Zezula, J.1
Sexl, V.2
Hutter, C.3
Karel, A.4
Schütz, W.5
Freissmuth, M.6
-
35
-
-
0033523778
-
Ras induces p21Cip1/Waf1 cyclin kinase inhibitor transcriptionally through Sp1-binding sites
-
in press
-
Kivinen, L., Tsubari, M., Haapajärvi, T., Datto, M. B., Wang, X-F., and Laiho, M. Ras induces p21Cip1/Waf1 cyclin kinase inhibitor transcriptionally through Sp1-binding sites. Oncogene, in press, 1999.
-
(1999)
Oncogene
-
-
Kivinen, L.1
Tsubari, M.2
Haapajärvi, T.3
Datto, M.B.4
Wang, X.-F.5
Laiho, M.6
-
36
-
-
0030835430
-
Raf-induced proliferation or cell cycle arrest is determined by the level of Raf-activity with arrest mediated by p21Cip1
-
Woods, D., Parry, D., Cherwinski, H., Bosch, E., Lees, E., and McMahon, M. Raf-induced proliferation or cell cycle arrest is determined by the level of Raf-activity with arrest mediated by p21Cip1. Mol. Cell. Biol., 17: 5598-5611, 1997.
-
(1997)
Mol. Cell. Biol.
, vol.17
, pp. 5598-5611
-
-
Woods, D.1
Parry, D.2
Cherwinski, H.3
Bosch, E.4
Lees, E.5
McMahon, M.6
-
37
-
-
0030871667
-
High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1
-
Sewing, A., Wiseman, B., Lloyd, A., and Land, H. High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1. Mol. Cell. Biol., 17: 5588-5597, 1997.
-
(1997)
Mol. Cell. Biol.
, vol.17
, pp. 5588-5597
-
-
Sewing, A.1
Wiseman, B.2
Lloyd, A.3
Land, H.4
-
38
-
-
0029664395
-
Regulation of p21WAF1/CIP1 expression through mitogen-activated protein kinase signaling pathway
-
Liu, Y., Martindale, J. L., Gorospe, M., and Holbrook, N. J. Regulation of p21WAF1/CIP1 expression through mitogen-activated protein kinase signaling pathway. Cancer Res., 56: 31-35, 1996.
-
(1996)
Cancer Res.
, vol.56
, pp. 31-35
-
-
Liu, Y.1
Martindale, J.L.2
Gorospe, M.3
Holbrook, N.J.4
-
39
-
-
0032537819
-
Signals from Ras and Rho GTPases interact to regulate expression of p21Waf1/Cip1
-
Olson, M. F., Paterson, H. F., and Marshall, C. J. Signals from Ras and Rho GTPases interact to regulate expression of p21Waf1/Cip1. Nature (Lond.), 394: 295-299, 1998.
-
(1998)
Nature (Lond.)
, vol.394
, pp. 295-299
-
-
Olson, M.F.1
Paterson, H.F.2
Marshall, C.J.3
-
40
-
-
0028176483
-
Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals
-
Polyak, K., Lee, M. H., Erdjument-Bromage, H., Koff, A., Roberts, J. M., Tempst, P., and Massagué, J. Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals. Cell, 78: 59-66, 1994.
-
(1994)
Cell
, vol.78
, pp. 59-66
-
-
Polyak, K.1
Lee, M.H.2
Erdjument-Bromage, H.3
Koff, A.4
Roberts, J.M.5
Tempst, P.6
Massagué, J.7
-
41
-
-
0032499802
-
Blockade in TGF β 3 upregulation of p27Kip1 and p21Cip1 by expression of RasN17 in epithelial cells
-
Yue, J., Buard, A., and Mulder, K. M. Blockade in TGF β 3 upregulation of p27Kip1 and p21Cip1 by expression of RasN17 in epithelial cells. Oncogene, 17: 47-55, 1998.
-
(1998)
Oncogene
, vol.17
, pp. 47-55
-
-
Yue, J.1
Buard, A.2
Mulder, K.M.3
-
42
-
-
0030692016
-
Functional components of fibroblast growth factor (FGF) signal transduction in pituitary cells
-
Schweppe, R. E., Frazer-Abel, A. A., Gutierrez-Hartmann, A., and Bradford, A. P. Functional components of fibroblast growth factor (FGF) signal transduction in pituitary cells. J. Biol. Chem., 272: 30852-30859, 1997.
-
(1997)
J. Biol. Chem.
, vol.272
, pp. 30852-30859
-
-
Schweppe, R.E.1
Frazer-Abel, A.A.2
Gutierrez-Hartmann, A.3
Bradford, A.P.4
-
43
-
-
0029790656
-
FGF and stress regulate CREB and ATF-1 via a pathway involving p38 MAP kinase and MAPKAP kinase-2
-
Tan, Y., Rouse, J., Zhang, A., Cariati, S., Cohen, P., and Comb, M. J. FGF and stress regulate CREB and ATF-1 via a pathway involving p38 MAP kinase and MAPKAP kinase-2. EMBO J., 15: 4629-4642, 1996.
-
(1996)
EMBO J.
, vol.15
, pp. 4629-4642
-
-
Tan, Y.1
Rouse, J.2
Zhang, A.3
Cariati, S.4
Cohen, P.5
Comb, M.J.6
-
44
-
-
0028239047
-
Independent effects of platelet-derived growth factor isoforms on mitogen-activated protein kinase activation and mitogenesis in human dermal fibroblasts
-
Lubinus, M., Meier, K. E., Smith, E. A., Gause, K. C., LeRoy, E. C., and Trojanowska, M. Independent effects of platelet-derived growth factor isoforms on mitogen-activated protein kinase activation and mitogenesis in human dermal fibroblasts. J. Biol. Chem., 269: 9822-9825, 1994.
-
(1994)
J. Biol. Chem.
, vol.269
, pp. 9822-9825
-
-
Lubinus, M.1
Meier, K.E.2
Smith, E.A.3
Gause, K.C.4
LeRoy, E.C.5
Trojanowska, M.6
-
45
-
-
0029153609
-
Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-β
-
Reynisdottir, I., Polyak, K., lavarone, A., and Massagué, J. Kip/Cip and Ink4 Cdk inhibitors cooperate to induce cell cycle arrest in response to TGF-β. Genes Dev., 9: 1831-1845, 1995.
-
(1995)
Genes Dev.
, vol.9
, pp. 1831-1845
-
-
Reynisdottir, I.1
Polyak, K.2
Lavarone, A.3
Massagué, J.4
-
46
-
-
0028884033
-
PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo
-
Alessi, D. R., Cuenda, A., Cohen, P., Dudley, D. T., and Saltiel, A. R. PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo. J. Biol. Chem., 270: 27489-27494, 1995.
-
(1995)
J. Biol. Chem.
, vol.270
, pp. 27489-27494
-
-
Alessi, D.R.1
Cuenda, A.2
Cohen, P.3
Dudley, D.T.4
Saltiel, A.R.5
-
47
-
-
0031058607
-
Transforming growth factor-β induces activation of Ras, Raf-1, MEK and MAPK in rat hepatic stellate cells
-
Reimann, T., Hempel, U., Krautwald, S., Axmann, A., Scheibe, R., Seidel, D., and Wenzel, K-W. Transforming growth factor-β induces activation of Ras, Raf-1, MEK and MAPK in rat hepatic stellate cells. FEBS Lett., 403: 57-60, 1997.
-
(1997)
FEBS Lett.
, vol.403
, pp. 57-60
-
-
Reimann, T.1
Hempel, U.2
Krautwald, S.3
Axmann, A.4
Scheibe, R.5
Seidel, D.6
Wenzel, K.-W.7
-
48
-
-
0029616117
-
Cell type-specific modulation of cell growth by transforming growth factor β1 does not correlate with mitogen-activated protein kinase activation
-
Chatani, Y., Tanimura, S., Miyoshi, N., Hattori, A., Sato, M., and Kohno, M. Cell type-specific modulation of cell growth by transforming growth factor β1 does not correlate with mitogen-activated protein kinase activation. J. Biol. Chem., 270: 30686-30692, 1995.
-
(1995)
J. Biol. Chem.
, vol.270
, pp. 30686-30692
-
-
Chatani, Y.1
Tanimura, S.2
Miyoshi, N.3
Hattori, A.4
Sato, M.5
Kohno, M.6
-
49
-
-
0026612306
-
Activation of p21 ras by transforming growth factor β in epithelial cells
-
Mulder, K. M., and Morris, S. L. Activation of p21 ras by transforming growth factor β in epithelial cells. J. Biol. Chem., 267: 5029-5031, 1992.
-
(1992)
J. Biol. Chem.
, vol.267
, pp. 5029-5031
-
-
Mulder, K.M.1
Morris, S.L.2
-
50
-
-
0028937610
-
Transforming growth factor β activation of p44mapk in proliferating cultures of epithelial cells
-
Hartsough, M. T., and Mulder, K. M. Transforming growth factor β activation of p44mapk in proliferating cultures of epithelial cells. J. Biol. Chem., 270: 7117-7124, 1995.
-
(1995)
J. Biol. Chem.
, vol.270
, pp. 7117-7124
-
-
Hartsough, M.T.1
Mulder, K.M.2
-
51
-
-
0031909865
-
p53-dependent elevation of p21Waf1 expression by UV light is mediated through mRNA stabilization and involves a vanadate-sensitive regulatory system
-
Gorospe, M., Wang, X., and Holbrook, N. J. p53-dependent elevation of p21Waf1 expression by UV light is mediated through mRNA stabilization and involves a vanadate-sensitive regulatory system. Mol. Cell. Biol., 18: 1400-1407, 1998.
-
(1998)
Mol. Cell. Biol.
, vol.18
, pp. 1400-1407
-
-
Gorospe, M.1
Wang, X.2
Holbrook, N.J.3
-
52
-
-
0029658129
-
p21ras-mediated decrease of the retinoblastoma protein in fibroblasts occurs through growth factor-dependent mechanisms
-
Kivinen, L., Tiihonen, E., Haapajärvi, T., and Laiho, M. p21ras-mediated decrease of the retinoblastoma protein in fibroblasts occurs through growth factor-dependent mechanisms. Cell Growth Differ., 7: 1705-1712, 1996.
-
(1996)
Cell Growth Differ.
, vol.7
, pp. 1705-1712
-
-
Kivinen, L.1
Tiihonen, E.2
Haapajärvi, T.3
Laiho, M.4
-
53
-
-
0027317086
-
Human retinoblastoma gene product prevents c-Ha-ras oncogene mediated cellular transformation of mouse fibroblasts
-
Kivinen, L., Pitkänen, K., and Laiho, M. Human retinoblastoma gene product prevents c-Ha-ras oncogene mediated cellular transformation of mouse fibroblasts. Oncogene, 8: 2703-2711, 1993.
-
(1993)
Oncogene
, vol.8
, pp. 2703-2711
-
-
Kivinen, L.1
Pitkänen, K.2
Laiho, M.3
-
54
-
-
0029133264
-
Cell cycle dependent effects of u.v.-radiation on p53 expression and retinoblastoma protein phosphorylation
-
Haapajärvi, T., Kivinen, L., Pitkänen, K., and Laiho, M. Cell cycle dependent effects of u.v.-radiation on p53 expression and retinoblastoma protein phosphorylation. Oncogene, 11: 151-159, 1995.
-
(1995)
Oncogene
, vol.11
, pp. 151-159
-
-
Haapajärvi, T.1
Kivinen, L.2
Pitkänen, K.3
Laiho, M.4
|