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Volumn 458, Issue 2, 1999, Pages 236-240
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Transgenic replacement of type V adenylyl cyclase identifies a critical mechanism of β-adrenergic receptor dysfunction in the G(αq) overexpressing mouse
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Author keywords
Adrenergic receptor; Adenylyl cyclase; cAMP; Desensitization; Hypertrophy
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Indexed keywords
ADENYLATE CYCLASE;
BETA ADRENERGIC RECEPTOR;
CYCLIC AMP;
GUANINE NUCLEOTIDE BINDING PROTEIN;
ISOENZYME;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CELL ACTIVATION;
CONTROLLED STUDY;
ENZYME ACTIVITY;
GENE OVEREXPRESSION;
HEART MUSCLE CELL;
HEART MUSCLE CONTRACTILITY;
MOUSE;
MUSCLE HYPERTROPHY;
NONHUMAN;
PRIORITY JOURNAL;
TRANSGENIC MOUSE;
ADENYLATE CYCLASE;
ANIMALS;
CARDIOMEGALY;
CATALYSIS;
DISEASE MODELS, ANIMAL;
GTP-BINDING PROTEIN ALPHA SUBUNITS, GQ-G11;
GTP-BINDING PROTEINS;
MALE;
MICE;
MICE, TRANSGENIC;
RECEPTORS, ADRENERGIC, BETA;
SIGNAL TRANSDUCTION;
VENTRICULAR FUNCTION;
ANIMALIA;
MUS MUSCULUS;
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EID: 0032874674
PISSN: 00145793
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-5793(99)01147-3 Document Type: Article |
Times cited : (53)
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References (16)
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