Hereditary deficiencies in complement C5 are associated with intensified neurodegenerative responses that implicate new roles for the C-system in neuronal and astrocytic functions

Neurobiology of Disease

Volumn 3, Issue 3, 1996, Pages 197-204

  Pasinetti, G M ^a   Tocco, G ^b   Sakhi, S ^c   Musleh, W D ^b   DeSimoni, M G ^d   Mascarucci, P ^d   Schreiber, S ^c   Baudry, M ^b   Finch, C E ^e  

^a MOUNT SINAI SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF SOUTHERN CALIFORNIA   (United States)

^c UNIVERSITY OF SOUTHERN CALIFORNIA   (United States)

^d MARIO NEGRI INSTITUTE FOR PHARMACOLOGICAL RESEARCH   (Italy)

^e UNIVERSITY OF SOUTHERN CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA AMINO 3 HYDROXY 5 METHYL 4 ISOXAZOLEPROPIONIC ACID; APOLIPOPROTEIN E; CALCIUM ION; COMPLEMENT COMPONENT C5; EXCITOTOXIN; GLIAL FIBRILLARY ACIDIC PROTEIN; GLUTAMIC ACID; INTERLEUKIN 6; KAINIC ACID; LIPOPOLYSACCHARIDE; MESSENGER RNA; N METHYL DEXTRO ASPARTIC ACID; TUMOR NECROSIS FACTOR;

ALZHEIMER DISEASE; ANIMAL CELL; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ASTROCYTE; BRAIN INJURY; COMPLEMENT DEFICIENCY; COMPLEMENT SYSTEM; CONTROLLED STUDY; DEGENERATIVE DISEASE; DISEASE ASSOCIATION; EXCITATORY POSTSYNAPTIC POTENTIAL; FRAMESHIFT MUTATION; HIPPOCAMPUS; MALE; MOUSE; NERVE CELL NECROSIS; NERVE CELL PLASTICITY; NEUROTRANSMISSION; NONHUMAN; PLEIOTROPY; PRIORITY JOURNAL;

EID: 0030175273     PISSN: 09699961     EISSN: None     Source Type: Journal    
DOI: 10.1006/nbdi.1996.0020     Document Type: Article

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