SCOPUS 정보 검색 플랫폼

Volumn 21, Issue 20, 2002, Pages 3190-3198

Overexpression of the HMGA2 gene in transgenic mice leads to the onset of pituitary adenomas

(13) Fedele, Monica a,b Battista, Sabrina a,b Kenyon, Lawrence c Baldassarre, Gustavo b Fidanza, Vincenzo b Klein Szanto, Andres J P d Parlow, A F e Visone, Rosa a Pierantoni, Giovanna M a Outwater, Eric f Santoro, Massimo a Croce, Carlo M b Fusco, Alfredo a,g

a UNIVERSITY OF NAPLES FEDERICO II (Italy)

b THOMAS JEFFERSON UNIVERSITY (United States)

c THOMAS JEFFERSON UNIVERSITY HOSPITAL (United States)

d Fox Chase Cancer Center (Italy)

e UCLA MEDICAL CENTER (United States)

f THOMAS JEFFERSON UNIVERSITY (United States)

g UNIVERSITY MAGNA GRAECIA OF CATANZARO (Italy)

Author keywords

Pit 1; Pituitary adenomas; Transgenic mice HMGA2; Tumorigenesis

Indexed keywords

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CLINICAL FEATURE; CONTROLLED STUDY; CYTOMEGALOVIRUS; FEMALE; GENE ISOLATION; GENE OVEREXPRESSION; GENE REARRANGEMENT; GROWTH HORMONE RELEASE; HISTOPATHOLOGY; HMGA2 GENE; HYPOPHYSIS ADENOMA; MALE; MESENCHYMOMA; MOUSE; NONHUMAN; PENETRANCE; PHENOTYPE; PRIORITY JOURNAL; PROLACTIN RELEASE; PROMOTER REGION; TRANSCRIPTION REGULATION; TRANSGENIC MOUSE; TUMOR CELL;

ABNORMALITIES; ADENOMA; ANIMALS; CELL TRANSFORMATION, NEOPLASTIC; CYTOMEGALOVIRUS; FEMALE; FEMALE UROGENITAL DISEASES; GENE EXPRESSION REGULATION; GROWTH HORMONE; HMGA2 PROTEIN; MALE; MALE UROGENITAL DISEASES; MICE; MICE, TRANSGENIC; NEOPLASMS, EXPERIMENTAL; PHENOTYPE; PITUITARY NEOPLASMS; PROLACTIN; PROLACTINOMA; RECOMBINANT FUSION PROTEINS; TRANSCRIPTION, GENETIC; TRANSGENES;

ANIMALIA; CYTOMEGALOVIRUS; MUS MUSCULUS;

EID: 85047700396 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205428 Document Type: Article

Times cited : (181)

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