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Volumn 21, Issue 12, 2002, Pages 1876-1881

Coincident inactivation of 14-3-3σ and p16INK4a is an early event in vulval squamous neoplasia

(10) Gasco, Milena a Sullivan, Alex b Repellin, Claire b Brooks, Louise b Farrell, Paul J b Tidy, John A c Dunne, Barbara d Gusterson, Barry d Evans, David J b Crook, Tim b

a S CROCE E CARLE HOSPITAL (Italy)

b IMPERIAL COLLEGE LONDON (United Kingdom)

c UNIVERSITY OF SHEFFIELD (United Kingdom)

d UNIVERSITY OF GLASGOW (United Kingdom)

Author keywords

14 3 3 ; CpG methylation; p16INK4a; Vulval cancer

Indexed keywords

PROTEIN P16;

ARTICLE; CPG ISLAND; EARLY DIAGNOSIS; FEMALE; GENE INACTIVATION; GENE MUTATION; GENE SILENCING; HETEROZYGOSITY LOSS; HUMAN; HUMAN TISSUE; MESSENGER RNA SYNTHESIS; METHYLATION; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; SQUAMOUS CELL CARCINOMA; VULVA CANCER;

CARCINOMA, SQUAMOUS CELL; CYCLIN-DEPENDENT KINASE INHIBITOR P16; DISEASE PROGRESSION; DNA METHYLATION; EXONUCLEASES; FEMALE; GENE SILENCING; HUMANS; LOSS OF HETEROZYGOSITY; NEOPLASM PROTEINS; PAPILLOMAVIRIDAE; PRECANCEROUS CONDITIONS; PROTEINS; RNA, MESSENGER; TRANSCRIPTION, GENETIC; TUMOR MARKERS, BIOLOGICAL; TUMOR SUPPRESSOR PROTEIN P53; VULVAR NEOPLASMS;

EID: 85047699461 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205256 Document Type: Article

Times cited : (93)

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