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Oncogene

Volumn 21, Issue 20, 2002, Pages 3247-3252

AML1 stimulates G1 to S progression via its transactivation domain

(2) Bernardin, Florence a Friedman, Alan D a

a JOHNS HOPKINS UNIVERSITY (United States)

Author keywords

AML1; CBF; Cell cycle; Leukemia; RUNX1

Indexed keywords

4 HYDROXYTAMOXIFEN; CORE BINDING FACTOR; DNA; HYBRID PROTEIN; METALLOTHIONEIN; MYELOPEROXIDASE; ONCOPROTEIN; TRANSACTIVATOR PROTEIN; TRANSCRIPTION FACTOR RUNX1; UNCLASSIFIED DRUG; ZINC;

ACUTE GRANULOCYTIC LEUKEMIA; ANIMAL CELL; APOPTOSIS; ARTICLE; CELL CYCLE G1 PHASE; CELL CYCLE S PHASE; CELL PROLIFERATION; CONTROLLED STUDY; LEUKEMOGENESIS; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DOMAIN; TRANSACTIVATION;

ACTIVE TRANSPORT, CELL NUCLEUS; ANIMALS; BINDING SITES; CCAAT-BINDING FACTOR; CELL LINE; CELL NUCLEUS; CORE BINDING FACTOR ALPHA 2 SUBUNIT; DNA; DNA, COMPLEMENTARY; DNA-BINDING PROTEINS; G1 PHASE; GENE EXPRESSION REGULATION; HEMATOPOIETIC STEM CELLS; HUMANS; MICE; PROTEIN STRUCTURE, TERTIARY; PROTO-ONCOGENE PROTEINS; RECOMBINANT FUSION PROTEINS; S PHASE; SEQUENCE DELETION; TAMOXIFEN; TRANS-ACTIVATION (GENETICS); TRANSCRIPTION FACTORS;

ANIMALIA;

EID: 85047696566 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205447 Document Type: Article

Times cited : (42)

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