A Brucella Type IV Effector Targets the COG Tethering Complex to Remodel Host Secretory Traffic and Promote Intracellular Replication

Cell Host and Microbe

Volumn 22, Issue 3, 2017, Pages 317-329.e7

  Miller, Cheryl N ^a   Smith, Erin P ^a   Cundiff, Jennifer A ^a   Knodler, Leigh A ^a   Bailey Blackburn, Jessica ^b   Lupashin, Vladimir ^b   Celli, Jean ^a  

^a WASHINGTON STATE UNIVERSITY   (United States)

^b UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

Author keywords

Brucella; COG complex; Golgi; macrophage; pathogenesis; Rab GTPases; secretory pathway; type IV secretion

Indexed keywords

ARTICLE; BONE MARROW DERIVED MACROPHAGE; BRUCELLA ABORTUS; CELL DIVISION; CELL VACUOLE; CONTROLLED STUDY; EMBRYO; GOLGI MEMBRANE; HEK293T CELL LINE; HELA CELL LINE; HUMAN; HUMAN CELL; NONHUMAN; ORGANELLE BIOGENESIS; PRIORITY JOURNAL; TYPE IV SECRETION SYSTEM; BRUCELLOSIS; CELL LINE; GENETICS; GOLGI COMPLEX; HOST PATHOGEN INTERACTION; METABOLISM; MICROBIOLOGY; PROTEIN TRANSPORT;

BACTERIAL PROTEIN;

BACTERIAL PROTEINS; BRUCELLA ABORTUS; BRUCELLOSIS; CELL LINE; GOLGI APPARATUS; HOST-PATHOGEN INTERACTIONS; HUMANS; PROTEIN TRANSPORT; TYPE IV SECRETION SYSTEMS; VACUOLES;

EID: 85028031149     PISSN: 19313128     EISSN: 19346069     Source Type: Journal    
DOI: 10.1016/j.chom.2017.07.017     Document Type: Article

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