-
1
-
-
84901850631
-
Monoamine oxidases as sources of oxidants in the heart
-
Kaludercic, N., Mialet-Perez, J., Paolocci, N., Parini, A., Di Lisa, F., Monoamine oxidases as sources of oxidants in the heart. J. Mol. Cell. Cardiol. 73 (2014), 34–42.
-
(2014)
J. Mol. Cell. Cardiol.
, vol.73
, pp. 34-42
-
-
Kaludercic, N.1
Mialet-Perez, J.2
Paolocci, N.3
Parini, A.4
Di Lisa, F.5
-
2
-
-
0348046389
-
The FAD binding sites of human monoamine oxidases A and B
-
Edmondson, D.E., Binda, C., Mattevi, A., The FAD binding sites of human monoamine oxidases A and B. Neurotoxicology 25 (2004), 63–72.
-
(2004)
Neurotoxicology
, vol.25
, pp. 63-72
-
-
Edmondson, D.E.1
Binda, C.2
Mattevi, A.3
-
3
-
-
79957936379
-
Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury
-
Kaludercic, N., Carpi, A., Menabo, R., Di Lisa, F., Paolocci, N., Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury. Biochim. Biophys. Acta 1813 (2011), 1323–1332.
-
(2011)
Biochim. Biophys. Acta
, vol.1813
, pp. 1323-1332
-
-
Kaludercic, N.1
Carpi, A.2
Menabo, R.3
Di Lisa, F.4
Paolocci, N.5
-
4
-
-
33645307953
-
The therapeutic potential of monoamine oxidase inhibitors
-
Youdim, M.B., Edmondson, D., Tipton, K.F., The therapeutic potential of monoamine oxidase inhibitors. Nat. Rev. Neurosci. 7 (2006), 295–309.
-
(2006)
Nat. Rev. Neurosci.
, vol.7
, pp. 295-309
-
-
Youdim, M.B.1
Edmondson, D.2
Tipton, K.F.3
-
5
-
-
84947764143
-
Pharmacology and clinical drug candidates in redox medicine
-
This review discusses the possibility of targeting the enzymatic sources of disease-relevant oxidative stress, rather than using antioxidants as therapeutic strategy. Pharmacological tools and their respective targets are reviewed in relation to disease.
-
5• Dao, V.T., Casas, A.I., Maghzal, G.J., Seredenina, T., Kaludercic, N., Robledinos-Anton, N., Di Lisa, F., Stocker, R., Ghezzi, P., Jaquet, V., et al. Pharmacology and clinical drug candidates in redox medicine. Antioxid. Redox Signal. 23 (2015), 1113–1129 This review discusses the possibility of targeting the enzymatic sources of disease-relevant oxidative stress, rather than using antioxidants as therapeutic strategy. Pharmacological tools and their respective targets are reviewed in relation to disease.
-
(2015)
Antioxid. Redox Signal.
, vol.23
, pp. 1113-1129
-
-
Dao, V.T.1
Casas, A.I.2
Maghzal, G.J.3
Seredenina, T.4
Kaludercic, N.5
Robledinos-Anton, N.6
Di Lisa, F.7
Stocker, R.8
Ghezzi, P.9
Jaquet, V.10
-
6
-
-
85012149148
-
Longer duration of MAO-B inhibitor exposure is associated with less clinical decline in Parkinson's disease: an analysis of NET-PD LS1
-
Hauser, R.A., Li, R., Perez, A., Ren, X., Weintraub, D., Elm, J., Goudreau, J.L., Morgan, J.C., Fang, J.Y., Aminoff, M.J., et al. Longer duration of MAO-B inhibitor exposure is associated with less clinical decline in Parkinson's disease: an analysis of NET-PD LS1. J. Parkinson's Dis. 7.1 (2016), 117–127.
-
(2016)
J. Parkinson's Dis.
, vol.7.1
, pp. 117-127
-
-
Hauser, R.A.1
Li, R.2
Perez, A.3
Ren, X.4
Weintraub, D.5
Elm, J.6
Goudreau, J.L.7
Morgan, J.C.8
Fang, J.Y.9
Aminoff, M.J.10
-
7
-
-
85050579297
-
Clinical pharmacology review of safinamide for the treatment of Parkinson's disease
-
Fabbri, M., Rosa, M.M., Abreu, D., Ferreira, J.J., Clinical pharmacology review of safinamide for the treatment of Parkinson's disease. Neurodegener. Dis. Manag. 5 (2015), 481–496.
-
(2015)
Neurodegener. Dis. Manag.
, vol.5
, pp. 481-496
-
-
Fabbri, M.1
Rosa, M.M.2
Abreu, D.3
Ferreira, J.J.4
-
8
-
-
84922381210
-
Heart disease and stroke statistics—2015 update: a report from the American Heart Association
-
Mozaffarian, D., Benjamin, E.J., Go, A.S., Arnett, D.K., Blaha, M.J., Cushman, M., de Ferranti, S., Despres, J.P., Fullerton, H.J., Howard, V.J., et al. Heart disease and stroke statistics—2015 update: a report from the American Heart Association. Circulation 131 (2015), e29–322.
-
(2015)
Circulation
, vol.131
, pp. e29-322
-
-
Mozaffarian, D.1
Benjamin, E.J.2
Go, A.S.3
Arnett, D.K.4
Blaha, M.J.5
Cushman, M.6
de Ferranti, S.7
Despres, J.P.8
Fullerton, H.J.9
Howard, V.J.10
-
9
-
-
84947732762
-
Reactive oxygen-related diseases: therapeutic targets and emerging clinical indications
-
This review covers the disease-relevant sources of ROS and discusses the reasons for the failure of chronic antioxidant therapies. It points out the importance of treating the disease-relevant ROS at the right moment, leaving the physiological ROS formation intact.
-
9• Casas, A.I., Dao, V.T., Daiber, A., Maghzal, G.J., Di Lisa, F., Kaludercic, N., Leach, S., Cuadrado, A., Jaquet, V., Seredenina, T., et al. Reactive oxygen-related diseases: therapeutic targets and emerging clinical indications. Antioxid. Redox Signal. 23 (2015), 1171–1185 This review covers the disease-relevant sources of ROS and discusses the reasons for the failure of chronic antioxidant therapies. It points out the importance of treating the disease-relevant ROS at the right moment, leaving the physiological ROS formation intact.
-
(2015)
Antioxid. Redox Signal.
, vol.23
, pp. 1171-1185
-
-
Casas, A.I.1
Dao, V.T.2
Daiber, A.3
Maghzal, G.J.4
Di Lisa, F.5
Kaludercic, N.6
Leach, S.7
Cuadrado, A.8
Jaquet, V.9
Seredenina, T.10
-
10
-
-
0030589009
-
The metabolism of tyramine by monoamine oxidase A/B causes oxidative damage to mitochondrial DNA
-
Hauptmann, N., Grimsby, J., Shih, J.C., Cadenas, E., The metabolism of tyramine by monoamine oxidase A/B causes oxidative damage to mitochondrial DNA. Arch. Biochem. Biophys. 335 (1996), 295–304.
-
(1996)
Arch. Biochem. Biophys.
, vol.335
, pp. 295-304
-
-
Hauptmann, N.1
Grimsby, J.2
Shih, J.C.3
Cadenas, E.4
-
11
-
-
84898670714
-
Monoamine oxidase is a major determinant of redox balance in human atrial myocardium and is associated with postoperative atrial fibrillation
-
2 in comparison to the mitochondrial respiratory chain. The Authors also show that MAO activity in this tissue is the major risk factor and predictor for the postoperative atrial fibrillation.
-
2 in comparison to the mitochondrial respiratory chain. The Authors also show that MAO activity in this tissue is the major risk factor and predictor for the postoperative atrial fibrillation.
-
(2014)
J. Am. Heart Assoc.
, vol.3
, pp. e000713
-
-
Anderson, E.J.1
Efird, J.T.2
Davies, S.W.3
O'Neal, W.T.4
Darden, T.M.5
Thayne, K.A.6
Katunga, L.A.7
Kindell, L.C.8
Ferguson, T.B.9
Anderson, C.A.10
-
12
-
-
33644874936
-
Oxidative stress by monoamine oxidase mediates receptor-independent cardiomyocyte apoptosis by serotonin and postischemic myocardial injury
-
Bianchi, P., Kunduzova, O., Masini, E., Cambon, C., Bani, D., Raimondi, L., Seguelas, M.H., Nistri, S., Colucci, W., Leducq, N., et al. Oxidative stress by monoamine oxidase mediates receptor-independent cardiomyocyte apoptosis by serotonin and postischemic myocardial injury. Circulation 112 (2005), 3297–3305.
-
(2005)
Circulation
, vol.112
, pp. 3297-3305
-
-
Bianchi, P.1
Kunduzova, O.2
Masini, E.3
Cambon, C.4
Bani, D.5
Raimondi, L.6
Seguelas, M.H.7
Nistri, S.8
Colucci, W.9
Leducq, N.10
-
13
-
-
33846047834
-
Oxidative stress-dependent sphingosine kinase-1 inhibition mediates monoamine oxidase A-associated cardiac cell apoptosis
-
Pchejetski, D., Kunduzova, O., Dayon, A., Calise, D., Seguelas, M.H., Leducq, N., Seif, I., Parini, A., Cuvillier, O., Oxidative stress-dependent sphingosine kinase-1 inhibition mediates monoamine oxidase A-associated cardiac cell apoptosis. Circ. Res. 100 (2007), 41–49.
-
(2007)
Circ. Res.
, vol.100
, pp. 41-49
-
-
Pchejetski, D.1
Kunduzova, O.2
Dayon, A.3
Calise, D.4
Seguelas, M.H.5
Leducq, N.6
Seif, I.7
Parini, A.8
Cuvillier, O.9
-
14
-
-
67649243697
-
The cardioprotective effects elicited by p66(Shc) ablation demonstrate the crucial role of mitochondrial ROS formation in ischemia/reperfusion injury
-
Carpi, A., Menabo, R., Kaludercic, N., Pelicci, P., Di Lisa, F., Giorgio, M., The cardioprotective effects elicited by p66(Shc) ablation demonstrate the crucial role of mitochondrial ROS formation in ischemia/reperfusion injury. Biochim. Biophys. Acta 1787 (2009), 774–780.
-
(2009)
Biochim. Biophys. Acta
, vol.1787
, pp. 774-780
-
-
Carpi, A.1
Menabo, R.2
Kaludercic, N.3
Pelicci, P.4
Di Lisa, F.5
Giorgio, M.6
-
15
-
-
84891823906
-
Monoamine oxidase B prompts mitochondrial and cardiac dysfunction in pressure overloaded hearts
-
2 formation primarily in the mitochondria and that MAO-dependent ROS generation can directly target mitochondria to induce mitochondrial dysfunction. In addition, the Authors show for the first time that MAO-dependent aldehyde generation is able to induce mitochondrial dysfunction, ultimately leading to heart failure.
-
2 formation primarily in the mitochondria and that MAO-dependent ROS generation can directly target mitochondria to induce mitochondrial dysfunction. In addition, the Authors show for the first time that MAO-dependent aldehyde generation is able to induce mitochondrial dysfunction, ultimately leading to heart failure.
-
(2014)
Antioxid. Redox Signal.
, vol.20
, pp. 267-280
-
-
Kaludercic, N.1
Carpi, A.2
Nagayama, T.3
Sivakumaran, V.4
Zhu, G.5
Lai, E.W.6
Bedja, D.7
De Mario, A.8
Chen, K.9
Gabrielson, K.L.10
-
16
-
-
74049129002
-
Monoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload
-
Kaludercic, N., Takimoto, E., Nagayama, T., Feng, N., Lai, E.W., Bedja, D., Chen, K., Gabrielson, K.L., Blakely, R.D., Shih, J.C., et al. Monoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload. Circ. Res. 106 (2010), 193–202.
-
(2010)
Circ. Res.
, vol.106
, pp. 193-202
-
-
Kaludercic, N.1
Takimoto, E.2
Nagayama, T.3
Feng, N.4
Lai, E.W.5
Bedja, D.6
Chen, K.7
Gabrielson, K.L.8
Blakely, R.D.9
Shih, J.C.10
-
17
-
-
84940373910
-
Monoamine oxidase-A is an important source of oxidative stress and promotes cardiac dysfunction, apoptosis, and fibrosis in diabetic cardiomyopathy
-
Umbarkar, P., Singh, S., Arkat, S., Bodhankar, S.L., Lohidasan, S., Sitasawad, S.L., Monoamine oxidase-A is an important source of oxidative stress and promotes cardiac dysfunction, apoptosis, and fibrosis in diabetic cardiomyopathy. Free Radic. Biol. Med. 87 (2015), 263–273.
-
(2015)
Free Radic. Biol. Med.
, vol.87
, pp. 263-273
-
-
Umbarkar, P.1
Singh, S.2
Arkat, S.3
Bodhankar, S.L.4
Lohidasan, S.5
Sitasawad, S.L.6
-
18
-
-
84870544267
-
p53-PGC-1alpha pathway mediates oxidative mitochondrial damage and cardiomyocyte necrosis induced by monoamine oxidase-A upregulation: role in chronic left ventricular dysfunction in mice
-
Villeneuve, C., Guilbeau-Frugier, C., Sicard, P., Lairez, O., Ordener, C., Duparc, T., De Paulis, D., Couderc, B., Spreux-Varoquaux, O., Tortosa, F., et al. p53-PGC-1alpha pathway mediates oxidative mitochondrial damage and cardiomyocyte necrosis induced by monoamine oxidase-A upregulation: role in chronic left ventricular dysfunction in mice. Antioxid. Redox Signal. 18 (2013), 5–18.
-
(2013)
Antioxid. Redox Signal.
, vol.18
, pp. 5-18
-
-
Villeneuve, C.1
Guilbeau-Frugier, C.2
Sicard, P.3
Lairez, O.4
Ordener, C.5
Duparc, T.6
De Paulis, D.7
Couderc, B.8
Spreux-Varoquaux, O.9
Tortosa, F.10
-
19
-
-
84978035652
-
Oxidative stress by monoamine oxidase-A impairs transcription factor EB activation and autophagosome clearance, leading to cardiomyocyte necrosis and heart failure
-
2 that, in turn, prevents TFEB translocation to the nucleus and lysosomal biogenesis.
-
2 that, in turn, prevents TFEB translocation to the nucleus and lysosomal biogenesis.
-
(2016)
Antioxid. Redox Signal.
, vol.25
, pp. 10-27
-
-
Santin, Y.1
Sicard, P.2
Vigneron, F.3
Guilbeau-Frugier, C.4
Dutaur, M.5
Lairez, O.6
Couderc, B.7
Manni, D.8
Korolchuk, V.I.9
Lezoualc'h, F.10
-
20
-
-
84858699672
-
Ammonia increases paracellular permeability of rat brain endothelial cells by a mechanism encompassing oxidative/nitrosative stress and activation of matrix metalloproteinases
-
Skowronska, M., Zielinska, M., Wojcik-Stanaszek, L., Ruszkiewicz, J., Milatovic, D., Aschner, M., Albrecht, J., Ammonia increases paracellular permeability of rat brain endothelial cells by a mechanism encompassing oxidative/nitrosative stress and activation of matrix metalloproteinases. J. Neurochem. 121 (2012), 125–134.
-
(2012)
J. Neurochem.
, vol.121
, pp. 125-134
-
-
Skowronska, M.1
Zielinska, M.2
Wojcik-Stanaszek, L.3
Ruszkiewicz, J.4
Milatovic, D.5
Aschner, M.6
Albrecht, J.7
-
21
-
-
84903649897
-
Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release
-
Zorov, D.B., Juhaszova, M., Sollott, S.J., Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release. Physiol. Rev. 94 (2014), 909–950.
-
(2014)
Physiol. Rev.
, vol.94
, pp. 909-950
-
-
Zorov, D.B.1
Juhaszova, M.2
Sollott, S.J.3
-
22
-
-
84964455476
-
Monoamine oxidase-induced hydroxyl radical production and cardiomyocyte injury during myocardial ischemia-reperfusion in rats
-
Inagaki, T., Akiyama, T., Du, C.K., Zhan, D.Y., Yoshimoto, M., Shirai, M., Monoamine oxidase-induced hydroxyl radical production and cardiomyocyte injury during myocardial ischemia-reperfusion in rats. Free Radic. Res. 50 (2016), 645–653.
-
(2016)
Free Radic. Res.
, vol.50
, pp. 645-653
-
-
Inagaki, T.1
Akiyama, T.2
Du, C.K.3
Zhan, D.Y.4
Yoshimoto, M.5
Shirai, M.6
-
23
-
-
77957870248
-
Oxidative stress by monoamine oxidases is causally involved in myofiber damage in muscular dystrophy
-
Menazza, S., Blaauw, B., Tiepolo, T., Toniolo, L., Braghetta, P., Spolaore, B., Reggiani, C., Di Lisa, F., Bonaldo, P., Canton, M., Oxidative stress by monoamine oxidases is causally involved in myofiber damage in muscular dystrophy. Hum. Mol. Genet. 19 (2010), 4207–4215.
-
(2010)
Hum. Mol. Genet.
, vol.19
, pp. 4207-4215
-
-
Menazza, S.1
Blaauw, B.2
Tiepolo, T.3
Toniolo, L.4
Braghetta, P.5
Spolaore, B.6
Reggiani, C.7
Di Lisa, F.8
Bonaldo, P.9
Canton, M.10
-
24
-
-
33645023189
-
Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization
-
Canton, M., Skyschally, A., Menabo, R., Boengler, K., Gres, P., Schulz, R., Haude, M., Erbel, R., Di Lisa, F., Heusch, G., Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolization. Eur. Heart J. 27 (2006), 875–881.
-
(2006)
Eur. Heart J.
, vol.27
, pp. 875-881
-
-
Canton, M.1
Skyschally, A.2
Menabo, R.3
Boengler, K.4
Gres, P.5
Schulz, R.6
Haude, M.7
Erbel, R.8
Di Lisa, F.9
Heusch, G.10
-
25
-
-
78651267358
-
Oxidation of myofibrillar proteins in human heart failure
-
Canton, M., Menazza, S., Sheeran, F.L., Polverino de Laureto, P., Di Lisa, F., Pepe, S., Oxidation of myofibrillar proteins in human heart failure. J. Am. Coll. Cardiol. 57 (2011), 300–309.
-
(2011)
J. Am. Coll. Cardiol.
, vol.57
, pp. 300-309
-
-
Canton, M.1
Menazza, S.2
Sheeran, F.L.3
Polverino de Laureto, P.4
Di Lisa, F.5
Pepe, S.6
-
26
-
-
34249908646
-
MAO-A-induced mitogenic signaling is mediated by reactive oxygen species, MMP-2, and the sphingolipid pathway
-
Coatrieux, C., Sanson, M., Negre-Salvayre, A., Parini, A., Hannun, Y., Itohara, S., Salvayre, R., Auge, N., MAO-A-induced mitogenic signaling is mediated by reactive oxygen species, MMP-2, and the sphingolipid pathway. Free Radic. Biol. Med. 43 (2007), 80–89.
-
(2007)
Free Radic. Biol. Med.
, vol.43
, pp. 80-89
-
-
Coatrieux, C.1
Sanson, M.2
Negre-Salvayre, A.3
Parini, A.4
Hannun, Y.5
Itohara, S.6
Salvayre, R.7
Auge, N.8
-
27
-
-
66949176522
-
Mitochondria and vascular pathology
-
Di Lisa, F., Kaludercic, N., Carpi, A., Menabo, R., Giorgio, M., Mitochondria and vascular pathology. Pharmacol. Rep. 61 (2009), 123–130.
-
(2009)
Pharmacol. Rep.
, vol.61
, pp. 123-130
-
-
Di Lisa, F.1
Kaludercic, N.2
Carpi, A.3
Menabo, R.4
Giorgio, M.5
-
28
-
-
78349297565
-
Oxidative stress and diabetic complications
-
Giacco, F., Brownlee, M., Oxidative stress and diabetic complications. Circ. Res. 107 (2010), 1058–1070.
-
(2010)
Circ. Res.
, vol.107
, pp. 1058-1070
-
-
Giacco, F.1
Brownlee, M.2
-
29
-
-
84880729947
-
Monoamine oxidases are mediators of endothelial dysfunction in the mouse aorta
-
Sturza, A., Leisegang, M.S., Babelova, A., Schroder, K., Benkhoff, S., Loot, A.E., Fleming, I., Schulz, R., Muntean, D.M., Brandes, R.P., Monoamine oxidases are mediators of endothelial dysfunction in the mouse aorta. Hypertension 62 (2013), 140–146.
-
(2013)
Hypertension
, vol.62
, pp. 140-146
-
-
Sturza, A.1
Leisegang, M.S.2
Babelova, A.3
Schroder, K.4
Benkhoff, S.5
Loot, A.E.6
Fleming, I.7
Schulz, R.8
Muntean, D.M.9
Brandes, R.P.10
-
30
-
-
24344499139
-
Hydrogen peroxide regulation of endothelial function: origins, mechanisms, and consequences
-
Cai, H., Hydrogen peroxide regulation of endothelial function: origins, mechanisms, and consequences. Cardiovasc. Res. 68 (2005), 26–36.
-
(2005)
Cardiovasc. Res.
, vol.68
, pp. 26-36
-
-
Cai, H.1
-
31
-
-
34548695863
-
Cysteine redox sensor in PKGIa enables oxidant-induced activation
-
Burgoyne, J.R., Madhani, M., Cuello, F., Charles, R.L., Brennan, J.P., Schroder, E., Browning, D.D., Eaton, P., Cysteine redox sensor in PKGIa enables oxidant-induced activation. Science 317 (2007), 1393–1397.
-
(2007)
Science
, vol.317
, pp. 1393-1397
-
-
Burgoyne, J.R.1
Madhani, M.2
Cuello, F.3
Charles, R.L.4
Brennan, J.P.5
Schroder, E.6
Browning, D.D.7
Eaton, P.8
-
32
-
-
79954608953
-
S-Glutathionylation reshapes our understanding of endothelial nitric oxide synthase uncoupling and nitric oxide/reactive oxygen species-mediated signaling
-
Zweier, J.L., Chen, C.A., Druhan, L.J., S-Glutathionylation reshapes our understanding of endothelial nitric oxide synthase uncoupling and nitric oxide/reactive oxygen species-mediated signaling. Antioxid. Redox Signal. 14 (2011), 1769–1775.
-
(2011)
Antioxid. Redox Signal.
, vol.14
, pp. 1769-1775
-
-
Zweier, J.L.1
Chen, C.A.2
Druhan, L.J.3
-
33
-
-
84907766487
-
Vascular oxidative stress, nitric oxide and atherosclerosis
-
Li, H., Horke, S., Forstermann, U., Vascular oxidative stress, nitric oxide and atherosclerosis. Atherosclerosis 237 (2014), 208–219.
-
(2014)
Atherosclerosis
, vol.237
, pp. 208-219
-
-
Li, H.1
Horke, S.2
Forstermann, U.3
-
34
-
-
33748302123
-
Targeting the heme-oxidized nitric oxide receptor for selective vasodilatation of diseased blood vessels
-
Stasch, J.P., Schmidt, P.M., Nedvetsky, P.I., Nedvetskaya, T.Y., HS, A.K., Meurer, S., Deile, M., Taye, A., Knorr, A., Lapp, H., et al. Targeting the heme-oxidized nitric oxide receptor for selective vasodilatation of diseased blood vessels. J. Clin. Investig. 116 (2006), 2552–2561.
-
(2006)
J. Clin. Investig.
, vol.116
, pp. 2552-2561
-
-
Stasch, J.P.1
Schmidt, P.M.2
Nedvetsky, P.I.3
Nedvetskaya, T.Y.4
HS, A.K.5
Meurer, S.6
Deile, M.7
Taye, A.8
Knorr, A.9
Lapp, H.10
-
35
-
-
84943517017
-
Monoamine oxidases are novel sources of cardiovascular oxidative stress in experimental diabetes
-
Sturza, A., Duicu, O.M., Vaduva, A., Danila, M.D., Noveanu, L., Varro, A., Muntean, D.M., Monoamine oxidases are novel sources of cardiovascular oxidative stress in experimental diabetes. Can. J. Physiol. Pharmacol. 93 (2015), 555–561.
-
(2015)
Can. J. Physiol. Pharmacol.
, vol.93
, pp. 555-561
-
-
Sturza, A.1
Duicu, O.M.2
Vaduva, A.3
Danila, M.D.4
Noveanu, L.5
Varro, A.6
Muntean, D.M.7
-
36
-
-
77958586636
-
Mitochondrial monoamine oxidase-A-mediated hydrogen peroxide generation enhances 5-hydroxytryptamine-induced contraction of rat basilar artery
-
Poon, C.C., Seto, S.W., Au, A.L., Zhang, Q., Li, R.W., Lee, W.Y., Leung, G.P., Kong, S.K., Yeung, J.H., Ngai, S.M., et al. Mitochondrial monoamine oxidase-A-mediated hydrogen peroxide generation enhances 5-hydroxytryptamine-induced contraction of rat basilar artery. Br. J. Pharmacol. 161 (2010), 1086–1098.
-
(2010)
Br. J. Pharmacol.
, vol.161
, pp. 1086-1098
-
-
Poon, C.C.1
Seto, S.W.2
Au, A.L.3
Zhang, Q.4
Li, R.W.5
Lee, W.Y.6
Leung, G.P.7
Kong, S.K.8
Yeung, J.H.9
Ngai, S.M.10
-
37
-
-
84989946446
-
Monoamine oxidase inhibition improves vascular function in mammary arteries from nondiabetic and diabetic patients with coronary heart disease
-
Lighezan, R., Sturza, A., Duicu, O.M., Ceausu, R.A., Vaduva, A., Gaspar, M., Feier, H., Vaida, M., Ivan, V., Lighezan, D., et al. Monoamine oxidase inhibition improves vascular function in mammary arteries from nondiabetic and diabetic patients with coronary heart disease. Can. J. Physiol. Pharmacol. 94 (2016), 1040–1047.
-
(2016)
Can. J. Physiol. Pharmacol.
, vol.94
, pp. 1040-1047
-
-
Lighezan, R.1
Sturza, A.2
Duicu, O.M.3
Ceausu, R.A.4
Vaduva, A.5
Gaspar, M.6
Feier, H.7
Vaida, M.8
Ivan, V.9
Lighezan, D.10
-
38
-
-
70449324440
-
The aminase oxidase inhibitors. Their current place in clinical medicine
-
Oblath, R.W., Griffith, G.C., The aminase oxidase inhibitors. Their current place in clinical medicine. Calif. Med. 93 (1960), 343–346.
-
(1960)
Calif. Med.
, vol.93
, pp. 343-346
-
-
Oblath, R.W.1
Griffith, G.C.2
-
39
-
-
70449301912
-
Amine oxidase inhibitors; their current place in the therapy of cardiovascular diseases
-
Griffith, G.C., Amine oxidase inhibitors; their current place in the therapy of cardiovascular diseases. Circulation 22 (1960), 1156–1165.
-
(1960)
Circulation
, vol.22
, pp. 1156-1165
-
-
Griffith, G.C.1
-
40
-
-
50549177031
-
The importance of the monoamine oxidase inhibitors in cardiovascular disease
-
Griffith, G.C., The importance of the monoamine oxidase inhibitors in cardiovascular disease. Am. J. Cardiol. 6 (1960), 1103–1105.
-
(1960)
Am. J. Cardiol.
, vol.6
, pp. 1103-1105
-
-
Griffith, G.C.1
-
41
-
-
85012079858
-
Glycaemic control by monoamine oxidase inhibition in a patient with type 1 diabetes
-
Emory, H., Mizrahi, N., Glycaemic control by monoamine oxidase inhibition in a patient with type 1 diabetes. Diabetes Vasc. Dis. Res. 14:2 (2016), 163–165.
-
(2016)
Diabetes Vasc. Dis. Res.
, vol.14
, Issue.2
, pp. 163-165
-
-
Emory, H.1
Mizrahi, N.2
-
42
-
-
84855869138
-
Molecular insights into human monoamine oxidase B inhibition by the glitazone anti-diabetes drugs
-
Binda, C., Aldeco, M., Geldenhuys, W.J., Tortorici, M., Mattevi, A., Edmondson, D.E., Molecular insights into human monoamine oxidase B inhibition by the glitazone anti-diabetes drugs. ACS Med. Chem. Lett. 3 (2011), 39–42.
-
(2011)
ACS Med. Chem. Lett.
, vol.3
, pp. 39-42
-
-
Binda, C.1
Aldeco, M.2
Geldenhuys, W.J.3
Tortorici, M.4
Mattevi, A.5
Edmondson, D.E.6
-
43
-
-
84907423671
-
Revisiting the “cheese reaction”: more than just a hypertensive crisis?
-
Alkhouli, M., Mathur, M., Patil, P., Revisiting the “cheese reaction”: more than just a hypertensive crisis?. J. Clin. Psychopharmacol. 34 (2014), 665–667.
-
(2014)
J. Clin. Psychopharmacol.
, vol.34
, pp. 665-667
-
-
Alkhouli, M.1
Mathur, M.2
Patil, P.3
-
44
-
-
78650046296
-
Phenelzine-induced myocardial injury: a case report
-
Ngo, A.S., Ho, R.Y., Olson, K.R., Phenelzine-induced myocardial injury: a case report. J. Med. Toxicol. 6 (2010), 431–434.
-
(2010)
J. Med. Toxicol.
, vol.6
, pp. 431-434
-
-
Ngo, A.S.1
Ho, R.Y.2
Olson, K.R.3
-
45
-
-
0037361293
-
Moclobemide: therapeutic use and clinical studies
-
Bonnet, U., Moclobemide: therapeutic use and clinical studies. CNS Drug Rev. 9 (2003), 97–140.
-
(2003)
CNS Drug Rev.
, vol.9
, pp. 97-140
-
-
Bonnet, U.1
-
46
-
-
0347415712
-
Therapeutic applications of selective and non-selective inhibitors of monoamine oxidase A and B that do not cause significant tyramine potentiation
-
Youdim, M.B., Weinstock, M., Therapeutic applications of selective and non-selective inhibitors of monoamine oxidase A and B that do not cause significant tyramine potentiation. Neurotoxicology 25 (2004), 243–250.
-
(2004)
Neurotoxicology
, vol.25
, pp. 243-250
-
-
Youdim, M.B.1
Weinstock, M.2
-
47
-
-
84865763460
-
Antidepressive treatment with monoamine oxidase inhibitors and the occurrence of intraoperative hemodynamic events: a retrospective observational cohort study
-
van Haelst, I.M., van Klei, W.A., Doodeman, H.J., Kalkman, C.J., Egberts, T.C., Group, M.S., Antidepressive treatment with monoamine oxidase inhibitors and the occurrence of intraoperative hemodynamic events: a retrospective observational cohort study. J. Clin. Psychiatry 73 (2012), 1103–1109.
-
(2012)
J. Clin. Psychiatry
, vol.73
, pp. 1103-1109
-
-
van Haelst, I.M.1
van Klei, W.A.2
Doodeman, H.J.3
Kalkman, C.J.4
Egberts, T.C.5
Group, M.S.6
|