Author Correction: Genetic and regulatory mechanism of susceptibility to high-hyperdiploid acute lymphoblastic leukaemia at 10q21.2 (Nature Communications (2017) 8 (14616) DOI: 10.1038/ncomms14616);Genetic and regulatory mechanism of susceptibility to high-hyperdiploid acute lymphoblastic leukaemia at 10p21.2

Nature Communications

Volumn 8, Issue , 2017, Pages

  Studd, James B ^a   Vijayakrishnan, Jayaram ^a   Yang, Minjun ^b   Migliorini, Gabriele ^a   Paulsson, Kajsa ^b   Houlston, Richard S ^a  

^a INSTITUTE OF CANCER RESEARCH   (United Kingdom)

^b LUND UNIVERSITY   (Sweden)

Author keywords

[No Author keywords available]

Indexed keywords

TRANSCRIPTION FACTOR RUNX3; ARID5B PROTEIN, HUMAN; DNA BINDING PROTEIN; RUNX3 PROTEIN, HUMAN; TRANSCRIPTION FACTOR;

ALLELE; CANCER; CYTOGENETICS; ETIOLOGY; GENE EXPRESSION; GENETIC VARIATION; GENOME; HEALTH RISK; HERITABILITY; POLYMORPHISM;

ACUTE LYMPHOBLASTIC LEUKEMIA; ALLELE; ARTICLE; BINDING AFFINITY; CHROMOSOME 10; CONTROLLED STUDY; ENHANCER REGION; GENE EXPRESSION; GENE FREQUENCY; GENE LINKAGE DISEQUILIBRIUM; GENE SILENCING; GENETIC ASSOCIATION; GENETIC RISK; GENETIC SUSCEPTIBILITY; GENETIC VARIATION; GENOTYPE; HAPLOTYPE; HELA CELL LINE; HUMAN; HUMAN CELL; IN VITRO STUDY; QUANTITATIVE TRAIT LOCUS; REGULATORY MECHANISM; SINGLE NUCLEOTIDE POLYMORPHISM; TRANSCRIPTION INITIATION SITE; WHOLE GENOME SEQUENCING; CHILD; DIPLOIDY; GENE EXPRESSION REGULATION; GENE LOCUS; GENETIC EPIGENESIS; GENETIC PREDISPOSITION; GENETICS; GENOME-WIDE ASSOCIATION STUDY; META ANALYSIS; METABOLISM; PRESCHOOL CHILD; SEQUENCE ANALYSIS; TUMOR CELL LINE;

ALLELES; CELL LINE, TUMOR; CHILD; CHILD, PRESCHOOL; CHROMOSOMES, HUMAN, PAIR 10; CORE BINDING FACTOR ALPHA 3 SUBUNIT; DIPLOIDY; DNA-BINDING PROTEINS; ENHANCER ELEMENTS, GENETIC; EPIGENESIS, GENETIC; GENE EXPRESSION REGULATION, LEUKEMIC; GENETIC LOCI; GENETIC PREDISPOSITION TO DISEASE; GENETIC VARIATION; GENOME-WIDE ASSOCIATION STUDY; HUMANS; POLYMORPHISM, SINGLE NUCLEOTIDE; PRECURSOR CELL LYMPHOBLASTIC LEUKEMIA-LYMPHOMA; SEQUENCE ANALYSIS, RNA; TRANSCRIPTION FACTORS;

EID: 85014578851     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms16204     Document Type: Erratum

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