Innate lymphoid cells are the predominant source of IL-17A during the early pathogenesis of acute respiratory distress syndrome

American Journal of Respiratory and Critical Care Medicine

Volumn 193, Issue 4, 2016, Pages 407-416

  Muir, Roshell ^a   Osbourn, Megan ^a   Dubois, Alice V ^a   Doran, Emma ^a   Small, Donna M ^a   Monahan, Avril ^a   O'Kane, Cecilia M ^a   McAllister, Katherine ^a   Fitzgerald, Denise C ^a   Kissenpfennig, Adrien ^a   McAuley, Daniel F ^a,b   Ingram, Rebecca J ^a  

^a QUEEN'S UNIVERSITY BELFAST   (United Kingdom)

^b ROYAL VICTORIA HOSPITAL   (United Kingdom)

Author keywords

Acute lung injury; Acute respiratory distress syndrome; Group 3 innate lymphoid cell; IL 17; Lymphocyte

Indexed keywords

CD45 ANTIGEN; CHEMOKINE RECEPTOR CCR6; INTERLEUKIN 17; INTERLEUKIN 23; INTERLEUKIN 6; INTERLEUKIN 8; LIPOPOLYSACCHARIDE; PROTEIN; RAG2 PROTEIN; RECOMBINANT CYTOKINE; RECOMBINANT INTERLEUKIN 2; RECOMBINANT INTERLEUKIN 23; RECOMBINANT INTERLEUKIN 7; RORGAMMAT PROTEIN; STEM CELL FACTOR RECEPTOR; UNCLASSIFIED DRUG;

ACUTE LUNG INJURY; ADULT RESPIRATORY DISTRESS SYNDROME; ALPHABETA T LYMPHOCYTE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BACTERIAL PNEUMONIA; CD4+ T LYMPHOCYTE; CD8+ T LYMPHOCYTE; CELL CULTURE; CONTROLLED STUDY; CYTOKINE PRODUCTION; DISEASE SEVERITY; FLOW CYTOMETRY; GENE EXPRESSION; HISTOLOGY; KNOCKOUT MOUSE; LYMPHOID CELL; MOUSE; NATURAL KILLER T CELL; NEUTROPHIL; NONHUMAN; PATHOGENESIS; PHENOTYPE; PNEUMONIA; PRIORITY JOURNAL; PSEUDOMONAS INFECTION; QUANTITATIVE ANALYSIS; REAL TIME POLYMERASE CHAIN REACTION; WILD TYPE; ANIMAL; BIOSYNTHESIS; C57BL MOUSE; DISEASE MODEL; FEMALE; LUNG; LYMPHOCYTE; MALE; METABOLISM; NEUTROPHIL CHEMOTAXIS; PATHOLOGY;

ANIMALS; DISEASE MODELS, ANIMAL; FEMALE; FLOW CYTOMETRY; INTERLEUKIN-17; LUNG; LYMPHOCYTES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEUTROPHIL INFILTRATION; RESPIRATORY DISTRESS SYNDROME, ADULT;

EID: 84975117326     PISSN: 1073449X     EISSN: 15354970     Source Type: Journal    
DOI: 10.1164/rccm.201410-1782OC     Document Type: Article

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