Gpr97 is dispensable for metabolic syndrome but is involved in macrophage inflammation in high-fat diet-induced obesity in mice

Scientific Reports

Volumn 6, Issue , 2016, Pages

  Shi, Jueping ^a   Zhang, Xiaoyu ^a   Wang, Shaoying ^a   Wang, Jinjin ^b   Du, Bing ^a   Wang, Zhugang ^b   Liu, Mingyao ^a   Jiang, Wenzheng ^a   Qian, Min ^a   Ren, Hua ^a  

^a EAST CHINA NORMAL UNIVERSITY   (China)

^b Shanghai Research Center for Model Organisms   (China)

Author keywords

[No Author keywords available]

Indexed keywords

G PROTEIN COUPLED RECEPTOR; GPR97 PROTEIN, MOUSE; TUMOR NECROSIS FACTOR;

ADVERSE EFFECTS; ANIMAL; C57BL MOUSE; GENETICS; INFLAMMATION; KIDNEY; LIPID DIET; LIVER; MACROPHAGE; MALE; METABOLIC SYNDROME X; METABOLISM; MOUSE; OBESITY; PATHOLOGY; WHITE ADIPOSE TISSUE;

ADIPOSE TISSUE, WHITE; ANIMALS; DIET, HIGH-FAT; INFLAMMATION; KIDNEY; LIVER; MACROPHAGES; MALE; METABOLIC SYNDROME X; MICE; MICE, INBRED C57BL; OBESITY; RECEPTORS, G-PROTEIN-COUPLED; TUMOR NECROSIS FACTOR-ALPHA;

EID: 84973161277     PISSN: None     EISSN: 20452322     Source Type: Journal    
DOI: 10.1038/srep24649     Document Type: Article

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