Dot1l deficiency leads to increased intercalated cells and upregulation of V-ATPase B1 in mice

Experimental Cell Research

Volumn 344, Issue 2, 2016, Pages 167-175

  Xiao, Zhou ^a,b   Chen, Lihe ^c   Zhou, Qiaoling ^a   Zhang, Wenzheng ^b,c,d  

^a CENTRAL SOUTH UNIVERSITY   (China)

^b UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

^c UNIVERSITY OF TEXAS   (United States)

^d ALBANY MEDICAL COLLEGE   (United States)

Author keywords

Conditional knockout; Histone methyltransferase Dot1l; Intercalated cells; Principal cells; V ATPase

Indexed keywords

ADENOSINE TRIPHOSPHATASE; AQUAPORIN 2; HISTONE H3; HISTONE METHYLTRANSFERASE; PENDRIN; UNCLASSIFIED DRUG; V ATPASE B1; AQUAPORIN 3; ATP6V1B1 PROTEIN, MOUSE; DOT1L PROTEIN, MOUSE; ERYTHROCYTE BAND 3 PROTEIN; HISTONE; LYSINE; MESSENGER RNA; METHYLTRANSFERASE; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL DIFFERENTIATION; CELL POPULATION; CHROMATIN IMMUNOPRECIPITATION; CONTROLLED STUDY; DNA FLANKING REGION; DOT1L GENE; ENZYME ACTIVITY; ENZYME DEFICIENCY; EPIGENETICS; GENE; GENE OVEREXPRESSION; IMMUNOFLUORESCENCE; INTERCALATED CELL; KIDNEY CELL; METHYLATION; MOUSE; NONHUMAN; PRINCIPAL CELL; PRIORITY JOURNAL; PROMOTER REGION; TRANSCRIPTION REGULATION; UPREGULATION; ANIMAL; CYTOLOGY; DEFICIENCY; DOWN REGULATION; FLUORESCENT ANTIBODY TECHNIQUE; GENETICS; KIDNEY; METABOLISM;

ANIMALS; ANION EXCHANGE PROTEIN 1, ERYTHROCYTE; AQUAPORIN 2; AQUAPORIN 3; DOWN-REGULATION; FLUORESCENT ANTIBODY TECHNIQUE; HISTONES; KIDNEY; LYSINE; METHYLATION; METHYLTRANSFERASES; MICE; PROMOTER REGIONS, GENETIC; RNA, MESSENGER; UP-REGULATION; VACUOLAR PROTON-TRANSLOCATING ATPASES;

EID: 84966710395     PISSN: 00144827     EISSN: 10902422     Source Type: Journal    
DOI: 10.1016/j.yexcr.2015.09.014     Document Type: Article

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