An Af9 cis-element directly targets Dot1a to mediate transcriptional repression of the αENaC gene

American Journal of Physiology - Renal Physiology

Volumn 304, Issue 4, 2013, Pages

  Zhang, Wenzheng ^a,b   Yu, Zhiyuan ^a   Wu, Hongyu ^a   Chen, Lihe ^b   Kong, Qun ^a   Kone, Bruce C ^a  

^a UNIVERSITY OF TEXAS MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF TEXAS   (United States)

Author keywords

Chromatin; Collecting duct; Gene expression; Transcription; Transcription factor

Indexed keywords

AF9 BINDING PROTEIN; ALDOSTERONE; CIS ACTING ELEMENT; DNA BINDING PROTEIN; DOT1A PROTEIN; EPITHELIAL SODIUM CHANNEL; EPITHELIAL SODIUM CHANNEL SUBUNIT ALPHA; HISTONE METHYLTRANSFERASE; MESSENGER RNA; OLIGOMER; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BINDING AFFINITY; CHROMATIN IMMUNOPRECIPITATION; CONTROLLED STUDY; DNA BINDING; DNA PROTEIN COMPLEX; ENZYME INACTIVATION; ENZYME INHIBITION; GENE DELETION; GENE REPRESSION; GENETIC ASSOCIATION; IMMUNOASSAY; KIDNEY COLLECTING TUBULE; KNOCKOUT GENE; MOUSE; MUTANT; NONHUMAN; POINT MUTATION; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN INTERACTION; PROTEIN TARGETING; TRANSCRIPTION INITIATION; TRANSCRIPTION INITIATION SITE; WILD TYPE;

EID: 84874049969     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.00537.2011     Document Type: Article

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