-
1
-
-
84881180497
-
Trigeminal nerve anatomy in neuropathic and non-neuropathic orofacial pain patients
-
PID: 23685183
-
Wilcox SL, et al. Trigeminal nerve anatomy in neuropathic and non-neuropathic orofacial pain patients. J Pain. 2013;14(8):865–72.
-
(2013)
J Pain
, vol.14
, Issue.8
, pp. 865-872
-
-
Wilcox, S.L.1
-
2
-
-
0034022325
-
Diversity of expression of the sensory neuron-specific TTX-resistant voltage-gated sodium ion channels SNS and SNS2
-
COI: 1:CAS:528:DC%2BD3cXjs1Ontrg%3D, PID: 10845770
-
Amaya F, et al. Diversity of expression of the sensory neuron-specific TTX-resistant voltage-gated sodium ion channels SNS and SNS2. Mol Cell Neurosci. 2000;15(4):331–42.
-
(2000)
Mol Cell Neurosci
, vol.15
, Issue.4
, pp. 331-342
-
-
Amaya, F.1
-
3
-
-
34247479044
-
Expression and localization of the Nav1.9 sodium channel in enteric neurons and in trigeminal sensory endings: implication for intestinal reflex function and orofacial pain
-
COI: 1:CAS:528:DC%2BD2sXkslCnsLY%3D, PID: 17363266
-
Padilla F, et al. Expression and localization of the Nav1.9 sodium channel in enteric neurons and in trigeminal sensory endings: implication for intestinal reflex function and orofacial pain. Mol Cell Neurosci. 2007;35(1):138–52.
-
(2007)
Mol Cell Neurosci
, vol.35
, Issue.1
, pp. 138-152
-
-
Padilla, F.1
-
4
-
-
84865638413
-
+ currents in rat trigeminal ganglion cells
-
COI: 1:CAS:528:DC%2BC38XhtlShtbjJ, PID: 22800565
-
+ currents in rat trigeminal ganglion cells. Neuroscience. 2012;222:205–14.
-
(2012)
Neuroscience
, vol.222
, pp. 205-214
-
-
Scroggs, R.S.1
-
5
-
-
0030041548
-
A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons
-
COI: 1:CAS:528:DyaK28Xmtl2nsg%3D%3D, PID: 8538791
-
Akopian AN, Sivilotti L, Wood JN. A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons. Nature. 1996;379(6562):257–62.
-
(1996)
Nature
, vol.379
, Issue.6562
, pp. 257-262
-
-
Akopian, A.N.1
Sivilotti, L.2
Wood, J.N.3
-
6
-
-
1542498432
-
Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain
-
COI: 1:CAS:528:DC%2BD2cXitFejtrs%3D, PID: 15030943
-
Black JA, et al. Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain. Pain. 2004;108(3):237–47.
-
(2004)
Pain
, vol.108
, Issue.3
, pp. 237-247
-
-
Black, J.A.1
-
7
-
-
33845916809
-
2+ currents, and mechanically activated cation currents in different populations of DRG neurons
-
COI: 1:CAS:528:DC%2BD2sXhtF2qsbc%3D, PID: 17190903
-
2+ currents, and mechanically activated cation currents in different populations of DRG neurons. J Gen Physiol. 2007;129(1):57–77.
-
(2007)
J Gen Physiol
, vol.129
, Issue.1
, pp. 57-77
-
-
Coste, B.1
Crest, M.2
Delmas, P.3
-
8
-
-
0036558150
-
NaN/Nav1.9: a sodium channel with unique properties
-
COI: 1:CAS:528:DC%2BD38XjtVShsL4%3D, PID: 11972962
-
Dib-Hajj S, et al. NaN/Nav1.9: a sodium channel with unique properties. Trends Neurosci. 2002;25(5):253–9.
-
(2002)
Trends Neurosci
, vol.25
, Issue.5
, pp. 253-259
-
-
Dib-Hajj, S.1
-
9
-
-
0032555278
-
NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy
-
COI: 1:CAS:528:DyaK1cXkvFaltLk%3D, PID: 9671787
-
Dib-Hajj SD, et al. NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy. Proc Natl Acad Sci USA. 1998;95(15):8963–8.
-
(1998)
Proc Natl Acad Sci USA
, vol.95
, Issue.15
, pp. 8963-8968
-
-
Dib-Hajj, S.D.1
-
10
-
-
33846863372
-
Effect of 8-bromo-cAMP on the tetrodotoxin-resistant sodium (Nav 1.8) current in small-diameter nodose ganglion neurons
-
COI: 1:CAS:528:DC%2BD2sXhvVyqtLo%3D, PID: 17140607
-
Matsumoto S, et al. Effect of 8-bromo-cAMP on the tetrodotoxin-resistant sodium (Nav 1.8) current in small-diameter nodose ganglion neurons. Neuropharmacology. 2007;52(3):904–24.
-
(2007)
Neuropharmacology
, vol.52
, Issue.3
, pp. 904-924
-
-
Matsumoto, S.1
-
11
-
-
33746223001
-
Intense isolectin-B4 binding in rat dorsal root ganglion neurons distinguishes C-fiber nociceptors with broad action potentials and high Nav1.9 expression
-
COI: 1:CAS:528:DC%2BD28XntFejsbY%3D, PID: 16822986
-
Fang X, et al. Intense isolectin-B4 binding in rat dorsal root ganglion neurons distinguishes C-fiber nociceptors with broad action potentials and high Nav1.9 expression. J Neurosci. 2006;26(27):7281–92.
-
(2006)
J Neurosci
, vol.26
, Issue.27
, pp. 7281-7292
-
-
Fang, X.1
-
12
-
-
78649469347
-
Sodium–calcium exchanger and multiple sodium channel isoforms in intra-epidermal nerve terminals
-
COI: 1:CAS:528:DC%2BC3cXhsFGqur3J, PID: 21118538
-
Persson AK, et al. Sodium–calcium exchanger and multiple sodium channel isoforms in intra-epidermal nerve terminals. Mol Pain. 2010;6:84.
-
(2010)
Mol Pain
, vol.6
, pp. 84
-
-
Persson, A.K.1
-
13
-
-
0036895970
-
2+ current in the action potentials of nociceptive sensory neurons
-
COI: 1:CAS:528:DC%2BD38XpsVamsLg%3D, PID: 12451128
-
2+ current in the action potentials of nociceptive sensory neurons. J Neurosci. 2002;22(23):10277–90.
-
(2002)
J Neurosci
, vol.22
, Issue.23
, pp. 10277-10290
-
-
Blair, N.T.1
Bean, B.P.2
-
14
-
-
0034554778
-
Glial-derived neurotrophic factor upregulates expression of functional SNS and NaN sodium channels and their currents in axotomized dorsal root ganglion neurons
-
COI: 1:CAS:528:DC%2BD3MXpslSg, PID: 11102483
-
Cummins TR, et al. Glial-derived neurotrophic factor upregulates expression of functional SNS and NaN sodium channels and their currents in axotomized dorsal root ganglion neurons. J Neurosci. 2000;20(23):8754–61.
-
(2000)
J Neurosci
, vol.20
, Issue.23
, pp. 8754-8761
-
-
Cummins, T.R.1
-
15
-
-
84858001496
-
NaV 1.8, but not NaV 1.9, is upregulated in the inflamed dental pulp tissue of human primary teeth
-
COI: 1:STN:280:DC%2BC383osFKrtg%3D%3D, PID: 22085016
-
Suwanchai A, et al. NaV 1.8, but not NaV 1.9, is upregulated in the inflamed dental pulp tissue of human primary teeth. Int Endod J. 2012;45(4):372–8.
-
(2012)
Int Endod J
, vol.45
, Issue.4
, pp. 372-378
-
-
Suwanchai, A.1
-
16
-
-
40849083674
-
Inflammatory mediators increase Nav1.9 current and excitability in nociceptors through a coincident detection mechanism
-
COI: 1:CAS:528:DC%2BD1cXktVKmsLw%3D, PID: 18270172
-
Maingret F, et al. Inflammatory mediators increase Nav1.9 current and excitability in nociceptors through a coincident detection mechanism. J Gen Physiol. 2008;131(3):211–25.
-
(2008)
J Gen Physiol
, vol.131
, Issue.3
, pp. 211-225
-
-
Maingret, F.1
-
17
-
-
84899477858
-
Specialized functions of Nav1.5 and Nav1.9 channels in electrogenesis of myenteric neurons in intact mouse ganglia
-
PID: 24719102
-
Osorio N, Korogod S, Delmas P. Specialized functions of Nav1.5 and Nav1.9 channels in electrogenesis of myenteric neurons in intact mouse ganglia. J Neurosci. 2014;34(15):5233–44.
-
(2014)
J Neurosci
, vol.34
, Issue.15
, pp. 5233-5244
-
-
Osorio, N.1
Korogod, S.2
Delmas, P.3
-
18
-
-
2342589514
-
Gating and modulation of presumptive NaV1.9 channels in enteric and spinal sensory neurons
-
COI: 1:CAS:528:DC%2BD2cXjs1Chur0%3D, PID: 15121184
-
Coste B, et al. Gating and modulation of presumptive NaV1.9 channels in enteric and spinal sensory neurons. Mol Cell Neurosci. 2004;26(1):123–34.
-
(2004)
Mol Cell Neurosci
, vol.26
, Issue.1
, pp. 123-134
-
-
Coste, B.1
-
19
-
-
0033572137
-
A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons
-
COI: 1:STN:280:DC%2BD3cvjsFGluw%3D%3D, PID: 10594087
-
Cummins TR, et al. A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons. J Neurosci. 1999;19(24):RC43.
-
(1999)
J Neurosci
, vol.19
, Issue.24
, pp. RC43
-
-
Cummins, T.R.1
-
20
-
-
8644230852
-
+ channel, Na(v)1.9, in mouse dorsal root ganglion neurons
-
COI: 1:CAS:528:DC%2BD2cXotVyrur8%3D, PID: 15290301
-
+ channel, Na(v)1.9, in mouse dorsal root ganglion neurons. Pflugers Arch. 2004;449(1):76–87.
-
(2004)
Pflugers Arch
, vol.449
, Issue.1
, pp. 76-87
-
-
Maruyama, H.1
-
21
-
-
80051602755
-
Nav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation
-
COI: 1:CAS:528:DC%2BC3MXhtFGhtLrF, PID: 21857998
-
Lolignier S, et al. Nav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation. PLoS One. 2011;6(8):e23083.
-
(2011)
PLoS One
, vol.6
, Issue.8
, pp. e23083
-
-
Lolignier, S.1
-
22
-
-
21544455502
-
Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behavior
-
COI: 1:CAS:528:DC%2BD2MXlvF2rsLg%3D, PID: 15964986
-
Priest BT, et al. Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behavior. Proc Natl Acad Sci USA. 2005;102(26):9382–7.
-
(2005)
Proc Natl Acad Sci USA
, vol.102
, Issue.26
, pp. 9382-9387
-
-
Priest, B.T.1
-
23
-
-
33748888226
-
Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice
-
COI: 1:CAS:528:DC%2BD28XhtFSisbjO, PID: 16857712
-
Hillsley K, et al. Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice. J Physiol. 2006;576(Pt 1):257–67.
-
(2006)
J Physiol
, vol.576
, pp. 257-267
-
-
Hillsley, K.1
-
24
-
-
84893371465
-
Pain without nociceptors? Nav1.7-independent pain mechanisms
-
COI: 1:CAS:528:DC%2BC2cXhs1GrsLw%3D, PID: 24440715
-
Minett MS, et al. Pain without nociceptors? Nav1.7-independent pain mechanisms. Cell Rep. 2014;6(2):301–12.
-
(2014)
Cell Rep
, vol.6
, Issue.2
, pp. 301-312
-
-
Minett, M.S.1
-
25
-
-
84901436064
-
Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy
-
PID: 24776970
-
Huang J, et al. Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy. Brain. 2014;137(Pt 6):1627–42.
-
(2014)
Brain
, vol.137
, pp. 1627-1642
-
-
Huang, J.1
-
26
-
-
84887045358
-
A de novo gain-of-function mutation in SCN11A causes loss of pain perception
-
COI: 1:CAS:528:DC%2BC3sXhsVeqs7%2FO, PID: 24036948
-
Leipold E, et al. A de novo gain-of-function mutation in SCN11A causes loss of pain perception. Nat Genet. 2013;45(11):1399–404.
-
(2013)
Nat Genet
, vol.45
, Issue.11
, pp. 1399-1404
-
-
Leipold, E.1
-
27
-
-
84930635038
-
Contribution and interaction of kinin receptors and dynorphin A in a model of trigeminal neuropathic pain in mice
-
COI: 1:CAS:528:DC%2BC2MXos1Oltb4%3D, PID: 25982562
-
Luiz AP, et al. Contribution and interaction of kinin receptors and dynorphin A in a model of trigeminal neuropathic pain in mice. Neuroscience. 2015;300:189–200.
-
(2015)
Neuroscience
, vol.300
, pp. 189-200
-
-
Luiz, A.P.1
-
28
-
-
77049108399
-
Kinin B(1) and B(2) receptors contribute to orofacial heat hyperalgesia induced by infraorbital nerve constriction injury in mice and rats
-
COI: 1:CAS:528:DC%2BC3cXis1SgsL4%3D, PID: 19914714
-
Luiz AP, et al. Kinin B(1) and B(2) receptors contribute to orofacial heat hyperalgesia induced by infraorbital nerve constriction injury in mice and rats. Neuropeptides. 2010;44(2):87–92.
-
(2010)
Neuropeptides
, vol.44
, Issue.2
, pp. 87-92
-
-
Luiz, A.P.1
-
29
-
-
84890177829
-
Gain-of-function mutations in SCN11A cause familial episodic pain
-
COI: 1:CAS:528:DC%2BC3sXhs12ltrvF, PID: 24207120
-
Zhang XY, et al. Gain-of-function mutations in SCN11A cause familial episodic pain. Am J Hum Genet. 2013;93(5):957–66.
-
(2013)
Am J Hum Genet
, vol.93
, Issue.5
, pp. 957-966
-
-
Zhang, X.Y.1
-
30
-
-
33845631301
-
The voltage-gated sodium channel Na(v)1.9 is an effector of peripheral inflammatory pain hypersensitivity
-
COI: 1:CAS:528:DC%2BD2sXhs1Ciug%3D%3D, PID: 17167076
-
Amaya F, et al. The voltage-gated sodium channel Na(v)1.9 is an effector of peripheral inflammatory pain hypersensitivity. J Neurosci. 2006;26(50):12852–60.
-
(2006)
J Neurosci
, vol.26
, Issue.50
, pp. 12852-12860
-
-
Amaya, F.1
-
31
-
-
77951911566
-
Exploring the role of nociceptor-specific sodium channels in pain transmission using Nav1.8 and Nav1.9 knockout mice
-
COI: 1:CAS:528:DC%2BC3cXhs1alsbg%3D, PID: 19931571
-
Leo S, D’Hooge R, Meert T. Exploring the role of nociceptor-specific sodium channels in pain transmission using Nav1.8 and Nav1.9 knockout mice. Behav Brain Res. 2010;208(1):149–57.
-
(2010)
Behav Brain Res
, vol.208
, Issue.1
, pp. 149-157
-
-
Leo, S.1
D’Hooge, R.2
Meert, T.3
-
32
-
-
84887022105
-
Behavioral testing in rodent models of orofacial neuropathic and inflammatory pain
-
PID: 23139912
-
Krzyzanowska A, Avendano C. Behavioral testing in rodent models of orofacial neuropathic and inflammatory pain. Brain Behav. 2012;2(5):678–97.
-
(2012)
Brain Behav
, vol.2
, Issue.5
, pp. 678-697
-
-
Krzyzanowska, A.1
Avendano, C.2
-
33
-
-
0037731500
-
GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones
-
COI: 1:CAS:528:DC%2BD3sXktlCjur4%3D, PID: 12651922
-
Baker MD, et al. GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones. J Physiol. 2003;548(Pt 2):373–82.
-
(2003)
J Physiol
, vol.548
, pp. 373-382
-
-
Baker, M.D.1
-
34
-
-
39449113609
-
+ current and enhanced nociceptor excitability require NaV1.9
-
COI: 1:CAS:528:DC%2BD1cXivFaqu7w%3D, PID: 18096591
-
+ current and enhanced nociceptor excitability require NaV1.9. J Physiol. 2008;586(4):1077–87.
-
(2008)
J Physiol
, vol.586
, Issue.4
, pp. 1077-1087
-
-
Ostman, J.A.1
-
35
-
-
0028325885
-
Behavioral evidence of trigeminal neuropathic pain following chronic constriction injury to the rat’s infraorbital nerve
-
COI: 1:STN:280:DyaK2c3jvVeguw%3D%3D, PID: 8182437
-
Vos BP, Strassman AM, Maciewicz RJ. Behavioral evidence of trigeminal neuropathic pain following chronic constriction injury to the rat’s infraorbital nerve. J Neurosci. 1994;14(5 Pt 1):2708–23.
-
(1994)
J Neurosci
, vol.14
, Issue.5 Part 1
, pp. 2708-2723
-
-
Vos, B.P.1
Strassman, A.M.2
Maciewicz, R.J.3
-
36
-
-
1842430764
-
Afferent pain pathways: a neuroanatomical review
-
COI: 1:CAS:528:DC%2BD2cXisF2htro%3D, PID: 15053950
-
Almeida TF, Roizenblatt S, Tufik S. Afferent pain pathways: a neuroanatomical review. Brain Res. 2004;1000(1–2):40–56.
-
(2004)
Brain Res
, vol.1000
, Issue.1-2
, pp. 40-56
-
-
Almeida, T.F.1
Roizenblatt, S.2
Tufik, S.3
-
37
-
-
0029903272
-
Saporin, a ribosome-inactivating protein used to prepare immunotoxins, induces cell death via apoptosis
-
COI: 1:CAS:528:DyaK28XkslahsLs%3D, PID: 8703804
-
Bergamaschi G, et al. Saporin, a ribosome-inactivating protein used to prepare immunotoxins, induces cell death via apoptosis. Br J Haematol. 1996;93(4):789–94.
-
(1996)
Br J Haematol
, vol.93
, Issue.4
, pp. 789-794
-
-
Bergamaschi, G.1
|