Retraction Note: Overexpression of miR-499-5p inhibits non-small cell lung cancer proliferation and metastasis by targeting VAV3 (Scientific Reports, (2016), 10.1038/srep23100);Overexpression of miR-499-5p inhibits non-small cell lung cancer proliferation and metastasis by targeting VAV3

Scientific Reports

Volumn 6, Issue , 2016, Pages

  Li, Ming ^a   Zhang, Shenjun ^b   Wu, Ning ^c   Wu, Liang ^a   Wang, Changhui ^a   Lin, Yinping ^a,b  

^a TONGJI UNIVERSITY   (China)

^b INSTITUTE OF GEOLOGY AND GEOPHYSICS   (China)

^c FUDAN UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

3' UNTRANSLATED REGION; MICRORNA; MIRN499 MICRORNA, HUMAN; VAV PROTEIN; VAV3 PROTEIN, HUMAN;

3' UNTRANSLATED REGION; A-549 CELL LINE; ANIMAL; APOPTOSIS; CELL CYCLE; CELL LINE; CELL PROLIFERATION; GENE EXPRESSION REGULATION; GENETICS; LUNG TUMOR; METABOLISM; METASTASIS; NON SMALL CELL LUNG CANCER; NUCLEOTIDE SEQUENCE; NUDE MOUSE; PATHOLOGY; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SEQUENCE HOMOLOGY; TUMOR CELL LINE; WESTERN BLOTTING; XENOGRAFT;

3' UNTRANSLATED REGIONS; A549 CELLS; ANIMALS; APOPTOSIS; BASE SEQUENCE; BLOTTING, WESTERN; CARCINOMA, NON-SMALL-CELL LUNG; CELL CYCLE; CELL LINE; CELL LINE, TUMOR; CELL PROLIFERATION; GENE EXPRESSION REGULATION, NEOPLASTIC; LUNG NEOPLASMS; MICE, NUDE; MICRORNAS; NEOPLASM METASTASIS; PROTO-ONCOGENE PROTEINS C-VAV; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; SEQUENCE HOMOLOGY, NUCLEIC ACID; TRANSPLANTATION, HETEROLOGOUS;

EID: 84961184524     PISSN: None     EISSN: 20452322     Source Type: Journal    
DOI: 10.1038/s41598-021-96814-6     Document Type: Erratum

References (24)

1. Siegel, R.; Ma, J.; Zou, Z. & Jemal, A. Cancer statistics, 2014. CA Cancer J Clin 64, 9-29 (2014)
2. Li, M. et al. Pemetrexed plus platinum as the first-line treatment option for advanced non-small cell lung cancer: a meta-analysis of randomized controlled trials. PLoS One 7, e37229 (2012)
3. American Cancer Society, Cancer Facts & Figures 2014, Atlanta, Georgia, 2014. [Online]. Available: http://www. cancer. org/ research/cancerfactsstatistics/cancerfactsfigures2014/. [Accessed 14 May 2015]
4. Bartel, D. P. MicroRNAs: genomics, biogenesis, mechanism, and function. Cell 116, 281-297 (2004)
5. Bartel, D. P. MicroRNAs: target recognition and regulatory functions. Cell 136, 215-233 (2009)
6. Kong, Y. W.; Ferland-McCollough, D.; Jackson, T. J. & Bushell, M. microRNAs in cancer management. Lancet Oncol 13, e249-258 (2012)
7. Hu, Z. et al. Genetic variants of miRNA sequences and non-small cell lung cancer survival. J Clin Invest 118, 2600-2608 (2008)
8. Esquela-Kerscher, A. & Slack, F. J. Oncomirs-microRNAs with a role in cancer. Nat Rev Cancer 6, 259-269 (2006)
9. Liu, X. et al. MicroRNA-499-5p promotes cellular invasion and tumor metastasis in colorectal cancer by targeting FOXO4 and PDCD4. Carcinogenesis 32, 1798-1805 (2011)
10. Hu, Z. et al. Serum microRNA signatures identified in a genome-wide serum microRNA expression profiling predict survival of non-small-cell lung cancer. J Clin Oncol 28, 1721-1726 (2010)
11. Li, M. et al. Serum miR-499 as a novel diagnostic and prognostic biomarker in non-small cell lung cancer. Oncol Rep 31, 1961-1967 (2014)
12. Sozzi, G.; Pastorino, U. & Croce, C. M. MicroRNAs and lung cancer: from markers to targets. Cell Cycle 10, 2045-2046 (2011)
13. Olivieri, F. et al. Diagnostic potential of circulating miR-499-5p in elderly patients with acute non ST-elevation myocardial infarction. Int J Cardiol 167, 531-536 (2013)
14. Movilla, N. & Bustelo, X. R. Biological and regulatory properties of Vav-3, a new member of the Vav family of oncoproteins. Mol Cell Biol 19, 7870-7885 (1999)
15. Katzav, S.; Martin-Zanca, D. & Barbacid, M. vav, a novel human oncogene derived from a locus ubiquitously expressed in hematopoietic cells. EMBO J 8, 2283-2290 (1989)
16. Citterio, C. et al. The rho exchange factors vav2 and vav3 control a lung metastasis-specific transcriptional program in breast cancer cells. Sci Signal 5, ra71 (2012)
17. Lyons, L. S. & Burnstein, K. L. Vav3, a Rho GTPase guanine nucleotide exchange factor, increases during progression to androgen independence in prostate cancer cells and potentiates androgen receptor transcriptional activity. Mol Endocrinol 20, 1061-1072 (2006)
18. Marques, R. B.; Dits, N. F.; Erkens-Schulze, S.; van Weerden, W. M. & Jenster, G. Bypass mechanisms of the androgen receptor pathway in therapy-resistant prostate cancer cell models. PLoS One 5, e13500 (2010)
19. Peacock, S. O.; Fahrenholtz, C. D. & Burnstein, K. L. Vav3 enhances androgen receptor splice variant activity and is critical for castration-resistant prostate cancer growth and survival. Mol Endocrinol 26, 1967-1979 (2012)
20. Lee, K. et al. Vav3 oncogene activates estrogen receptor and its overexpression may be involved in human breast cancer. BMC Cancer 8, 158 (2008)
21. Lazer, G.; Idelchuk, Y.; Schapira, V.; Pikarsky, E. & Katzav, S. The haematopoietic specific signal transducer Vav1 is aberrantly expressed in lung cancer and plays a role in tumourigenesis. J Pathol 219, 25-34 (2009)
22. Di Leva, G.; Garofalo, M. & Croce, C. M. MicroRNAs in cancer. Annu Rev Pathol 9, 287-314 (2014)
23. Ha, M. & Kim, V. N. Regulation of microRNA biogenesis. Nat Rev Mol Cell Biol 15, 509-524 (2014)
24. Lin, K. T. et al. Vav3-rac1 signaling regulates prostate cancer metastasis with elevated Vav3 expression correlating with prostate cancer progression and posttreatment recurrence. Cancer Res 72, 3000-3009 (2012)