Impaired mitochondrial network excitability in failing Guinea-pig cardiomyocytes

Cardiovascular Research

Volumn 109, Issue 1, 2016, Pages 79-89

  Goh, Kah Yong ^a,b   Qu, Jing ^a,b   Hong, Huixian ^a,b   Liu, Ting ^c   Dell'italia, Louis J ^a,b   Wu, Yong ^d   O'rourke, Brian ^c   Zhou, Lufang ^a,b,e  

^a University of Alabama at Birmingham   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^c JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

^e and Radiation Oncology   (United States)

Author keywords

Excitability; Heart failure; Mitochondrial network; Mitofusin protein; ROS induced ROS release

Indexed keywords

GLUTATHIONE; HYBRID PROTEIN; MITOFUSIN 1; MITOFUSIN 2; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE;

ADULT; ANIMAL CELL; ANIMAL MODEL; ARTICLE; CELL ULTRASTRUCTURE; DEPOLARIZATION; GENE EXPRESSION; GUINEA PIG; HEART FAILURE; HEART MUSCLE CELL; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; NONHUMAN; OSCILLATION; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; ANIMAL; CARDIAC MUSCLE CELL; METABOLISM; PATHOPHYSIOLOGY; PHYSIOLOGY; ULTRASTRUCTURE;

ANIMALS; GUINEA PIGS; HEART FAILURE; MITOCHONDRIA; MYOCYTES, CARDIAC; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES;

EID: 84959903452     PISSN: 00086363     EISSN: 17553245     Source Type: Journal    
DOI: 10.1093/cvr/cvv230     Document Type: Article

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