-
1
-
-
84864279572
-
Cell death: Multitasking p53 promotes necrosis
-
Baumann, K. (2012). Cell death: multitasking p53 promotes necrosis. Nat Rev Mol Cell Biol 13, 480-481.
-
(2012)
Nat Rev Mol Cell Biol
, vol.13
, pp. 480-481
-
-
Baumann, K.1
-
2
-
-
79958244812
-
Activation of AMP-activated protein kinase contributes to doxorubicin-induced cell death and apoptosis in cultured myocardial H9c2 cells
-
Chen, M. B., et al. (2011). Activation of AMP-activated protein kinase contributes to doxorubicin-induced cell death and apoptosis in cultured myocardial H9c2 cells. Cell Biochem Biophys 60, 311-322.
-
(2011)
Cell Biochem Biophys
, vol.60
, pp. 311-322
-
-
Chen, M.B.1
-
3
-
-
84875672536
-
TAK1activates AMPK-dependent cell death pathway in hydrogen peroxide-treated cardiomyocytes, inhibited by heat shock protein-70
-
Chen, Z., et al. (2013). TAK1activates AMPK-dependent cell death pathway in hydrogen peroxide-treated cardiomyocytes, inhibited by heat shock protein-70. Mol Cell Biochem 377, 35-44.
-
(2013)
Mol Cell Biochem
, vol.377
, pp. 35-44
-
-
Chen, Z.1
-
4
-
-
78649403525
-
SKI-II, an inhibitor of sphingosine kinase, ameliorates antigen-induced bronchial smooth muscle hyperresponsiveness, but not airway inflammation, in mice
-
Chiba, Y., et al. (2010). SKI-II, an inhibitor of sphingosine kinase, ameliorates antigen-induced bronchial smooth muscle hyperresponsiveness, but not airway inflammation, in mice. J Pharmacol Sci 114, 304-310.
-
(2010)
J Pharmacol Sci
, vol.114
, pp. 304-310
-
-
Chiba, Y.1
-
5
-
-
0033539994
-
ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells
-
Ekhterae, D., et al. (1999). ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells. Circ Res 85, e70-e77.
-
(1999)
Circ Res
, vol.85
, pp. e70-e77
-
-
Ekhterae, D.1
-
6
-
-
45449120119
-
K6PC-5 a direct activator of sphingosine kinase 1, promotes epidermal differentiation through intracellular Ca2+ signaling
-
Hong, J. H., et al. (2008). K6PC-5, a direct activator of sphingosine kinase 1, promotes epidermal differentiation through intracellular Ca2+ signaling. J Invest Dermatol 128, 2166-2178.
-
(2008)
J Invest Dermatol
, vol.128
, pp. 2166-2178
-
-
Hong, J.H.1
-
7
-
-
0025815672
-
Endothelin-1 induces hypertrophy with enhanced expression of muscle-specific genes in cultured neonatal rat cardiomyocytes
-
Ito, H., et al. (1991). Endothelin-1 induces hypertrophy with enhanced expression of muscle-specific genes in cultured neonatal rat cardiomyocytes. Circ Res 69, 209-215.
-
(1991)
Circ Res
, vol.69
, pp. 209-215
-
-
Ito, H.1
-
8
-
-
84929131732
-
K6PC-5 a novel sphingosine kinase 1 (SphK1) activator, alleviates dexamethasone-induced damages to osteoblasts through activating SphK1-Akt signaling
-
Ji, F., et al. (2015). K6PC-5, a novel sphingosine kinase 1 (SphK1) activator, alleviates dexamethasone-induced damages to osteoblasts through activating SphK1-Akt signaling. Biochem Biophys Res Commun 458, 568-575.
-
(2015)
Biochem Biophys Res Commun
, vol.458
, pp. 568-575
-
-
Ji, F.1
-
9
-
-
0036083489
-
Cardioprotectionmediated by sphingosine-1-phosphate and ganglioside GM-1 in wild-type and PKC epsilon knockout mouse hearts
-
Jin, Z. Q., et al. (2002). Cardioprotectionmediated by sphingosine-1-phosphate and ganglioside GM-1 in wild-type and PKC epsilon knockout mouse hearts. Am J Physiol Heart Circ Physiol 282, H1970-H1977.
-
(2002)
Am J Physiol Heart Circ Physiol
, vol.282
, pp. H1970-H1977
-
-
Jin, Z.Q.1
-
10
-
-
84868686651
-
Is p53 the long-sought molecular trigger for cyclophilin D-regulated mitochondrial permeability transition pore formation and necrosis?
-
Karch, J., et al. (2012). Is p53 the long-sought molecular trigger for cyclophilin D-regulated mitochondrial permeability transition pore formation and necrosis? Circ Res 111, 1258-1260.
-
(2012)
Circ Res
, vol.111
, pp. 1258-1260
-
-
Karch, J.1
-
11
-
-
0034856123
-
The lysophospholipids sphingosine-1-phosphate and lysophosphatidic acid enhance survival during hypoxia in neonatal rat cardiac myocytes
-
Karliner, J. S., et al. (2001). The lysophospholipids sphingosine-1-phosphate and lysophosphatidic acid enhance survival during hypoxia in neonatal rat cardiac myocytes. J Mol Cell Cardiol 33, 1713-1717.
-
(2001)
J Mol Cell Cardiol
, vol.33
, pp. 1713-1717
-
-
Karliner, J.S.1
-
12
-
-
0036173656
-
Inhibition of recombinant sphingosine kinases by novel inhibitors of microbial origin, F-12509A and B-5354c
-
Kono, K., et al. (2002). Inhibition of recombinant sphingosine kinases by novel inhibitors of microbial origin, F-12509A and B-5354c. J Antibiot (Tokyo) 55, 99-103.
-
(2002)
J Antibiot (Tokyo)
, vol.55
, pp. 99-103
-
-
Kono, K.1
-
13
-
-
34548384398
-
Novel synthetic ceramide derivatives increase intracellular calcium levels and promote epidermal keratinocyte differentiation
-
Kwon, Y. B., et al. (2007). Novel synthetic ceramide derivatives increase intracellular calcium levels and promote epidermal keratinocyte differentiation. J Lipid Res 48, 1936-1943.
-
(2007)
J Lipid Res
, vol.48
, pp. 1936-1943
-
-
Kwon, Y.B.1
-
14
-
-
84921756222
-
A sphingosine kinase-1 inhibitor, SKI-II, induces growth inhibition and apoptosis in human gastric cancer cells
-
Li, P. H., et al. (2014). A sphingosine kinase-1 inhibitor, SKI-II, induces growth inhibition and apoptosis in human gastric cancer cells. Asian Pac J Cancer Prev 15, 10381-10385.
-
(2014)
Asian Pac J Cancer Prev
, vol.15
, pp. 10381-10385
-
-
Li, P.H.1
-
15
-
-
77952241179
-
Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes
-
Marambio, P., et al. (2010). Glucose deprivation causes oxidative stress and stimulates aggresome formation and autophagy in cultured cardiac myocytes. Biochim Biophys Acta 1802, 509-518.
-
(2010)
Biochim Biophys Acta
, vol.1802
, pp. 509-518
-
-
Marambio, P.1
-
16
-
-
84860507660
-
Ceramide and apoptosis: Exploring the enigmatic connections between sphingolipid metabolism and programmed cell death
-
Mullen, T. D., et al. (2012). Ceramide and apoptosis: exploring the enigmatic connections between sphingolipid metabolism and programmed cell death. Anticancer Agents Med Chem12, 340-363.
-
(2012)
Anticancer Agents Med Chem
, vol.12
, pp. 340-363
-
-
Mullen, T.D.1
-
17
-
-
84855718052
-
A tale of coronary artery disease and myocardial infarction
-
Nabel, E. G., et al. (2012). A tale of coronary artery disease and myocardial infarction. N Engl J Med 366, 54-63.
-
(2012)
N Engl J Med
, vol.366
, pp. 54-63
-
-
Nabel, E.G.1
-
18
-
-
0034070228
-
Protective effect of estrogens against oxidative damage to heart and skeletal muscle in vivo and in vitro
-
Persky, A. M., et al. (2000). Protective effect of estrogens against oxidative damage to heart and skeletal muscle in vivo and in vitro. Proc Soc Exp Biol Med 223, 59-66.
-
(2000)
Proc Soc Exp Biol Med
, vol.223
, pp. 59-66
-
-
Persky, A.M.1
-
19
-
-
52449092057
-
Targeting SphK1 as a new strategy against cancer
-
Shida, D., et al. (2008). Targeting SphK1 as a new strategy against cancer. Curr Drug Targets 9, 662-673.
-
(2008)
Curr Drug Targets
, vol.9
, pp. 662-673
-
-
Shida, D.1
-
20
-
-
0030008542
-
Cytokines and insulin induce cationic amino acid transporter (CAT) expression in cardiac myocytes. Regulation of l-arginine transport and no production by cat-1 cat-2a, and cat-2b
-
Simmons, W. W., et al. (1996). Cytokines and insulin induce cationic amino acid transporter (CAT) expression in cardiac myocytes. Regulation of L-arginine transport and no production by CAT-1, CAT-2A, and CAT-2B. J Biol Chem 271, 11694-11702.
-
(1996)
J Biol Chem
, vol.271
, pp. 11694-11702
-
-
Simmons, W.W.1
-
21
-
-
84861483434
-
Stress and cardiovascular disease
-
Steptoe, A., et al. (2012). Stress and cardiovascular disease. Nat Rev Cardiol 9, 360-370.
-
(2012)
Nat Rev Cardiol
, vol.9
, pp. 360-370
-
-
Steptoe, A.1
-
22
-
-
51749100808
-
The role of sphingosine kinase 1 in cancer: Oncogene or non-oncogene addiction?
-
Vadas, M., et al. (2008). The role of sphingosine kinase 1 in cancer: oncogene or non-oncogene addiction? Biochim Biophys Acta 1781, 442-447.
-
(2008)
Biochim Biophys Acta
, vol.1781
, pp. 442-447
-
-
Vadas, M.1
-
23
-
-
84862675016
-
P53 opens the mitochondrial permeability transition pore to trigger necrosis
-
Vaseva, A. V., et al. (2012). p53 opens the mitochondrial permeability transition pore to trigger necrosis. Cell 149, 1536-1548.
-
(2012)
Cell
, vol.149
, pp. 1536-1548
-
-
Vaseva, A.V.1
-
24
-
-
1342346691
-
Cardiac physiology at the cellular level: Use of cultured HL-1 cardiomyocytes for studies of cardiac muscle cell structure and function
-
White, S. M., et al. (2004). Cardiac physiology at the cellular level: use of cultured HL-1 cardiomyocytes for studies of cardiac muscle cell structure and function. Am J Physiol Heart Circ Physiol 286, H823-H829.
-
(2004)
Am J Physiol Heart Circ Physiol
, vol.286
, pp. H823-H829
-
-
White, S.M.1
-
25
-
-
84864292058
-
Co-administration phenoxodiol with doxorubicin synergistically inhibit the activity of sphingosine kinase-1 (SphK1), a potential oncogene of osteosarcoma, to suppress osteosarcoma cell growth both in vivo and in vitro
-
Yao, C., et al. (2012). Co-administration phenoxodiol with doxorubicin synergistically inhibit the activity of sphingosine kinase-1 (SphK1), a potential oncogene of osteosarcoma, to suppress osteosarcoma cell growth both in vivo and in vitro. Mol Oncol 6, 392-404.
-
(2012)
Mol Oncol
, vol.6
, pp. 392-404
-
-
Yao, C.1
-
26
-
-
84903461276
-
P53 dependent mitochondrial permeability transition pore opening is required for dexamethasoneinduced death of osteoblasts
-
Zhen, Y. F., et al. (2014). P53 dependent mitochondrial permeability transition pore opening is required for dexamethasoneinduced death of osteoblasts. J Cell Physiol 229, 1475-1483.
-
(2014)
J Cell Physiol
, vol.229
, pp. 1475-1483
-
-
Zhen, Y.F.1
-
27
-
-
84906283880
-
Salidroside inhibits oxygen glucose deprivation (OGD)/re-oxygenation-induced H9c2 cell necrosis through activating ofAkt-Nrf2 signaling
-
Zheng, K., et al. (2014). Salidroside inhibits oxygen glucose deprivation (OGD)/re-oxygenation-induced H9c2 cell necrosis through activating ofAkt-Nrf2 signaling. BiochemBiophys Res Commun 451, 79-85.
-
(2014)
BiochemBiophys Res Commun
, vol.451
, pp. 79-85
-
-
Zheng, K.1
|