|
Volumn 3, Issue 10, 2015, Pages 737-739
|
Elucidating COPD pathogenesis by large-scale genetic analyses
|
Author keywords
[No Author keywords available]
|
Indexed keywords
HISTONE DEACETYLASE 2;
HISTONE DEACETYLASE 4;
NICOTINIC RECEPTOR;
BRAIN TISSUE;
CD4+ T LYMPHOCYTE;
CHROMATIN;
CHROMOSOME 11;
CHROMOSOME 2;
CHRONIC OBSTRUCTIVE LUNG DISEASE;
DNA METHYLATION;
EPIGENETICS;
FORCED EXPIRATORY VOLUME;
GENE;
GENE EXPRESSION;
GENE FUNCTION;
GENE LOCUS;
GENETIC ANALYSIS;
GENETIC ASSOCIATION;
GENETIC RISK;
GENETIC SUSCEPTIBILITY;
GENETIC VARIABILITY;
GENOTYPE;
HISTONE ACETYLATION;
HUMAN;
KANSL1 GENE;
LUNG FUNCTION;
NCAM1 GENE;
NOTE;
PATHOGENESIS;
PRIORITY JOURNAL;
SINGLE NUCLEOTIDE POLYMORPHISM;
SMOKING;
TET2 GENE;
TH1 CELL;
TH17 CELL;
TOBACCO DEPENDENCE;
FEMALE;
GENETICS;
LUNG;
MALE;
PATHOPHYSIOLOGY;
FEMALE;
HUMANS;
LUNG;
MALE;
PULMONARY DISEASE, CHRONIC OBSTRUCTIVE;
SMOKING;
|
EID: 84943415424
PISSN: 22132600
EISSN: 22132619
Source Type: Journal
DOI: 10.1016/S2213-2600(15)00360-4 Document Type: Note |
Times cited : (7)
|
References (8)
|