The role of TGFBI (βig-H3) in gastrointestinal tract tumorigenesis

Molecular Cancer

Volumn 14, Issue 1, 2015, Pages

  Han, Bing ^a   Cai, Haolei ^b   Chen, Ying ^b   Hu, Bing ^c   Luo, Hongyu ^a   Wu, Yulian ^b   Wu, Jiangping ^a  

^a CENTRE HOSPITALIER DE L UNIVERSITÉ DE MONTRÉAL   (Canada)

^b THE SECOND AFFILIATED HOSPITAL OF ZHEJIANG UNIVERSITY SCHOOL OF MEDICINE   (China)

^c AmeriPath Central Florida Orlando   (United States)

Author keywords

Carcinogenesis; Gastrointestinal tract tumors; Gene knockout mice; Patient serum TGFBI levels; Transgenic mice; ig H3 TGFBI

Indexed keywords

DIETHYLNITROSAMINE; DNA; FOCAL ADHESION KINASE; MAMMALIAN TARGET OF RAPAMYCIN; PROTEIN AKT1S1; PROTEIN EIF4EBP; PROTEIN KINASE B; PROTEIN PRS6; TRANSFORMING GROWTH FACTOR BETA1; UNCLASSIFIED DRUG; BETAIG-H3 PROTEIN; CARCINOGEN; SCLEROPROTEIN; TRANSFORMING GROWTH FACTOR BETA;

AGED; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BILE DUCT CARCINOMA; CANCER INCIDENCE; CANCER RISK; CARCINOGENESIS; CELL PROLIFERATION; CELL SURVIVAL; CONTROLLED STUDY; DNA DAMAGE; ENZYME LINKED IMMUNOSORBENT ASSAY; GASTROINTESTINAL TUMOR; HUMAN; HUMAN TISSUE; IRRADIATION; KNOCKOUT MOUSE; LIVER CELL CARCINOMA; LIVER TUMOR; MAJOR CLINICAL STUDY; MALE; MOLECULAR MECHANICS; MOUSE; NONHUMAN; PROMOTER REGION; PROTEIN BLOOD LEVEL; PROTEIN EXPRESSION; PROTEIN FUNCTION; SIGNAL TRANSDUCTION; STOMACH CARCINOMA; TRANSGENIC MOUSE; WILD TYPE MOUSE; ANIMAL; APOPTOSIS; BLOOD; C57BL MOUSE; GASTROINTESTINAL TRACT; METABOLISM; PATHOLOGY; PHOSPHORYLATION;

ANIMALS; APOPTOSIS; CARCINOGENESIS; CARCINOGENS; CELL PROLIFERATION; CELL SURVIVAL; DNA DAMAGE; EXTRACELLULAR MATRIX PROTEINS; GASTROINTESTINAL NEOPLASMS; GASTROINTESTINAL TRACT; HUMANS; LIVER NEOPLASMS; MICE, INBRED C57BL; MICE, KNOCKOUT; PHOSPHORYLATION; SIGNAL TRANSDUCTION; TRANSFORMING GROWTH FACTOR BETA;

EID: 84929439428     PISSN: None     EISSN: 14764598     Source Type: Journal    
DOI: 10.1186/s12943-015-0335-z     Document Type: Article

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