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Volumn 35, Issue 17, 2015, Pages 6893-6902

Nitric oxide signaling is recruited as a compensatory mechanism for sustaining synaptic plasticity in Alzheimer’s disease mice

Author keywords

Calcium; Homeostasis; Nitric oxide; Ryanodine receptor; Synaptic depression; Synaptic plasticity

Indexed keywords

AXIN; BETA CATENIN; GLYCOGEN SYNTHASE KINASE 3BETA; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; NESTIN; WNT3A PROTEIN; AMYLOID PRECURSOR PROTEIN; ENZYME INHIBITOR; N ACETYL S NITROSOPENICILLAMINE; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE DONOR; PRESENILIN 1; PSEN1 PROTEIN, HUMAN; RYANODINE RECEPTOR; TAU PROTEIN;

EID: 84929347124     PISSN: 02706474     EISSN: 15292401     Source Type: Journal    
DOI: 10.1523/JNEUROSCI.4002-14.2015     Document Type: Article
Times cited : (74)

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