-
1
-
-
84895424744
-
Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-gamma-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment
-
Tillack C, Ehmann LM, Friedrich M, et al. Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-gamma-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment. Gut. 2014;63:567-577.
-
(2014)
Gut.
, vol.63
, pp. 567-577
-
-
Tillack, C.1
Ehmann, L.M.2
Friedrich, M.3
-
2
-
-
78649713662
-
Severe skin lesions cause patients with inflammatory bowel disease to discontinue anti-tumor necrosis factor therapy
-
Rahier JF, Buche S, Peyrin-Biroulet L, et al. Severe skin lesions cause patients with inflammatory bowel disease to discontinue anti-tumor necrosis factor therapy. Clin Gastroenterol Hepatol. 2010;8:1048-1055.
-
(2010)
Clin Gastroenterol Hepatol.
, vol.8
, pp. 1048-1055
-
-
Rahier, J.F.1
Buche, S.2
Peyrin-Biroulet, L.3
-
3
-
-
67650216399
-
Induction and exacerbation of psoriasis with TNF-blockade therapy: A review and analysis of 127 cases
-
Ko JM, Gottlieb AB, Kerbleski JF. Induction and exacerbation of psoriasis with TNF-blockade therapy: a review and analysis of 127 cases. J Dermatolog Treat. 2009;20:100-108.
-
(2009)
J Dermatolog Treat.
, vol.20
, pp. 100-108
-
-
Ko, J.M.1
Gottlieb, A.B.2
Kerbleski, J.F.3
-
4
-
-
81355151359
-
Psoriasis associated with antitumour necrosis factor therapy in inflammatory bowel disease: A new series and a review of 120 cases from the literature
-
Cullen G, Kroshinsky D, Cheifetz AS, et al. Psoriasis associated with antitumour necrosis factor therapy in inflammatory bowel disease: a new series and a review of 120 cases from the literature. Aliment Pharmacol Ther. 2011;34:1318-1327.
-
(2011)
Aliment Pharmacol Ther.
, vol.34
, pp. 1318-1327
-
-
Cullen, G.1
Kroshinsky, D.2
Cheifetz, A.S.3
-
5
-
-
84872675083
-
Psoriasis and palmoplantar pustulosis associated with tumor necrosis factor-alpha inhibitors: The Mayo Clinic experience, 1998 to 2010
-
Shmidt E, Wetter DA, Ferguson SB, et al. Psoriasis and palmoplantar pustulosis associated with tumor necrosis factor-alpha inhibitors: the Mayo Clinic experience, 1998 to 2010. J Am Acad Dermatol. 2012;67: e179-e185.
-
(2012)
J Am Acad Dermatol.
, vol.67
, pp. e179-e185
-
-
Shmidt, E.1
Wetter, D.A.2
Ferguson, S.B.3
-
6
-
-
79953099459
-
Infliximab and adalimumab-induced psoriasis in Crohn's disease: A paradoxical side effect
-
Iborra M, Beltran B, Bastida G, et al. Infliximab and adalimumab-induced psoriasis in Crohn's disease: a paradoxical side effect. J Crohns Colitis. 2011;5:157-161.
-
(2011)
J Crohns Colitis.
, vol.5
, pp. 157-161
-
-
Iborra, M.1
Beltran, B.2
Bastida, G.3
-
7
-
-
84860835230
-
Induction of psoriasis with anti-TNF agents in patients with inflammatory bowel disease: A report of 21 cases
-
Guerra I, Algaba A, Perez-Calle JL, et al. Induction of psoriasis with anti-TNF agents in patients with inflammatory bowel disease: a report of 21 cases. J Crohns Colitis. 2012;6:518-523.
-
(2012)
J Crohns Colitis.
, vol.6
, pp. 518-523
-
-
Guerra, I.1
Algaba, A.2
Perez-Calle, J.L.3
-
8
-
-
84859905688
-
Methotrexate for psoriasiform lesions associated with anti-tumour necrosis factor therapy in inflammatory bowel disease
-
Buisson A, Cuny JF, Barbaud A, et al. Methotrexate for psoriasiform lesions associated with anti-tumour necrosis factor therapy in inflammatory bowel disease. Aliment Pharmacol Ther. 2012;35:1175-1180.
-
(2012)
Aliment Pharmacol Ther.
, vol.35
, pp. 1175-1180
-
-
Buisson, A.1
Cuny, J.F.2
Barbaud, A.3
-
9
-
-
84886952283
-
Paradoxical immune-mediated inflammation in inflammatory bowel disease patients receiving anti-TNF-alpha agents
-
Fiorino G, Danese S, Pariente B, et al. Paradoxical immune-mediated inflammation in inflammatory bowel disease patients receiving anti-TNF-alpha agents. Autoimmun Rev. 2014;13:15-19.
-
(2014)
Autoimmun Rev.
, vol.13
, pp. 15-19
-
-
Fiorino, G.1
Danese, S.2
Pariente, B.3
-
10
-
-
63849102069
-
Review article: Anti-TNF-alpha induced psoriasis in patients with inflammatory bowel disease
-
Fiorino G, Allez M, Malesci A, et al. Review article: anti-TNF-alpha induced psoriasis in patients with inflammatory bowel disease. Aliment Pharmacol Ther. 2009;29:921-927.
-
(2009)
Aliment Pharmacol Ther.
, vol.29
, pp. 921-927
-
-
Fiorino, G.1
Allez, M.2
Malesci, A.3
-
11
-
-
84895501093
-
Anti-TNF and skin inflammation in IBD: A new paradox in gastroenterology?
-
Niess JH, Danese S. Anti-TNF and skin inflammation in IBD: a new paradox in gastroenterology? Gut. 2014;63:533-535.
-
(2014)
Gut
, vol.63
, pp. 533-535
-
-
Niess, J.H.1
Danese, S.2
-
12
-
-
84876399144
-
Skin side effects of inflammatory bowel disease therapy
-
Torres J, Buche S, Delaporte E, et al. Skin side effects of inflammatory bowel disease therapy. Inflamm Bowel Dis. 2013;19:1086-1098.
-
(2013)
Inflamm Bowel Dis.
, vol.19
, pp. 1086-1098
-
-
Torres, J.1
Buche, S.2
Delaporte, E.3
-
13
-
-
14744276518
-
Cross-regulation of TNF and IFN-alpha in autoimmune diseases
-
Palucka AK, Blanck JP, Bennett L, et al. Cross-regulation of TNF and IFN-alpha in autoimmune diseases. Proc Natl Acad Sci U S A. 2005;102: 3372-3377.
-
(2005)
Proc Natl Acad Sci U S A.
, vol.102
, pp. 3372-3377
-
-
Palucka, A.K.1
Blanck, J.P.2
Bennett, L.3
-
14
-
-
58149316658
-
Induction of IL-17+ T cell trafficking and development by IFN-gamma: Mechanism and pathological relevance in psoriasis
-
Kryczek I, Bruce AT, Gudjonsson JE, et al. Induction of IL-17+ T cell trafficking and development by IFN-gamma: mechanism and pathological relevance in psoriasis. J Immunol. 2008;181:4733-4741.
-
(2008)
J Immunol.
, vol.181
, pp. 4733-4741
-
-
Kryczek, I.1
Bruce, A.T.2
Gudjonsson, J.E.3
-
15
-
-
38849187386
-
IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammation
-
Ma HL, Liang S, Li J, et al. IL-22 is required for Th17 cell-mediated pathology in a mouse model of psoriasis-like skin inflammation. J Clin Invest. 2008;118:597-607.
-
(2008)
J Clin Invest.
, vol.118
, pp. 597-607
-
-
Ma, H.L.1
Liang, S.2
Li, J.3
-
16
-
-
33646552450
-
IL-22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: A potential role in psoriasis
-
Wolk K, Witte E, Wallace E, et al. IL-22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis. Eur J Immunol. 2006;36: 1309-1323.
-
(2006)
Eur J Immunol.
, vol.36
, pp. 1309-1323
-
-
Wolk, K.1
Witte, E.2
Wallace, E.3
-
17
-
-
72849131815
-
Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling
-
Eyerich S, Eyerich K, Pennino D, et al. Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling. J Clin Invest. 2009;119:3573-3585.
-
(2009)
J Clin Invest.
, vol.119
, pp. 3573-3585
-
-
Eyerich, S.1
Eyerich, K.2
Pennino, D.3
-
18
-
-
80855137955
-
Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: Implications in psoriasis pathogenesis
-
Carrier Y, Ma HL, Ramon HE, et al. Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: implications in psoriasis pathogenesis. J Invest Dermatol. 2011;131:2428-2437.
-
(2011)
J Invest Dermatol.
, vol.131
, pp. 2428-2437
-
-
Carrier, Y.1
Ma, H.L.2
Ramon, H.E.3
-
19
-
-
79951821248
-
IL-1F5,-F6,-F8, and-F9: A novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression
-
Johnston A, Xing X, Guzman AM, et al. IL-1F5,-F6,-F8, and-F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression. J Immunol. 2011;186:2613-2622.
-
(2011)
J Immunol.
, vol.186
, pp. 2613-2622
-
-
Johnston, A.1
Xing, X.2
Guzman, A.M.3
-
20
-
-
84868613788
-
Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk
-
Tortola L, Rosenwald E, Abel B, et al. Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk. J Clin Invest. 2012;122:3965-3976.
-
(2012)
J Clin Invest.
, vol.122
, pp. 3965-3976
-
-
Tortola, L.1
Rosenwald, E.2
Abel, B.3
-
22
-
-
84872275425
-
The emerging role of IL-17 in the pathogenesis of psoriasis: Preclinical and clinical findings
-
Martin DA, Towne JE, Kricorian G, et al. The emerging role of IL-17 in the pathogenesis of psoriasis: preclinical and clinical findings. J Invest Dermatol. 2013;133:17-26.
-
(2013)
J Invest Dermatol.
, vol.133
, pp. 17-26
-
-
Martin, D.A.1
Towne, J.E.2
Kricorian, G.3
-
23
-
-
84874256199
-
Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation
-
Johnston A, Fritz Y, Dawes SM, et al. Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation. J Immunol. 2013;190: 2252-2262.
-
(2013)
J Immunol.
, vol.190
, pp. 2252-2262
-
-
Johnston, A.1
Fritz, Y.2
Dawes, S.M.3
-
24
-
-
58349105187
-
Characterization of the interleukin-17 isoforms and receptors in lesional psoriatic skin
-
Johansen C, Usher PA, Kjellerup RB, et al. Characterization of the interleukin-17 isoforms and receptors in lesional psoriatic skin. Br J Dermatol. 2009;160:319-324.
-
(2009)
Br J Dermatol.
, vol.160
, pp. 319-324
-
-
Johansen, C.1
Usher, P.A.2
Kjellerup, R.B.3
-
25
-
-
79960413975
-
Tumor necrosis factor alphamediated induction of interleukin 17C in human keratinocytes is controlled by nuclear factor kappaB
-
Johansen C, Riis JL, Gedebjerg A, et al. Tumor necrosis factor alphamediated induction of interleukin 17C in human keratinocytes is controlled by nuclear factor kappaB. J Biol Chem. 2011;286:25487-25494.
-
(2011)
J Biol Chem.
, vol.286
, pp. 25487-25494
-
-
Johansen, C.1
Riis, J.L.2
Gedebjerg, A.3
-
26
-
-
79955738020
-
Role of p38 mitogen-activated protein kinase isoforms in murine skin inflammation induced by 12-Otetradecanoylphorbol 13-acetate
-
Lilleholt LL, Johansen C, Arthur JS, et al. Role of p38 mitogen-activated protein kinase isoforms in murine skin inflammation induced by 12-Otetradecanoylphorbol 13-acetate. Acta Derm Venereol. 2011;91:271-278.
-
(2011)
Acta Derm Venereol.
, vol.91
, pp. 271-278
-
-
Lilleholt, L.L.1
Johansen, C.2
Arthur, J.S.3
-
27
-
-
81255158784
-
IL-17C regulates the innate immune function of epithelial cells in an autocrine manner
-
Ramirez-Carrozzi V, Sambandam A, Luis E, et al. IL-17C regulates the innate immune function of epithelial cells in an autocrine manner. Nat Immunol. 2011;12:1159-1166.
-
(2011)
Nat Immunol.
, vol.12
, pp. 1159-1166
-
-
Ramirez-Carrozzi, V.1
Sambandam, A.2
Luis, E.3
-
28
-
-
80755133721
-
Interleukin-17C promotes Th17 cell responses and autoimmune disease via interleukin-17 receptor e
-
Chang SH, Reynolds JM, Pappu BP, et al. Interleukin-17C promotes Th17 cell responses and autoimmune disease via interleukin-17 receptor E. Immunity. 2011;35:611-621.
-
(2011)
Immunity.
, vol.35
, pp. 611-621
-
-
Chang, S.H.1
Reynolds, J.M.2
Pappu, B.P.3
-
29
-
-
81255158786
-
IL-17RE is the functional receptor for IL-17C and mediates mucosal immunity to infection with intestinal pathogens
-
Song X, Zhu S, Shi P, et al. IL-17RE is the functional receptor for IL-17C and mediates mucosal immunity to infection with intestinal pathogens. Nat Immunol. 2011;12:1151-1158.
-
(2011)
Nat Immunol.
, vol.12
, pp. 1151-1158
-
-
Song, X.1
Zhu, S.2
Shi, P.3
-
30
-
-
33846974080
-
Psoriasis and pustular dermatitis triggered by TNF-{alpha} inhibitors in patients with rheumatologic conditions
-
de Gannes GC, Ghoreishi M, Pope J, et al. Psoriasis and pustular dermatitis triggered by TNF-{alpha} inhibitors in patients with rheumatologic conditions. Arch Dermatol. 2007;143:223-231.
-
(2007)
Arch Dermatol.
, vol.143
, pp. 223-231
-
-
De Gannes, G.C.1
Ghoreishi, M.2
Pope, J.3
-
31
-
-
71949120395
-
Cytokine imbalance with increased production of interferon-alpha in psoriasiform eruptions associated with antitumour necrosis factor-alpha treatments
-
Seneschal J, Milpied B, Vergier B, et al. Cytokine imbalance with increased production of interferon-alpha in psoriasiform eruptions associated with antitumour necrosis factor-alpha treatments. Br J Dermatol. 2009;161:1081-1088.
-
(2009)
Br J Dermatol.
, vol.161
, pp. 1081-1088
-
-
Seneschal, J.1
Milpied, B.2
Vergier, B.3
-
32
-
-
78249237580
-
Strong overexpression of CXCR3 axis components in childhood inflammatory bowel disease
-
Schroepf S, Kappler R, Brand S, et al. Strong overexpression of CXCR3 axis components in childhood inflammatory bowel disease. Inflamm Bowel Dis. 2010;16:1882-1890.
-
(2010)
Inflamm Bowel Dis.
, vol.16
, pp. 1882-1890
-
-
Schroepf, S.1
Kappler, R.2
Brand, S.3
-
33
-
-
84925643390
-
IL-17A alone weakly affects the transcriptome of intestinal epithelial cells but strongly modulates the TNF-A-induced expression of inflammatory mediators and inflammatory bowel disease susceptibility genes
-
Friedrich M, Diegelmann J, Beigel F, et al. IL-17A alone weakly affects the transcriptome of intestinal epithelial cells but strongly modulates the TNF-a-induced expression of inflammatory mediators and inflammatory bowel disease susceptibility genes. Inflamm Bowel Dis. 2012;20:1502-1515.
-
(2012)
Inflamm Bowel Dis.
, vol.20
, pp. 1502-1515
-
-
Friedrich, M.1
Diegelmann, J.2
Beigel, F.3
-
34
-
-
42449084096
-
Role of the novel Th17 cytokine IL-17F in inflammatory bowel disease (IBD): Upregulated colonic IL-17F expression in active Crohn's disease and analysis of the IL17F p. His161Arg polymorphism in IBD
-
Seiderer J, Elben I, Diegelmann J, et al. Role of the novel Th17 cytokine IL-17F in inflammatory bowel disease (IBD): upregulated colonic IL-17F expression in active Crohn's disease and analysis of the IL17F p.His161Arg polymorphism in IBD. Inflamm Bowel Dis. 2008;14:437-445.
-
(2008)
Inflamm Bowel Dis.
, vol.14
, pp. 437-445
-
-
Seiderer, J.1
Elben, I.2
Diegelmann, J.3
-
35
-
-
33645829078
-
IL-22 is increased in active Crohn's disease and promotes proinflammatory gene expression and intestinal epithelial cell migration
-
Brand S, Beigel F, Olszak T, et al. IL-22 is increased in active Crohn's disease and promotes proinflammatory gene expression and intestinal epithelial cell migration. Am J Physiol Gastrointest Liver Physiol. 2006; 290:G827-G838.
-
(2006)
Am J Physiol Gastrointest Liver Physiol.
, vol.290
, pp. G827-G838
-
-
Brand, S.1
Beigel, F.2
Olszak, T.3
-
36
-
-
68849124464
-
The role of the novel Th17 cytokine IL-26 in intestinal inflammation
-
Dambacher J, Beigel F, Zitzmann K, et al. The role of the novel Th17 cytokine IL-26 in intestinal inflammation. Gut. 2009;58:1207-1217.
-
(2009)
Gut.
, vol.58
, pp. 1207-1217
-
-
Dambacher, J.1
Beigel, F.2
Zitzmann, K.3
-
37
-
-
33645005339
-
Cell differentiation dependent expressed CCR6 mediates ERK-1/2, SAPK/JNK, and Akt signaling resulting in proliferation and migration of colorectal cancer cells
-
Brand S, Olszak T, Beigel F, et al. Cell differentiation dependent expressed CCR6 mediates ERK-1/2, SAPK/JNK, and Akt signaling resulting in proliferation and migration of colorectal cancer cells. J Cell Biochem. 2006;97:709-723.
-
(2006)
J Cell Biochem.
, vol.97
, pp. 709-723
-
-
Brand, S.1
Olszak, T.2
Beigel, F.3
-
38
-
-
67650224449
-
Crohn's disease: Th1, Th17 or both? the change of a paradigm: New immunological and genetic insights implicate Th17 cells in the pathogenesis of Crohn's disease
-
Brand S. Crohn's disease: Th1, Th17 or both? the change of a paradigm: new immunological and genetic insights implicate Th17 cells in the pathogenesis of Crohn's disease. Gut. 2009;58:1152-1167.
-
(2009)
Gut.
, vol.58
, pp. 1152-1167
-
-
Brand, S.1
-
39
-
-
39649121919
-
Linking genetic susceptibility to Crohn's disease with Th17 cell function: IL-22 serum levels are increased in Crohn's disease and correlate with disease activity and IL23R genotype status
-
Schmechel S, Konrad A, Diegelmann J, et al. Linking genetic susceptibility to Crohn's disease with Th17 cell function: IL-22 serum levels are increased in Crohn's disease and correlate with disease activity and IL23R genotype status. Inflamm Bowel Dis. 2008;14:204-212.
-
(2008)
Inflamm Bowel Dis.
, vol.14
, pp. 204-212
-
-
Schmechel, S.1
Konrad, A.2
Diegelmann, J.3
-
40
-
-
0037216498
-
Increased expression of interleukin 17 in inflammatory bowel disease
-
Fujino S, Andoh A, Bamba S, et al. Increased expression of interleukin 17 in inflammatory bowel disease. Gut. 2003;52:65-70.
-
(2003)
Gut.
, vol.52
, pp. 65-70
-
-
Fujino, S.1
Andoh, A.2
Bamba, S.3
|