-
1
-
-
84924565565
-
2 activity
-
2 activity. J Physiol 593, 1509-1521.
-
(2015)
J Physiol
, vol.593
, pp. 1509-1521
-
-
Aronsen, J.M.1
Skogestad, J.2
Lewalle, A.3
Louch, W.E.4
Hougen, K.5
Stokke, M.K.6
Swift, F.7
Niederer, S.8
Smith, N.P.9
Sejersted, O.M.10
Sjaastad, I.11
-
3
-
-
84924529893
-
+ channel regulation, structure, trafficking and sequestration
-
+ channel regulation, structure, trafficking and sequestration. J Physiol 593, 1347-1360.
-
(2015)
J Physiol
, vol.593
, pp. 1347-1360
-
-
Chen-Izu, Y.1
Shaw, R.M.2
Pitt, G.S.3
Yarov-Yarovoy, V.4
Sack, J.T.5
Abriel, H.6
Aldrich, R.W.7
Belardinelli, L.8
Cannell, M.B.9
Catterall, W.A.10
Chazin, W.J.11
Chiamvimonvat, N.12
Deschenes, I.13
Grandi, E.14
Hund, T.J.15
Izu, L.T.16
Maier, L.S.17
Maltsev, V.A.18
Marionneau, C.19
Mohler, P.J.20
Rajamani, S.21
Rasmusson, R.L.22
Sobie, E.A.23
Clancy, C.E.24
Bers, D.M.25
more..
-
4
-
-
84924559521
-
Deranged sodium to sudden death
-
Clancy CE, Chen-Izu Y, Bers DM, Belardinelli L, Boyden PA, Csernoch L, Despa S, Fermini B, Hool LC, Izu L, Kass RS, Lederer WJ, Louch WE, Maack C, Matiazzi A, Qu Z, Rajamani S, Rippinger CM, Sejersted OM, O'Rourke B, Weiss JN, Varró A & Zaza A (2015). Deranged sodium to sudden death. J Physiol 593, 1331-1345.
-
(2015)
J Physiol
, vol.593
, pp. 1331-1345
-
-
Clancy, C.E.1
Chen-Izu, Y.2
Bers, D.M.3
Belardinelli, L.4
Boyden, P.A.5
Csernoch, L.6
Despa, S.7
Fermini, B.8
Hool, L.C.9
Izu, L.10
Kass, R.S.11
Lederer, W.J.12
Louch, W.E.13
Maack, C.14
Matiazzi, A.15
Qu, Z.16
Rajamani, S.17
Rippinger, C.M.18
Sejersted, O.M.19
O'Rourke, B.20
Weiss, J.N.21
Varró, A.22
Zaza, A.23
more..
-
5
-
-
84880324424
-
+ transport in the normal and failing heart - remember the balance
-
+ transport in the normal and failing heart - remember the balance. J Mol Cell Cardiol 61, 2-10.
-
(2013)
J Mol Cell Cardiol
, vol.61
, pp. 2-10
-
-
Despa, S.1
Bers, D.M.2
-
7
-
-
84924527564
-
2+ release and excitation-contraction coupling in atrial myocytes from normal and failing hearts
-
2+ release and excitation-contraction coupling in atrial myocytes from normal and failing hearts. J Physiol 593, 1459-1477.
-
(2015)
J Physiol
, vol.593
, pp. 1459-1477
-
-
Hohendanner, F.1
Walther, S.2
Maxwell, J.T.3
Kettlewell, S.4
Awad, S.5
Smith, G.L.6
Lonchyna, V.A.7
Blatter, L.A.8
-
9
-
-
84924560328
-
Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts
-
Lin X, O'Malley H, Chen C, Auerbach D, Foster M, Shekhar A, Zhang M, Coetzee W, Jalife J, Fishman GI, Isom L & Delmar M (2015). Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts. J Physiol 593, 1389-1407.
-
(2015)
J Physiol
, vol.593
, pp. 1389-1407
-
-
Lin, X.1
O'Malley, H.2
Chen, C.3
Auerbach, D.4
Foster, M.5
Shekhar, A.6
Zhang, M.7
Coetzee, W.8
Jalife, J.9
Fishman, G.I.10
Isom, L.11
Delmar, M.12
-
10
-
-
84924591365
-
Alternating membrane potential/calcium interplay underlies repetitive focal activity in a genetic model of calcium-dependent atrial arrhythmias
-
Lou Q, Belevych AE, Radwański PB, Liu B, Kalyanasundaram A, Knollmann BC, Fedorov VV & Györke S (2015). Alternating membrane potential/calcium interplay underlies repetitive focal activity in a genetic model of calcium-dependent atrial arrhythmias. J Physiol 593, 1443-1458.
-
(2015)
J Physiol
, vol.593
, pp. 1443-1458
-
-
Lou, Q.1
Belevych, A.E.2
Radwański, P.B.3
Liu, B.4
Kalyanasundaram, A.5
Knollmann, B.C.6
Fedorov, V.V.7
Györke, S.8
-
12
-
-
84924530069
-
+ current and increased ryanodine receptor sensitivity synergistically contribute to sustained focal arrhythmia in the intact rabbit heart
-
+ current and increased ryanodine receptor sensitivity synergistically contribute to sustained focal arrhythmia in the intact rabbit heart. J Physiol 593, 1479-1493.
-
(2015)
J Physiol
, vol.593
, pp. 1479-1493
-
-
Myles, R.C.1
Wang, L.2
Bers, D.M.3
Ripplinger, C.M.4
-
13
-
-
84924534405
-
2+ release refractoriness by β-adrenergic stimulation requires dual activation of kinases PKA and CaMKII in mouse ventricular myocytes
-
2+ release refractoriness by β-adrenergic stimulation requires dual activation of kinases PKA and CaMKII in mouse ventricular myocytes. J Physiol 593, 1495-1507.
-
(2015)
J Physiol
, vol.593
, pp. 1495-1507
-
-
Poláková, E.1
Illaste, A.2
Niggli, E.3
Sobie, E.A.4
-
14
-
-
84924588019
-
+-ATPase in the heart
-
+-ATPase in the heart. J Physiol 593, 1361-1382.
-
(2015)
J Physiol
, vol.593
, pp. 1361-1382
-
-
Shattock, M.J.1
Ottolia, M.2
Bers, D.M.3
Blaustein, M.P.4
Boguslavskyi, A.5
Bossuyt, J.6
Bridge, J.H.B.7
Chen-Izu, Y.8
Clancy, C.E.9
Edwards, A.10
Goldhaber, J.11
Kaplan, J.12
Lingrel, J.B.13
Pavlovic, D.14
Philipson, K.15
Sipido, K.R.16
Xie, Z.-J.17
-
15
-
-
84924546284
-
Na
-
Na. J Physiol 593, 1429-1442.
-
(2015)
J Physiol
, vol.593
, pp. 1429-1442
-
-
Yang, P.-C.1
Song, Y.2
Giles, W.R.3
Horvath, B.4
Chen-Izu, Y.5
Belardinelli, L.6
Rajamani, S.7
Clancy, C.E.8
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