Enterocolitis causes profound lymphoid depletion in endothelin receptor B- and endothelin 3-null mouse models of Hirschsprung-associated enterocolitis

European Journal of Immunology

Volumn 45, Issue 3, 2015, Pages 807-817

  Frykman, Philip K ^a   Cheng, Zhi ^a   Wang, Xiao ^a   Dhall, Deepti ^a  

^a CEDARS SINAI MEDICAL CENTER   (United States)

Author keywords

Corticosterone; Endothelins; Enterocolitis; Glucocorticoids; HAEC; Hirschsprung disease; Lymphoid depletion

Indexed keywords

CORTICOSTERONE; ENDOTHELIN 3; ENDOTHELIN RECEPTOR; ENDOTHELIN RECEPTOR B; UNCLASSIFIED DRUG; ENDOTHELIN B RECEPTOR;

ADOPTIVE TRANSFER; AGANGLIONOSIS; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BONE MARROW CELL; BONE MARROW TRANSPLANTATION; CD4+ T LYMPHOCYTE; CD8+ T LYMPHOCYTE; CELL COUNT; CONTROLLED STUDY; CORTICOSTERONE BLOOD LEVEL; ENTEROCOLITIS; FLUORESCENCE ACTIVATED CELL SORTING; GENOTYPE; HEMATOPOIETIC STEM CELL; HIRSCHSPRUNG ASSOCIATED ENTEROCOLITIS; HIRSCHSPRUNG DISEASE; IMMUNOSUPPRESSIVE TREATMENT; LYMPHOCYTE COUNT; LYMPHOCYTE DEPLETION; LYMPHOCYTE SUBPOPULATION; LYMPHOCYTOPENIA; LYMPHOID PROGENITOR CELL; LYMPHOPOIESIS; MEGACOLON; MOUSE; NEWBORN SEPSIS; NONHUMAN; PHENOTYPE; PHYSIOLOGICAL STRESS; POSTNATAL DEVELOPMENT; PRE B LYMPHOCYTE; PRIORITY JOURNAL; SPLEEN CELL; THYMOCYTE; THYMUS; ANIMAL; COLON; DISEASE MODEL; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; PATHOLOGY; SPLEEN;

ANIMALS; COLON; CORTICOSTERONE; DISEASE MODELS, ANIMAL; ENDOTHELIN-3; ENTEROCOLITIS; HIRSCHSPRUNG DISEASE; MICE; MICE, KNOCKOUT; RECEPTOR, ENDOTHELIN B; SPLEEN; THYMUS GLAND;

EID: 84924534785     PISSN: 00142980     EISSN: 15214141     Source Type: Journal    
DOI: 10.1002/eji.201444737     Document Type: Article

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