Insulin resistance caused by lipotoxicity is related to oxidative stress and endoplasmic reticulum stress in LPL gene knockout heterozygous mice

Atherosclerosis

Volumn 239, Issue 1, 2015, Pages 276-282

  Li, Yang Xue ^a   Han, Ting Ting ^a   Liu, Yang ^a   Zheng, Shuang ^a   Zhang, Yao ^a   Liu, Wei ^a   Hu, Yao Min ^a  

^a SHANGHAI JIAO TONG UNIVERSITY SCHOOL OF MEDICINE   (China)

Author keywords

Endoplasmic reticulum stress; Insulin resistance; Lipoprotein lipase; Oxidative stress

Indexed keywords

BIOLOGICAL MARKER; GLUCOSE; INSULIN; LIPID; LIPOPROTEIN LIPASE; MALONALDEHYDE; PROTEIN KINASE B; REACTIVE OXYGEN METABOLITE; RECOMBINANT HUMAN INSULIN; SERINE; GLUCOSE BLOOD LEVEL;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CLINICAL ASSESSMENT TOOL; CONTROLLED STUDY; ENDOPLASMIC RETICULUM STRESS; FAT MASS; FEMALE; GLUCOSE BLOOD LEVEL; GLUCOSE METABOLISM; GLUCOSE TOLERANCE TEST; HOMA IR; HYPERTRIGLYCERIDEMIA; IMPAIRED GLUCOSE TOLERANCE; INSULIN BLOOD LEVEL; INSULIN RESISTANCE; INSULIN SENSITIVITY; INTRAABDOMINAL FAT; LIPID BLOOD LEVEL; LIPID PEROXIDATION; LIPID STORAGE; LIPOTOXICITY; LIVER; MOUSE; NONHUMAN; OXIDATIVE STRESS; PANCREAS; PLASMA; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; SKELETAL MUSCLE; TISSUE SPECIFICITY; TISSUE STRUCTURE; ANALYSIS; ANIMAL; BODY COMPOSITION; GENETICS; HETEROZYGOTE; HOMEOSTASIS; KNOCKOUT MOUSE; METABOLISM; PATHOLOGY; PHOSPHORYLATION; SIGNAL TRANSDUCTION; TIME FACTOR;

ANIMALS; BLOOD GLUCOSE; BODY COMPOSITION; ENDOPLASMIC RETICULUM STRESS; FEMALE; GLUCOSE; GLUCOSE TOLERANCE TEST; HETEROZYGOTE; HOMEOSTASIS; HYPERTRIGLYCERIDEMIA; INSULIN; INSULIN RESISTANCE; LIPID PEROXIDATION; LIPOPROTEIN LIPASE; LIVER; MICE; MICE, KNOCKOUT; MUSCLE, SKELETAL; OXIDATIVE STRESS; PHOSPHORYLATION; SIGNAL TRANSDUCTION; TIME FACTORS;

EID: 84921743550     PISSN: 00219150     EISSN: 18791484     Source Type: Journal    
DOI: 10.1016/j.atherosclerosis.2015.01.020     Document Type: Article

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