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Volumn 7, Issue 344, 2014, Pages 1c2-

Response to comment on "controlling long-term signaling: Receptor dynamics determine attenuation and refractory behavior of the TGF-b pathway"-Smad2/3 activity does not predict the dynamics of transcription

Author keywords

[No Author keywords available]

Indexed keywords

SMAD2 PROTEIN; SMAD3 PROTEIN; TRANSFORMING GROWTH FACTOR BETA; TRANSFORMING GROWTH FACTOR BETA RECEPTOR; UBIQUITIN PROTEIN LIGASE E3;

EID: 84907506703     PISSN: 19450877     EISSN: 19379145     Source Type: Journal    
DOI: 10.1126/scisignal.2005669     Document Type: Article
Times cited : (3)

References (10)
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    • (2013) Sci. Signal. , vol.6 , pp. ra106
    • Vizán, P.1    Miller, D.S.2    Gori, I.3    Das, D.4    Schmierer, B.5    Hill, C.S.6
  • 2
    • 84905752998 scopus 로고    scopus 로고
    • Encoding of temporal signals by the TGF-b pathway and implications for embryonic patterning
    • B. Sorre, A. Warmflash, E. D. Siggia, A. H. Brivanlou, Encoding of temporal signals by the TGF-b pathway and implications for embryonic patterning. Dev. Cell 30, 334-342 (2014)
    • (2014) Dev. Cell , vol.30 , pp. 342-334
    • Sorre, B.1    Warmflash, A.2    Siggia, E.D.3    Brivanlou, A.H.4
  • 3
  • 5
    • 24344483878 scopus 로고    scopus 로고
    • Smad4 dependency defines two classes of transforming growth factor b (TGF-b) target genes and distinguishes TGF-b-induced epithelial-mesenchymal transition from its antiproliferative and migratory responses
    • L. Levy, C. S. Hill, Smad4 dependency defines two classes of transforming growth factor b (TGF-b) target genes and distinguishes TGF-b-induced epithelial-mesenchymal transition from its antiproliferative and migratory responses. Mol. Cell. Biol. 25, 8108-8125 (2005)
    • (2005) Mol. Cell. Biol. , vol.25 , pp. 8108-8125
    • Levy, L.1    Hill, C.S.2
  • 6
    • 0038548187 scopus 로고    scopus 로고
    • Attenuation of the TGF-b-Smadsignaling pathway in pancreatic tumor cells confers resistance to TGF-b-induced growth arrest
    • F. J. Nicolás, C. S. Hill,Attenuation of the TGF-b-Smadsignaling pathway in pancreatic tumor cells confers resistance to TGF-b-induced growth arrest. Oncogene 22, 3698-3711 (2003)
    • (2003) Oncogene , vol.22 , pp. 3698-3711
    • Nicolás, F.J.1    Hill, C.S.2
  • 8
    • 34548238145 scopus 로고    scopus 로고
    • Arkadia activates Smad3/Smad4-dependent transcription by triggering signal-induced SnoN degradation
    • L. Levy, M. Howell, D. Das, S. Harkin, V. Episkopou, C. S. Hill, Arkadia activates Smad3/Smad4-dependent transcription by triggering signal-induced SnoN degradation. Mol. Cell. Biol. 27, 6068-6083 (2007)
    • (2007) Mol. Cell. Biol. , vol.27 , pp. 6068-6083
    • Levy, L.1    Howell, M.2    Das, D.3    Harkin, S.4    Episkopou, V.5    Hill, C.S.6
  • 9
    • 0033595704 scopus 로고    scopus 로고
    • Negative feedback regulation of TGF-b signaling by the SnoN oncoprotein
    • S. L. Stroschein, W. Wang, S. Zhou, Q. Zhou, K. Luo, Negative feedback regulation of TGF-b signaling by the SnoN oncoprotein. Science 286, 771-774 (1999)
    • (1999) Science , vol.286 , pp. 771-774
    • Stroschein, S.L.1    Wang, W.2    Zhou, S.3    Zhou, Q.4    Luo, K.5
  • 10
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    • A self-enabling TGFb response coupled to stress signaling: Smad engages stress response factor ATF3 for Id1 repression in epithelial cells
    • Y. Kang, C. R. Chen, J. Massagué, A self-enabling TGFb response coupled to stress signaling: Smad engages stress response factor ATF3 for Id1 repression in epithelial cells. Mol. Cell 11, 915-926 (2003).
    • (2003) Mol. Cell , vol.11 , pp. 915-926
    • Kang, Y.1    Chen, C.R.2    Massagué, J.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.