Dysfunctional mitochondrial Ca2+ handling in mutant SOD1 mouse models of fALS: Integration of findings from motor neuron somata and motor terminals

Frontiers in Cellular Neuroscience

Volumn 8, Issue JULY, 2014, Pages

  Barrett, Ellen F ^a   Barrett, John N ^a   David, Gavriel ^a  

^a UNIVERSITY OF MIAMI MILLER SCHOOL OF MEDICINE   (United States)

Author keywords

Mitochondria; Motor nerve terminal; Motor neuron; Mutant SOD1 models of fALS

Indexed keywords

CALCIUM; COPPER ZINC SUPEROXIDE DISMUTASE; CYCLOPHILIN D; MITOCHONDRIAL PERMEABILITY TRANSITION PORE;

AMYOTROPHIC LATERAL SCLEROSIS; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CALCIUM TRANSPORT; DISORDERS OF MITOCHONDRIAL FUNCTIONS; GENE INACTIVATION; KNOCKOUT MOUSE; MOTONEURON; MUSCLE DENERVATION; NERVE CELL MEMBRANE STEADY POTENTIAL; NERVE ENDING; NERVE FIBER; NERVE STIMULATION; NONHUMAN; PH;

EID: 84904069426     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2014.00184     Document Type: Article

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