Aging causes exacerbated ischemic brain injury and failure of sevoflurane post-conditioning: Role of B-cell lymphoma-2

Neuroscience

Volumn 275, Issue , 2014, Pages 2-11

  Dong, P ^a   Zhao, J ^b   Zhang, Y ^c   Dong, J ^a   Zhang, L ^a   Li, D ^a   Li, L ^a   Zhang, X ^a   Yang, B ^a   Lei, W ^a  

^a SHANDONG UNIVERSITY   (China)

^b Department of Internal Medicine   (China)

^c First People's Hospital of Jining   (China)

Author keywords

Aging; Anesthetic post conditioning; Apoptosis; B cell lymphoma 2; Ischemic stroke

Indexed keywords

PROTEIN BAX; PROTEIN BCL 2; SEVOFLURANE; ETHER DERIVATIVE; NEUROPROTECTIVE AGENT;

ADULT; AGING; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN CORTEX; BRAIN EDEMA; BRAIN INFARCTION; BRAIN INFARCTION SIZE; BRAIN INJURY; BRAIN ISCHEMIA; CONDITIONING; CONTROLLED STUDY; DISEASE ASSOCIATION; DISEASE EXACERBATION; DNA SEQUENCE; DOWN REGULATION; DRUG EFFECT; MALE; MOTOR COORDINATION; NEUROPROTECTION; NICK END LABELING; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; REPERFUSION INJURY; UPREGULATION; AGE; ANIMAL; BRAIN; DISEASE MODEL; DRUG EFFECTS; FISCHER 344 RAT; ISCHEMIC POSTCONDITIONING; METABOLISM; PATHOLOGY; PROCEDURES; STROKE; TREATMENT OUTCOME;

AGE FACTORS; AGING; ANIMALS; APOPTOSIS; BRAIN; BRAIN ISCHEMIA; DISEASE MODELS, ANIMAL; ISCHEMIC POSTCONDITIONING; MALE; METHYL ETHERS; NEUROPROTECTIVE AGENTS; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; RATS, INBRED F344; STROKE; TREATMENT OUTCOME;

EID: 84903521029     PISSN: 03064522     EISSN: 18737544     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2014.05.064     Document Type: Article

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