Ischemic postconditioning reduces infarct size through the α1-adrenergic receptor pathway

Journal of Cardiovascular Pharmacology

Volumn 63, Issue 6, 2014, Pages 504-511

  Buchholz, Bruno ^a   D́annunzio, Verónica ^a   Giani, Jorge F ^b   Siachoque, Nadezda ^a   Dominici, Fernando P ^b   Turyn, Daniel ^b   Perez, Virginia ^a   Donato, Martín ^a   Gelpi, Ricardo J ^a  

^a University of Buenos Aires   (Argentina)

^b UNIVERSIDAD DE BUENOS AIRES   (Argentina)

Author keywords

Akt; Glycogen synthase kinase 3 ; Infarct size; Ischemic postconditioning; 1 adrenergic receptor

Indexed keywords

ALPHA 1 ADRENERGIC RECEPTOR; CLONIDINE; GLYCOGEN SYNTHASE KINASE 3BETA; PHENYLEPHRINE; PRAZOSIN; PROTEIN KINASE B; TRIPHENYLTETRAZOLIUM;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; HEART INFARCTION SIZE; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; ISCHEMIC POSTCONDITIONING; ISOLATED HEART; MALE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; RAT; WESTERN BLOTTING; ANIMAL; METABOLISM; MYOCARDIAL INFARCTION; PHYSIOLOGY; PROCEDURES; SIGNAL TRANSDUCTION; WISTAR RAT;

ANIMALS; ISCHEMIC POSTCONDITIONING; MALE; MYOCARDIAL INFARCTION; RATS; RATS, WISTAR; RECEPTORS, ADRENERGIC, ALPHA-1; SIGNAL TRANSDUCTION;

EID: 84902287843     PISSN: 01602446     EISSN: 15334023     Source Type: Journal    
DOI: 10.1097/FJC.0000000000000074     Document Type: Article

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