The histone acetyltransferase MOF overexpression blunts cardiac hypertrophy by targeting ROS in mice

Biochemical and Biophysical Research Communications

Volumn 448, Issue 4, 2014, Pages 379-384

  Qiao, Weiwei ^a   Zhang, Weili ^a   Gai, Yusheng ^a   Zhao, Lan ^a   Fan, Juexin ^a  

^a YANTAISHAN HOSPITAL   (China)

Author keywords

Cardiac hypertrophy; Histone acetyltransferase; MOF; ROS

Indexed keywords

CATALASE; HISTONE ACETYLTRANSFERASE; MALES ABSENT ON THE FIRST; MANGANESE SUPEROXIDE DISMUTASE; MESSENGER RNA; RAF PROTEIN; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; BODY WEIGHT; BONE LENGTH; CONTROLLED STUDY; DOWN REGULATION; GENE EXPRESSION; HEART FAILURE; HEART LEFT VENTRICLE WALL; HEART VENTRICLE HYPERTROPHY; HEART WEIGHT; HUMAN; HUMAN TISSUE; LUNG WEIGHT; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; TIBIA; TRANSGENIC MOUSE; TRANSTHORACIC ECHOCARDIOGRAPHY; WILD TYPE;

MURINAE; MUS; MUS MUSCULUS;

CARDIAC HYPERTROPHY; HISTONE ACETYLTRANSFERASE; MOF; ROS;

ANIMALS; CARDIOMEGALY; DISEASE MODELS, ANIMAL; DOWN-REGULATION; HEART FAILURE; HISTONE ACETYLTRANSFERASES; HUMANS; MALE; MAP KINASE SIGNALING SYSTEM; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; REACTIVE OXYGEN SPECIES; RNA, MESSENGER; UP-REGULATION;

EID: 84901832766     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2014.04.112     Document Type: Article

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