Podocyte-specific RAP1GAP expression contributes to focal segmental glomerulosclerosis-associated glomerular injury

Journal of Clinical Investigation

Volumn 124, Issue 4, 2014, Pages 1757-1769

  Potla, Uma ^a   Ni, Jie ^a   Vadaparampil, Justin ^a   Yang, Guozhe ^a,b   Leventhal, Jeremy S ^a,b   Campbell, Kirk N ^a   Chuang, Peter Y ^a   Morozov, Alexei ^c   He, John C ^a,b   D'Agati, Vivette D ^d   Klotman, Paul E ^e   Kaufman, Lewis ^a,b  

^a MOUNT SINAI SCHOOL OF MEDICINE   (United States)

^b VETERANS AFFAIRS MEDICAL CENTER   (United States)

^c VIRGINIA TECH CARILION SCHOOL OF MEDICINE   (United States)

^d NewYork Presbyterian Hospital   (United States)

^e BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BETA1 INTEGRIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL ADHESION; CONTROLLED STUDY; FOCAL GLOMERULOSCLEROSIS; GENE; GENE INACTIVATION; GENE OVEREXPRESSION; GENE RAP1GAP; GLOMERULAR INJURY; GLOMERULOSCLEROSIS; HAPLOINSUFFICIENCY; HUMAN; HUMAN TISSUE; INJURY; KIDNEY BIOPSY; MOUSE; MOUSE MODEL; MURINE MODEL; MUTAGENICITY; NONHUMAN; PODOCYTE; PREMATURITY; PRIORITY JOURNAL;

ANIMALS; ANTIGENS, CD29; GLOMERULOSCLEROSIS, FOCAL SEGMENTAL; GTPASE-ACTIVATING PROTEINS; HAPLOINSUFFICIENCY; HUMANS; KIDNEY GLOMERULUS; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; PODOCYTES; RAP GTP-BINDING PROTEINS; RAP1 GTP-BINDING PROTEINS; RNA, MESSENGER;

EID: 84897475640     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI67846     Document Type: Article

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