Prenatal vitamin C deficiency results in differential levels of oxidative stress during late gestation in foetal guinea pig brains

Redox Biology

Volumn 2, Issue 1, 2014, Pages 361-367

  Paidi, Maya D ^a   Schjoldager, Janne G ^a   Lykkesfeldt, Jens ^a   Tveden Nyborg, Pernille ^a  

^a UNIVERSITY OF COPENHAGEN   (Denmark)

Author keywords

Brain; Deficiency; Development; Guinea pig; Oxidative stress; Vitamin C

Indexed keywords

3 NITROTYROSINE; 4 HYDROXYNONENAL; ALPHA TOCOPHEROL; CASPASE 3; MALONALDEHYDE; SUPEROXIDE DISMUTASE; ANTIOXIDANT;

ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; ASCORBIC ACID DEFICIENCY; BRAIN CORTEX; BRAIN DEVELOPMENT; BRAIN LEVEL; BRAIN MATURATION; BRAIN PROTECTION; CONTROLLED STUDY; ENZYME ACTIVITY; FEMALE; FETUS; FETUS DEVELOPMENT; GENE EXPRESSION; GESTATIONAL AGE; GUINEA PIG; LIPID PEROXIDATION; MATERNAL NUTRITION; MOLECULAR DYNAMICS; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN CLEAVAGE; PROTEIN EXPRESSION; PROTEIN MODIFICATION; REGULATORY MECHANISM; SIGNAL TRANSDUCTION; ANIMAL MODEL; CASPASE 3 GENE; NUCLEOTIDE SEQUENCE; ANIMAL; BRAIN; EMBRYOLOGY; GENE EXPRESSION REGULATION; METABOLISM; PATHOLOGY; PREGNANCY; PRENATAL INJURY;

CAVIA;

ANIMALS; ANTIOXIDANTS; APOPTOSIS; ASCORBIC ACID DEFICIENCY; BRAIN; FEMALE; GENE EXPRESSION REGULATION, DEVELOPMENTAL; GUINEA PIGS; OXIDATIVE STRESS; PREGNANCY; PRENATAL INJURIES; SIGNAL TRANSDUCTION;

EID: 84894025117     PISSN: 22132317     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.redox.2014.01.009     Document Type: Article

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