Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis

Infection and Immunity

Volumn 82, Issue 2, 2014, Pages 532-543

  Peters, Brian M ^a   Palmer, Glen E ^b   Nash, Andrea K ^a   Lilly, Elizabeth A ^a   Fidel, Paul L ^a,b   Noverra, Mairi C ^a,b  

^a LOUISIANA STATE UNIVERSITY HEALTH SCIENCES CENTER   (United States)

^b LOUISIANA STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

INTERLEUKIN 1BETA; LACTATE DEHYDROGENASE; TRANSCRIPTION FACTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CANDIDA ALBICANS; ENZYME ACTIVITY; FEMALE; FUNGAL STRAIN; FUNGAL VIRULENCE; FUNGUS HYPHAE; GENE; GENE DELETION; GENE OVEREXPRESSION; IMMUNOPATHOGENESIS; IMMUNOPATHOLOGY; IN VIVO STUDY; INFLAMMATION; INNATE IMMUNITY; INOCULATION; LAVAGE; MICROBIAL COLONIZATION; MORPHOGENESIS; MOUSE; MUCOSAL IMMUNITY; NEUTROPHIL; NONHUMAN; NRG1 GENE; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; UME6 GENE; VAGINA MYCOSIS;

ANIMALS; CANDIDA ALBICANS; CANDIDIASIS, VULVOVAGINAL; CYTOKINES; DISEASE MODELS, ANIMAL; FEMALE; GENE DELETION; L-LACTATE DEHYDROGENASE; LONGITUDINAL STUDIES; MICE; MICE, INBRED C57BL; NEUTROPHILS; TRANSCRIPTION FACTORS; VAGINAL DOUCHING; VIRULENCE FACTORS;

EID: 84893002772     PISSN: 00199567     EISSN: 10985522     Source Type: Journal    
DOI: 10.1128/IAI.01417-13     Document Type: Article

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