Calpain-mediated proteolysis of polycystin-1 C-terminus induces JAK2 and ERK signal alterations

Experimental Cell Research

Volumn 320, Issue 1, 2014, Pages 62-68

  Kim, Hyunho ^a,b   Kang, Ah Young ^a,c   Ko, Ah ra ^d   Park, Hayne Cho ^a,e   So, Insuk ^a   Park, Jong Hoon ^f   Cheong, Hae Il ^a,e,g   Hwang, Young Hwan ^e,h   Ahn, Curie ^a,c,e  

^a Seoul National University College of Medicine   (South Korea)

^b UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^c Seoul National University   (South Korea)

^d Samsung Medical Center   (South Korea)

^e Seoul National University Hospital   (South Korea)

^f Sookmyung Women's University   (South Korea)

^g Seoul National University Children's Hospital   (South Korea)

^h Eulji General Hospital   (South Korea)

Author keywords

Calcium homeostasis; Calpain; PEST; Polycystic kidney disease; Polycystin 1

Indexed keywords

CALCIMYCIN; CALPAIN 1; CALPAIN 2; CALPASTATIN; CALPEPTIN; JANUS KINASE 2; MITOGEN ACTIVATED PROTEIN KINASE; POLYCYSTIN 1;

ANIMAL CELL; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CARBOXY TERMINAL SEQUENCE; CELL POLARITY; CELL PROLIFERATION; CELL STRAIN HEK293; IN VITRO STUDY; INTRACELLULAR SIGNALING; MOUSE; NONHUMAN; POLYACRYLAMIDE GEL ELECTROPHORESIS; PRIORITY JOURNAL; PROTEIN DEGRADATION; WESTERN BLOTTING;

EID: 84888198672     PISSN: 00144827     EISSN: 10902422     Source Type: Journal    
DOI: 10.1016/j.yexcr.2013.10.012     Document Type: Article

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