IRAK1-dependent signaling mediates mortality in polymicrobial sepsis

Inflammation

Volumn 36, Issue 6, 2013, Pages 1503-1512

  Chandra, Rachna ^a   Federici, Stephanie ^a   Bishwas, Tripti ^a   Németh, Zoltán H ^a,b   Deitch, Edwin A ^a   Thomas, James A ^c   Spolarics, Zoltán ^a  

^a RUTGERS NEW JERSEY MEDICAL SCHOOL   (United States)

^b MORRISTOWN MEDICAL CENTER   (United States)

^c UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

infection; interleukin; macrophages; signaling; survival; TLR

Indexed keywords

INTERLEUKIN 1 RECEPTOR ASSOCIATED KINASE 1; INTERLEUKIN 10; INTERLEUKIN 1BETA; INTERLEUKIN 6; TOLL LIKE RECEPTOR 7; TOLL LIKE RECEPTOR 8;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; B LYMPHOCYTE; CELL ISOLATION; CONTROLLED STUDY; DOWN REGULATION; EX VIVO STUDY; FLOW CYTOMETRY; GRANULOCYTOSIS; LIGATION; LYMPHOCYTE DEPLETION; MALE; MORTALITY; MOUSE; MOUSE STRAIN; NONHUMAN; PERITONEUM MACROPHAGE; PERITONITIS; PROTEIN DEFICIENCY; PUNCTURE; SEPSIS; SIGNAL TRANSDUCTION; SPLEEN CELL; WILD TYPE;

MUS; RODENTIA;

ANIMALS; B-LYMPHOCYTES; COINFECTION; DOWN-REGULATION; INFLAMMATION; INTERLEUKIN-1 RECEPTOR-ASSOCIATED KINASES; INTERLEUKIN-10; INTERLEUKIN-1BETA; INTERLEUKIN-6; MACROPHAGES; MALE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; SEPSIS; SIGNAL TRANSDUCTION; TOLL-LIKE RECEPTOR 4;

EID: 84888198099     PISSN: 03603997     EISSN: 15732576     Source Type: Journal    
DOI: 10.1007/s10753-013-9692-1     Document Type: Article

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