Nitric oxide induces apoptosis associated with TRPV1 channel-mediated Ca2+ entry via S-nitrosylation in osteoblasts

European Journal of Pharmacology

Volumn 715, Issue 1-3, 2013, Pages 280-285

  Pan, Leiting ^a   Song, Kun ^a   Hu, Fen ^a   Sun, Wenwu ^b   Lee, Imshik ^a  

^a NANKAI UNIVERSITY   (China)

^b GENERAL HOSPITAL OF NORTHERN THEATER COMMAND   (China)

Author keywords

Apoptosis; Nitric oxide; Osteoblast; S nitrosylation; TRPV1

Indexed keywords

CALCIUM ION; CAPSAZEPINE; CYTOKINE; N ETHYLMALEIMIDE; NIFEDIPINE; NITRIC OXIDE; NITROPRUSSIDE SODIUM; RUTHENIUM RED; VANILLOID RECEPTOR 1;

ANIMAL CELL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CALCIUM CELL LEVEL; CELL VIABILITY; CONCENTRATION RESPONSE; CONTROLLED STUDY; IN VITRO STUDY; MITOCHONDRION; NITROSYLATION; NONHUMAN; OSTEOBLAST; OSTEOPOROSIS; PRIORITY JOURNAL; RAT;

APOPTOSIS; CA(2+); NITRIC OXIDE; OSTEOBLAST; S-NITROSYLATION; TRPV1;

ANIMALS; APOPTOSIS; BIOLOGICAL TRANSPORT; CALCIUM; CALCIUM SIGNALING; GENE EXPRESSION REGULATION; MITOCHONDRIA; NITRIC OXIDE; NITROPRUSSIDE; OSTEOBLASTS; RATS; RATS, WISTAR; S-NITROSOTHIOLS; TRPV CATION CHANNELS;

EID: 84885477784     PISSN: 00142999     EISSN: 18790712     Source Type: Journal    
DOI: 10.1016/j.ejphar.2013.05.009     Document Type: Article

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