Delayed post-injury administration of C5a improves regeneration and functional recovery after spinal cord injury in mice

Clinical and Experimental Immunology

Volumn 174, Issue 2, 2013, Pages 318-325

  Guo, Q ^a   Cheng, J ^b   Zhang, J ^a   Su, B ^c   Bian, C ^a   Lin, S ^c   Zhong, C ^a  

^a THIRD MILITARY MEDICAL UNIVERSITY   (China)

^b CHONGQING NORMAL UNIVERSITY   (China)

^c CHENGDU MEDICAL COLLEGE   (China)

Author keywords

Apoptosis; Complement; Neurite outgrowth; Neuroprotection; Spinal cord injury

Indexed keywords

CASPASE 3; COMPLEMENT COMPONENT C5A; COMPLEMENT COMPONENT C5A RECEPTOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; CONVALESCENCE; FEMALE; HISTOPATHOLOGY; HUMAN; HUMAN TISSUE; LOCOMOTION; MOUSE; NERVE CELL; NERVE FIBER GROWTH; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; RECEPTOR BINDING; SPINAL CORD INJURY; SPINAL CORD REGENERATION;

APOPTOSIS; COMPLEMENT; NEURITE OUTGROWTH; NEUROPROTECTION; SPINAL CORD INJURY;

ANIMALS; APOPTOSIS; CELL GROWTH PROCESSES; CELLS, CULTURED; COMPLEMENT ACTIVATION; COMPLEMENT C5A; DISEASE MODELS, ANIMAL; DRUG ADMINISTRATION SCHEDULE; FEMALE; HUMANS; INJECTIONS, INTRAPERITONEAL; MICE; MICE, INBRED C57BL; MOTOR ACTIVITY; NEURONS; RECOVERY OF FUNCTION; SPINAL CORD INJURIES; SPINAL CORD REGENERATION;

EID: 84885165115     PISSN: 00099104     EISSN: 13652249     Source Type: Journal    
DOI: 10.1111/cei.12175     Document Type: Article

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