Evidence for a role of Nav1.6 in facilitating increases in neuronal hyperexcitability during epileptogenesis

Journal of Neurophysiology

Volumn 110, Issue 5, 2013, Pages 1144-1157

  Hargus, Nicholas J ^a   Nigam, Aradhya ^a   Bertram III, Edward H ^a   Patel, Manoj K ^a  

^a UNIVERSITY OF VIRGINIA HEALTH SYSTEM   (United States)

Author keywords

Entorhinal cortex; Sodium channels; Status epilepticus

Indexed keywords

SODIUM CHANNEL NAV1.6; TETRODOTOXIN; ANHYDROTETRODOTOXIN; DRUG DERIVATIVE; SODIUM; SODIUM CHANNEL; SODIUM CHANNEL BLOCKING AGENT;

ACTION POTENTIAL; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL FUNCTION; CELL SIZE; CONTROLLED STUDY; ENTORHINAL CORTEX; EPILEPTIC DISCHARGE; EPILEPTIC STATE; EPILEPTOGENESIS; IMMUNOCYTOCHEMISTRY; LATENT PERIOD; MALE; NERVE CELL STIMULATION; NONHUMAN; PRIORITY JOURNAL; RAT; STEADY STATE; ANIMAL; DRUG EFFECT; EPILEPSY; IN VITRO STUDY; METABOLISM; NERVE CELL; PATHOPHYSIOLOGY; PHYSIOLOGY; SPRAGUE DAWLEY RAT; TIME; ANALOGS AND DERIVATIVES; DRUG EFFECTS;

ENTORHINAL CORTEX; SODIUM CHANNELS; STATUS EPILEPTICUS;

ANIMALS; ENTORHINAL CORTEX; EPILEPSY; MALE; NAV1.6 VOLTAGE-GATED SODIUM CHANNEL; NEURONS; RATS; RATS, SPRAGUE-DAWLEY; SODIUM; SODIUM CHANNEL BLOCKERS; TETRODOTOXIN; TIME FACTORS; IN VITRO TECHNIQUES;

EID: 84883320254     PISSN: 00223077     EISSN: 15221598     Source Type: Journal    
DOI: 10.1152/jn.00383.2013     Document Type: Article

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