TMEM16C facilitates Na + -activated K + currents in rat sensory neurons and regulates pain processing

Nature Neuroscience

Volumn 16, Issue 9, 2013, Pages 1284-1290

  Huang, Fen ^a,b   Wang, Xidao ^a   Ostertag, Eric M ^c   Nuwal, Tulip ^a   Huang, Bo ^a   Jan, Yuh Nung ^a,b   Basbaum, Allan I ^a   Jan, Lily Yeh ^a,b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b HOWARD HUGHES MEDICAL INSTITUTE   (United States)

^c Transposagen Biopharmaceuticals Inc   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ISOLECTIN B4; MEMBRANE PROTEIN; POTASSIUM CHANNEL; POTASSIUM ION; SLACK PROTEIN; SMALL INTERFERING RNA; SODIUM ION; TMEM16C PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL STRAIN HEK293; CHANNEL GATING; CONTROLLED STUDY; FEMALE; GENE SILENCING; HUMAN; HUMAN CELL; IMMUNOCYTOCHEMISTRY; IN VIVO STUDY; NERVE CELL EXCITABILITY; NERVE POTENTIAL; NOCICEPTION; NONHUMAN; PAIN RECEPTOR; POTASSIUM CURRENT; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; RAT; SENSORY NERVE CELL; SPINAL GANGLION;

ANIMALS; CADMIUM; CELLS, CULTURED; CHLORIDE CHANNELS; GANGLIA, SPINAL; GENE EXPRESSION REGULATION; HUMANS; HYPERALGESIA; MEMBRANE POTENTIALS; MICE; MUTATION; NERVE TISSUE PROTEINS; NEW BRUNSWICK; PAIN MEASUREMENT; PAIN THRESHOLD; POTASSIUM CHANNELS; RATS; RATS, TRANSGENIC; RNA, SMALL INTERFERING; SENSORY RECEPTOR CELLS; SODIUM; SODIUM CHLORIDE; SPINAL CORD;

EID: 84883313607     PISSN: 10976256     EISSN: 15461726     Source Type: Journal    
DOI: 10.1038/nn.3468     Document Type: Article

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