MiR-21 is overexpressed in response to high glucose and protects endothelial cells from apoptosis

Experimental and Clinical Endocrinology and Diabetes

Volumn 121, Issue 7, 2013, Pages 425-430

  Zeng, J ^a   Xiong, Y ^a   Li, G ^a   Liu, M ^a   He, T ^a   Tang, Y ^a   Chen, Y ^a   Cai, L ^a   Jiang, R ^a   Tao, J ^a  

^a SICHUAN PROVINCIAL PEOPLE'S HOSPITAL   (China)

Author keywords

apoptosis; diabetes; endothelial dysfunction; high glucose; miR 21

Indexed keywords

GLUCOSE; MICRORNA 21;

ADULT; AGED; APOPTOSIS; ARTICLE; BLOOD SAMPLING; CELL PROLIFERATION; CELL PROTECTION; CLINICAL ARTICLE; CYTOTOXICITY; DIABETES MELLITUS; DIABETIC PATIENT; ENDOTHELIAL DYSFUNCTION; ENDOTHELIAL PROGENITOR CELL; ENDOTHELIUM CELL; FEMALE; GLUCOSE BLOOD LEVEL; HUMAN; MALE; OXIDATIVE STRESS; PATHOGENESIS; PRIORITY JOURNAL; PUBLIC HEALTH PROBLEM; VASCULAR DISEASE;

ADAPTOR PROTEINS, SIGNAL TRANSDUCING; AGED; AGED, 80 AND OVER; ANNEXIN A2; APOPTOSIS; CELLS, CULTURED; DIABETIC ANGIOPATHIES; ENDOTHELIAL CELLS; ENDOTHELIUM, VASCULAR; FEMALE; GLUCOSE; HUMANS; MALE; MICRORNAS; MIDDLE AGED; NUCLEAR PROTEINS; S100 PROTEINS; STEM CELLS; SUPEROXIDE DISMUTASE; SWEETENING AGENTS; THIOREDOXINS;

EID: 84880061862     PISSN: 09477349     EISSN: 14393646     Source Type: Journal    
DOI: 10.1055/s-0033-1345169     Document Type: Article

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