Dying to live: How the death modality of the infected macrophage modulates immunity to Tuberculosis

Advances in Experimental Medicine and Biology

Volumn 783, Issue , 2013, Pages 103-120

  Divangahi, Maziar ^a   Behar, Samuel M ^b   Remold, Heinz ^b  

^a MCGILL UNIVERSITY   (Canada)

^b BRIGHAM AND WOMEN S HOSPITAL   (United States)

Author keywords

Apoptosis; B cell lymphoma 2 (Bcl 2); BCG vaccine; Bcl 2 associated X Protein (BAX); Bcl 2 homologous antagonist killer (BAK); BH3 interacting domain (BID); Cell death program; Eicosanoids; Extrinsic pathway; FLICE inhibitory protein (FLIPS); Intrinsic pathway; Lipoxins (LX); Macrophages; Mitochondrial outer membrane permeabilization (MOMP); Mitochondrial permeability transition (MPT); Mycobacterial antigens; Mycobacterium Tuberculosis (Mtb); Necrosis; Plasma membrane microdisruptions; Prostaglandins (PG); T cell response

Indexed keywords

ARACHIDONATE 5 LIPOXYGENASE; EARLY SECRETORY ANTIGENIC TARGET 6; ICOSANOID; LIPOXIN A; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; MYCOBACTERIUM ANTIGEN; PROSTAGLANDIN E2; APOPTOSIS REGULATORY PROTEIN; BACTERIAL ANTIGEN; LIPOXIN; PROSTAGLANDIN E RECEPTOR;

ADAPTIVE IMMUNITY; ANTIGEN PRESENTATION; APOPTOSIS; ARTICLE; BACTERIAL GROWTH; BACTERIAL VIABILITY; BACTERIAL VIRULENCE; CD4+ T LYMPHOCYTE; CD8+ T LYMPHOCYTE; CROSS PRESENTATION; DENDRITIC CELL; HUMAN; IMMUNE EVASION; IMMUNOMODULATION; INNATE IMMUNITY; LUNG TUBERCULOSIS; MACROPHAGE; MEMBRANE DAMAGE; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL PERMEABILITY; MYCOBACTERIUM TUBERCULOSIS; NECROSIS; NONHUMAN; PRIORITY JOURNAL; PROSTAGLANDIN SYNTHESIS; TUBERCULOSIS; ANIMAL; BACTERIAL SECRETION SYSTEM; BIOLOGICAL MODEL; CELL MEMBRANE; GENETICS; IMMUNOLOGY; MICROBIOLOGY; MITOCHONDRION; MOUSE; MOUSE MUTANT; PATHOGENICITY; PATHOLOGY; PHYSIOLOGY; REVIEW; VIRULENCE;

ANIMALS; ANTIGEN PRESENTATION; ANTIGENS, BACTERIAL; APOPTOSIS; APOPTOSIS REGULATORY PROTEINS; BACTERIAL SECRETION SYSTEMS; CELL MEMBRANE; DENDRITIC CELLS; DINOPROSTONE; HUMANS; IMMUNE EVASION; LIPOXINS; MACROPHAGES; MICE; MICE, KNOCKOUT; MITOCHONDRIA; MODELS, IMMUNOLOGICAL; MYCOBACTERIUM TUBERCULOSIS; NECROSIS; RECEPTORS, PROSTAGLANDIN E; TUBERCULOSIS; VIRULENCE;

MLCS; MLOWN;

EID: 84879533832     PISSN: 00652598     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-1-4614-6111-1_6     Document Type: Article

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