-
1
-
-
0033552883
-
Atherosclerosis - An inflammatory disease
-
DOI 10.1056/NEJM199901143400207
-
Ross R., Atherosclerosis-an inflammatory disease. New England Journal of Medicine 1999 340 2 115 126 2-s2.0-0033552883 10.1056/NEJM199901143400207 (Pubitemid 29054847)
-
(1999)
New England Journal of Medicine
, vol.340
, Issue.2
, pp. 115-126
-
-
Ross, R.1
-
2
-
-
82555161682
-
Redox imbalance of red blood cells impacts T lymphocyte homeostasis: Implication in carotid atherosclerosis
-
10.1160/TH11-02-0110
-
Profumo E., Buttari B., Petrone L., Redox imbalance of red blood cells impacts T lymphocyte homeostasis: implication in carotid atherosclerosis. Thrombosis and Haemostasis 2011 106 6 1117 1126 10.1160/TH11-02-0110
-
(2011)
Thrombosis and Haemostasis
, vol.106
, Issue.6
, pp. 1117-1126
-
-
Profumo, E.1
Buttari, B.2
Petrone, L.3
-
3
-
-
84865504765
-
Fueling the flame: Bioenergy couples metabolism and inflammation
-
Liu T. F., Brown C. M., El Gazzar M., Fueling the flame: bioenergy couples metabolism and inflammation. Journal of Leukocyte Biology 2012 92 3 49 507
-
(2012)
Journal of Leukocyte Biology
, vol.92
, Issue.3
, pp. 49-507
-
-
Liu, T.F.1
Brown, C.M.2
El Gazzar, M.3
-
4
-
-
0036549988
-
Lipid oxidation products and atherosclerosis
-
DOI 10.1016/S1537-1891(02)00168-4, PII S1537189102001684
-
Berliner J., Lipid oxidation products and atherosclerosis. Vascular Pharmacology 2002 38 4 187 191 2-s2.0-0036549988 10.1016/S1537-1891(02)00168-4 (Pubitemid 36398264)
-
(2002)
Vascular Pharmacology
, vol.38
, Issue.4
, pp. 187-191
-
-
Berliner, J.1
-
5
-
-
11144225245
-
Oxidative stress and vascular disease
-
DOI 10.1161/01.ATV.0000150649.39934.13
-
Madamanchi N. R., Vendrov A., Runge M. S., Oxidative stress and vascular disease. Arteriosclerosis, Thrombosis, and Vascular Biology 2005 25 1 29 38 2-s2.0-11144225245 10.1161/01.ATV.0000150649.39934.13 (Pubitemid 40054251)
-
(2005)
Arteriosclerosis, Thrombosis, and Vascular Biology
, vol.25
, Issue.1
, pp. 29-38
-
-
Madamanchi, N.R.1
Vendrov, A.2
Runge, M.S.3
-
6
-
-
5344238178
-
Paraoxonases 1, 2, and 3, oxidative stress, and macrophage foam cell formation during atherosclerosis development
-
DOI 10.1016/j.freeradbiomed.2004.06.030, PII S0891584904005246
-
Aviram M., Rosenblat M., Paraoxonases 1, 2, and 3, oxidative stress, and macrophage foam cell formation during atherosclerosis development. Free Radical Biology and Medicine 2004 37 9 1304 1316 2-s2.0-5344238178 10.1016/j. freeradbiomed.2004.06.030 (Pubitemid 39349234)
-
(2004)
Free Radical Biology and Medicine
, vol.37
, Issue.9
, pp. 1304-1316
-
-
Aviram, M.1
Rosenblat, M.2
-
7
-
-
61449118867
-
The paraoxonases: Role in human diseases and methodological difficulties in measurement
-
2-s2.0-61449118867 10.1080/10408360802610878
-
Camps J., Marsillach J., Joven J., The paraoxonases: role in human diseases and methodological difficulties in measurement. Critical Reviews in Clinical Laboratory Sciences 2009 46 2 83 106 2-s2.0-61449118867 10.1080/10408360802610878
-
(2009)
Critical Reviews in Clinical Laboratory Sciences
, vol.46
, Issue.2
, pp. 83-106
-
-
Camps, J.1
Marsillach, J.2
Joven, J.3
-
8
-
-
17644367506
-
Structure-reactivity studies of serum paraoxonase PON1 suggest that its native activity is lactonase
-
DOI 10.1021/bi047440d
-
Khersonsky O., Tawfik D. S., Structure-reactivity studies of serum paraoxonase PON1 suggest that its native activity is lactonase. Biochemistry 2005 44 16 6371 6382 2-s2.0-17644367506 10.1021/bi047440d (Pubitemid 40570731)
-
(2005)
Biochemistry
, vol.44
, Issue.16
, pp. 6371-6382
-
-
Khersonsky, O.1
Tawfik, D.S.2
-
9
-
-
79551664540
-
Immunohistochemical analysis of paraoxonases-1 and 3 in human atheromatous plaques
-
2-s2.0-79551664540 10.1111/j.1365-2362.2010.02411.x
-
Marsillach J., Camps J., Beltran-Debón R., Rull A., Aragones G., Maestre-Martínez C., Sabench F., Hernández M., Castillo D. D., Joven J., Mackness M., Mackness B., Immunohistochemical analysis of paraoxonases-1 and 3 in human atheromatous plaques. European Journal of Clinical Investigation 2011 41 3 308 314 2-s2.0-79551664540 10.1111/j.1365-2362.2010. 02411.x
-
(2011)
European Journal of Clinical Investigation
, vol.41
, Issue.3
, pp. 308-314
-
-
Marsillach, J.1
Camps, J.2
Beltran-Debón, R.3
Rull, A.4
Aragones, G.5
Maestre-Martínez, C.6
Sabench, F.7
Hernández, M.8
Castillo, D.D.9
Joven, J.10
MacKness, M.11
MacKness, B.12
-
10
-
-
0032537833
-
Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis
-
DOI 10.1038/28406
-
Shih D. M., Gu L., Xia Y. R., Navab M., Li W. F., Hama S., Castellani L. W., Furlong C. E., Costa L. G., Fogelman A. M., Lusis A. J., Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis. Nature 1998 394 6690 284 287 2-s2.0-0032537833 10.1038/28406 (Pubitemid 28335041)
-
(1998)
Nature
, vol.394
, Issue.6690
, pp. 284-287
-
-
Shih, D.M.1
Gu, L.2
Xia, Y.-R.3
Navab, M.4
Li, W.-F.5
Hama, S.6
Castellani, L.W.7
Furlong, C.E.8
Costa, L.G.9
Fogelman, A.M.10
Lusis, A.J.11
-
11
-
-
2442575983
-
Paraoxonase-1 inhibits oxidised LDL-induced MCP-1 production by endothelial cells
-
DOI 10.1016/j.bbrc.2004.04.056, PII S0006291X04007806
-
Mackness B., Hine D., Liu Y., Mastorikou M., Mackness M., Paraoxonase-1 inhibits oxidised LDL-induced MCP-1 production by endothelial cells. Biochemical and Biophysical Research Communications 2004 318 3 680 683 2-s2.0-2442575983 10.1016/j.bbrc.2004.04.056 (Pubitemid 38624207)
-
(2004)
Biochemical and Biophysical Research Communications
, vol.318
, Issue.3
, pp. 680-683
-
-
Mackness, B.1
Hine, D.2
Liu, Y.3
Mastorikou, M.4
Mackness, M.5
-
12
-
-
0000001577
-
Lipoprotein analysis for clinical studies
-
Oxford, UK IRL Press
-
Mackness M. I., Durrington P. N., Converse C. A., Skinner E. R., Lipoprotein analysis for clinical studies. Lipoprotein Analysis: A Practical Approach 1992 Oxford, UK IRL Press 1 42
-
(1992)
Lipoprotein Analysis: A Practical Approach
, pp. 1-42
-
-
MacKness, M.I.1
Durrington, P.N.2
Converse, C.A.3
Skinner, E.R.4
-
13
-
-
77952048387
-
Decreased paraoxonase-1 activity is associated with alterations of high-density lipoprotein particles in chronic liver impairment
-
2-s2.0-77952048387 10.1186/1476-511X-9-46
-
Marsillach J., Aragones G., Mackness B., Mackness M., Rull A., Beltran-Debon R., Pedro-Botet J., Alonso-Villaverde C., Joven J., Camps J., Decreased paraoxonase-1 activity is associated with alterations of high-density lipoprotein particles in chronic liver impairment. Lipids in Health and Disease 2010 9 46 2-s2.0-77952048387 10.1186/1476-511X-9-46
-
(2010)
Lipids in Health and Disease
, vol.9
, pp. 46
-
-
Marsillach, J.1
Aragones, G.2
MacKness, B.3
MacKness, M.4
Rull, A.5
Beltran-Debon, R.6
Pedro-Botet, J.7
Alonso-Villaverde, C.8
Joven, J.9
Camps, J.10
-
14
-
-
84864612118
-
Biomarkers of inflammation, immunosuppression and stress with active disease are revealed by metabolomic profiling of tuberculosis patients
-
e40221
-
Weiner J. III, Parida S. K., Maertzdorf J., Biomarkers of inflammation, immunosuppression and stress with active disease are revealed by metabolomic profiling of tuberculosis patients. PLoS One 2012 7 7 e40221
-
(2012)
PLoS One
, vol.7
, Issue.7
-
-
Iii, W.J.1
Parida, S.K.2
Maertzdorf, J.3
-
15
-
-
0030056027
-
Oxidized low density lipoprotein inhibits lipopolysaccharide-induced binding of nuclear factor-κB to DNA and the subsequent expression of tumor necrosis factor-α and interleukin-1β in macrophages
-
Ohisson B. G., Englund M. C. O., Karlsson A. L. K., Knutsen E., Erixon C., Skribeck H., Liu Y., Bondjers G., Wiklund O., Oxidized low density lipoprotein inhibits lipopolysaccharide-induced binding of nuclear factor- B to DNA and the subsequent expression of tumor necrosis factor- α and interleukin-1 β in macrophages. Journal of Clinical Investigation 1996 98 1 78 89 2-s2.0-0030056027 (Pubitemid 26243823)
-
(1996)
Journal of Clinical Investigation
, vol.98
, Issue.1
, pp. 78-89
-
-
Ohisson, B.G.1
Englund, M.C.O.2
Karlsson, A.-L.K.3
Knutsen, E.4
Erixon, C.5
Skribeck, H.6
Liu, Y.7
Bondjers, G.8
Wiklund, O.9
-
16
-
-
57649087483
-
The measurement of the lactonase activity of paraoxonase-1 in the clinical evaluation of patients with chronic liver impairment
-
2-s2.0-57649087483 10.1016/j.clinbiochem.2008.09.120
-
Marsillach J., Aragonès G., Beltrán R., Caballeria J., Pedro-Botet J., Morcillo-Suárez C., Navarro A., Joven J., Camps J., The measurement of the lactonase activity of paraoxonase-1 in the clinical evaluation of patients with chronic liver impairment. Clinical Biochemistry 2009 42 1-2 91 98 2-s2.0-57649087483 10.1016/j.clinbiochem.2008.09.120
-
(2009)
Clinical Biochemistry
, vol.42
, Issue.1-2
, pp. 91-98
-
-
Marsillach, J.1
Aragonès, G.2
Beltrán, R.3
Caballeria, J.4
Pedro-Botet, J.5
Morcillo-Suárez, C.6
Navarro, A.7
Joven, J.8
Camps, J.9
-
17
-
-
28844462418
-
Role of poly(ADP-ribose) polymerase-1 activation in the pathogenesis of diabetic complications: Endothelial dysfunction, as a common underlying theme
-
DOI 10.1089/ars.2005.7.1568
-
Pacher P., Szabó C., Role of poly(ADP-ribose) polymerase-1 activation in the pathogenesis of diabetic complications: endothelial dysfunction, as a common underlying theme. Antioxidants and Redox Signaling 2005 7 11-12 1568 1580 2-s2.0-28844462418 10.1089/ars.2005.7.1568 (Pubitemid 41767992)
-
(2005)
Antioxidants and Redox Signaling
, vol.7
, Issue.11-12
, pp. 1568-1580
-
-
Pacher, P.1
Szabo, C.2
-
18
-
-
79955753583
-
Regulation of endothelial function by mitochondrial reactive oxygen species
-
2-s2.0-79955753583 10.1089/ars.2010.3642
-
Widlansky M. E., Gutterman D. D., Regulation of endothelial function by mitochondrial reactive oxygen species. Antioxidants and Redox Signaling 2011 15 6 1517 1530 2-s2.0-79955753583 10.1089/ars.2010.3642
-
(2011)
Antioxidants and Redox Signaling
, vol.15
, Issue.6
, pp. 1517-1530
-
-
Widlansky, M.E.1
Gutterman, D.D.2
-
19
-
-
77950334145
-
Circulating endothelial cells are elevated in patients with type 1 diabetes mellitus
-
2-s2.0-77950334145 10.1530/EJE-09-0795
-
Asicioglu E., Yavuz D. G., Koc M., Ozben B., Yazici D., Deyneli O., Akalin S., Circulating endothelial cells are elevated in patients with type 1 diabetes mellitus. European Journal of Endocrinology 2010 162 4 711 717 2-s2.0-77950334145 10.1530/EJE-09-0795
-
(2010)
European Journal of Endocrinology
, vol.162
, Issue.4
, pp. 711-717
-
-
Asicioglu, E.1
Yavuz, D.G.2
Koc, M.3
Ozben, B.4
Yazici, D.5
Deyneli, O.6
Akalin, S.7
-
20
-
-
43049162677
-
Paraoxonase 1 (PON1) attenuates diabetes development in mice through its antioxidative properties
-
2-s2.0-43049162677 10.1016/j.freeradbiomed.2008.02.012
-
Rozenberg O., Shiner M., Aviram M., Hayek T., Paraoxonase 1 (PON1) attenuates diabetes development in mice through its antioxidative properties. Free Radical Biology and Medicine 2008 44 11 1951 1959 2-s2.0-43049162677 10.1016/j.freeradbiomed.2008.02.012
-
(2008)
Free Radical Biology and Medicine
, vol.44
, Issue.11
, pp. 1951-1959
-
-
Rozenberg, O.1
Shiner, M.2
Aviram, M.3
Hayek, T.4
-
21
-
-
82955195724
-
The antioxidant HDL-associated paraoxonase-1 (PON1) attenuates diabetes development and stimulates β -cell insulin release
-
10.1016/j.atherosclerosis.2011.07.119
-
Koren-Gluzer M., Aviram M., Meilin E., Hayek T., The antioxidant HDL-associated paraoxonase-1 (PON1) attenuates diabetes development and stimulates β -cell insulin release. Atherosclerosis 2011 219 2 510 518 10.1016/j.atherosclerosis.2011.07.119
-
(2011)
Atherosclerosis
, vol.219
, Issue.2
, pp. 510-518
-
-
Koren-Gluzer, M.1
Aviram, M.2
Meilin, E.3
Hayek, T.4
-
22
-
-
84859959656
-
A molecular view on signal transduction by the apoptosome
-
10.1016/j.cellsig.2012.03.007
-
Reubold T. F., Eschenburg S., A molecular view on signal transduction by the apoptosome. Cell Signaling 2012 24 7 1420 1425 10.1016/j.cellsig.2012.03.007
-
(2012)
Cell Signaling
, vol.24
, Issue.7
, pp. 1420-1425
-
-
Reubold, T.F.1
Eschenburg, S.2
-
23
-
-
79959919890
-
Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease
-
2-s2.0-79959919890 10.1172/JCI42946
-
Besler C., Heinrich K., Rohrer L., Doerries C., Riwanto M., Shih D. M., Chroni A., Yonekawa K., Stein S., Schaefer N., Mueller M., Akhmedov A., Daniil G., Manes C., Templin C., Wyss C., Maier W., Tanner F. C., Matter C. M., Corti R., Furlong C., Lusis A. J., Von Eckardstein A., Fogelman A. M., Lüscher T. F., Landmesser U., Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease. Journal of Clinical Investigation 2011 121 7 2693 2708 2-s2.0-79959919890 10.1172/JCI42946
-
(2011)
Journal of Clinical Investigation
, vol.121
, Issue.7
, pp. 2693-2708
-
-
Besler, C.1
Heinrich, K.2
Rohrer, L.3
Doerries, C.4
Riwanto, M.5
Shih, D.M.6
Chroni, A.7
Yonekawa, K.8
Stein, S.9
Schaefer, N.10
Mueller, M.11
Akhmedov, A.12
Daniil, G.13
Manes, C.14
Templin, C.15
Wyss, C.16
Maier, W.17
Tanner, F.C.18
Matter, C.M.19
Corti, R.20
Furlong, C.21
Lusis, A.J.22
Von Eckardstein, A.23
Fogelman, A.M.24
Lüscher, T.F.25
Landmesser, U.26
more..
-
24
-
-
84865468940
-
Cholesterol-induced membrane microvesicles as novel carriers of damage-associated molecular patterns: Mechanisms of formation, action, and detoxification
-
10.1161/ATVBAHA.112.255471
-
Liu M. L., Scalia R., Mehta J. L., Williams K. J., Cholesterol-induced membrane microvesicles as novel carriers of damage-associated molecular patterns: mechanisms of formation, action, and detoxification. Arteriosclerosis Thrombosis and Vascular Biology 2012 32 9 2113 2121 10.1161/ATVBAHA.112.255471
-
(2012)
Arteriosclerosis Thrombosis and Vascular Biology
, vol.32
, Issue.9
, pp. 2113-2121
-
-
Liu, M.L.1
Scalia, R.2
Mehta, J.L.3
Williams, K.J.4
-
25
-
-
84874469648
-
Altered activation of endothelial anti- and pro-apoptotic pathways by high-density lipoprotein from patients with coronary artery disease: Role of HDL-proteome remodeling
-
10.1161/CIRCULATIONAHA.112.108753
-
Riwanto M., Rohrer L., Roschitzki B., Altered activation of endothelial anti- and pro-apoptotic pathways by high-density lipoprotein from patients with coronary artery disease: role of HDL-proteome remodeling. Circulation 2013 127 8 891 904 10.1161/CIRCULATIONAHA.112.108753
-
(2013)
Circulation
, vol.127
, Issue.8
, pp. 891-904
-
-
Riwanto, M.1
Rohrer, L.2
Roschitzki, B.3
-
26
-
-
27944475065
-
Muscle-specific loss of apoptosis-inducing factor leads to mitochondrial dysfunction, skeletal muscle atrophy, and dilated cardiomyopathy
-
DOI 10.1128/MCB.25.23.10261-10272.2005
-
Joza N., Oudit G. Y., Brown D., Bénit P., Kassiri Z., Vahsen N., Benoit L., Patel M. M., Nowikovsky K., Vassault A., Backx P. H., Wada T., Kroemer G., Rustin P., Penninger J. M., Muscle-specific loss of apoptosis-inducing factor leads to mitochondrial dysfunction, skeletal muscle atrophy, and dilated cardiomyopathy. Molecular and Cellular Biology 2005 25 23 10261 10272 2-s2.0-27944475065 10.1128/MCB.25.23.10261-10272.2005 (Pubitemid 41681953)
-
(2005)
Molecular and Cellular Biology
, vol.25
, Issue.23
, pp. 10261-10272
-
-
Joza, N.1
Oudit, G.Y.2
Brown, D.3
Benit, P.4
Kassiri, Z.5
Vahsen, N.6
Benoit, L.7
Patel, M.M.8
Nowikovsky, K.9
Vassault, A.10
Backx, P.H.11
Wada, T.12
Kroemer, G.13
Rustin, P.14
Penninger, J.M.15
-
27
-
-
0033039311
-
Low serum paraoxonase: A risk factor for atherosclerotic disease?
-
DOI 10.1016/S0009-2797(99)00050-2, PII S0009279799000502
-
Mackness M. I., Durrington P. N., Ayub A., Mackness B., Low serum paraoxonase: a risk factor for atherosclerotic disease? Chemico-Biological Interactions 1999 119-120 389 397 2-s2.0-0033039311 10.1016/S0009-2797(99)00050- 2 (Pubitemid 29278575)
-
(1999)
Chemico-Biological Interactions
, vol.119-120
, pp. 389-397
-
-
Mackness, M.I.1
Durrington, P.N.2
Ayub, A.3
Mackness, B.4
-
28
-
-
61349170673
-
Paraoxonase-1 is related to inflammation, fibrosis and PPAR delta in experimental liver disease
-
2-s2.0-61349170673 10.1186/1471-230X-9-3
-
Marsillach J., Camps J., Ferré N., Beltran R., Rull A., Mackness B., Mackness M., Joven J., Paraoxonase-1 is related to inflammation, fibrosis and PPAR delta in experimental liver disease. BMC Gastroenterology 2009 9 3 2-s2.0-61349170673 10.1186/1471-230X-9-3
-
(2009)
BMC Gastroenterology
, vol.9
, Issue.3
-
-
Marsillach, J.1
Camps, J.2
Ferré, N.3
Beltran, R.4
Rull, A.5
MacKness, B.6
MacKness, M.7
Joven, J.8
-
29
-
-
84555196936
-
The anti-inflammatory effect of paraoxonase 1 against oxidized lipids depends on its association with high density lipoproteins
-
10.1016/j.lfs.2011.10.018
-
Loued S., Isabelle M., Berrougui H., Khalil A., The anti-inflammatory effect of paraoxonase 1 against oxidized lipids depends on its association with high density lipoproteins. Life Sciences 2012 90 1-2 82 88 10.1016/j.lfs.2011. 10.018
-
(2012)
Life Sciences
, vol.90
, Issue.1-2
, pp. 82-88
-
-
Loued, S.1
Isabelle, M.2
Berrougui, H.3
Khalil, A.4
-
30
-
-
84155166966
-
Injection of paraoxonase 1 (PON1) to mice stimulates their HDL and macrophage antiatherogenicity
-
10.1002/biof.188
-
Rosenblat M., Volkova N., Aviram M., Injection of paraoxonase 1 (PON1) to mice stimulates their HDL and macrophage antiatherogenicity. Biofactors 2011 37 6 462 467 10.1002/biof.188
-
(2011)
Biofactors
, vol.37
, Issue.6
, pp. 462-467
-
-
Rosenblat, M.1
Volkova, N.2
Aviram, M.3
-
31
-
-
84878113868
-
Paraoxonase 1 (PON1) reduces macrophage inflammatory responses
-
10.1016/j.atherosclerosis.2013.03.005
-
Aharoni S., Aviram M., Fuhrman B., Paraoxonase 1 (PON1) reduces macrophage inflammatory responses. Atherosclerosis 2013 10.1016/j. atherosclerosis.2013.03.005
-
(2013)
Atherosclerosis
-
-
Aharoni, S.1
Aviram, M.2
Fuhrman, B.3
-
32
-
-
84855869657
-
Coincubation of PON1, APO A1, and LCAT increases the time HDL is able to prevent LDL oxidation
-
10.1002/iub.588
-
Hine D., Mackness B., Mackness M., Coincubation of PON1, APO A1, and LCAT increases the time HDL is able to prevent LDL oxidation. IUBMB Life 2012 64 2 157 161 10.1002/iub.588
-
(2012)
IUBMB Life
, vol.64
, Issue.2
, pp. 157-161
-
-
Hine, D.1
MacKness, B.2
MacKness, M.3
|