INOS participates in apoptosis of spinal cord neurons via p-BAD dephosphorylation following ischemia/reperfusion (I/R) injury in rat spinal cord

Neuroscience Letters

Volumn 545, Issue , 2013, Pages 117-122

  Li, Yiming ^a   Gu, Jun ^b   Liu, Yuwen ^a   Long, Hao ^a   Wang, Guannan ^c   Yin, Guoyong ^a   Fan, Jin ^a  

^a THE FIRST AFFILIATED HOSPITAL OF NANJING MEDICAL UNIVERSITY   (China)

^b Xishan People's Hospital   (China)

^c XUZHOU MEDICAL UNIVERSITY   (China)

Author keywords

BAD; INOS; Ischemia reperfusion injury; Spinal cord

Indexed keywords

CYTOCHROME C; INDUCIBLE NITRIC OXIDE SYNTHASE; MESSENGER RNA; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; PROTEIN 14 3 3; PROTEIN BAD;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELLULAR DISTRIBUTION; CONTROLLED STUDY; DIMERIZATION; ENZYME ACTIVITY; ENZYME INHIBITION; GENE EXPRESSION; IMMUNOPRECIPITATION; LUMBAR SPINAL CORD; MALE; MOTOR PERFORMANCE; NEUROLOGIC EXAMINATION; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEPHOSPHORYLATION; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; RAT; REPERFUSION INJURY; SHAM PROCEDURE; SPINAL CORD FUNCTION; SPINAL CORD ISCHEMIA; SPINAL CORD NERVE CELL; TRANSMISSION ELECTRON MICROSCOPY;

ANIMALS; APOPTOSIS; BCL-ASSOCIATED DEATH PROTEIN; MALE; NEURONS; NITRIC OXIDE SYNTHASE TYPE II; PHOSPHORYLATION; RATS; RATS, SPRAGUE-DAWLEY; REPERFUSION INJURY; SPINAL CORD ISCHEMIA;

EID: 84878568494     PISSN: 03043940     EISSN: 18727972     Source Type: Journal    
DOI: 10.1016/j.neulet.2013.04.043     Document Type: Article

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