Fasudil inhibits ER stress-induced VCAM-1 expression by modulating unfolded protein response in endothelial cells

Biochemical and Biophysical Research Communications

Volumn 435, Issue 2, 2013, Pages 171-175

  Kawanami, Daiji ^a   Matoba, Keiichiro ^a   Okada, Rina ^a   Tsukamoto, Masami ^a   Kinoshita, Jun ^a   Ishizawa, Sho ^a   Kanazawa, Yasushi ^a   Yokota, Tamotsu ^a   Utsunomiya, Kazunori ^a  

^a JIKEI UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

Atherosclerosis; ER stress; Fasudil; Rho kinase

Indexed keywords

ACTIVATING TRANSCRIPTION FACTOR 4; CCAAT ENHANCER BINDING PROTEIN; FASUDIL; MITOGEN ACTIVATED PROTEIN KINASE P38; RHO KINASE; TUNICAMYCIN; VASCULAR CELL ADHESION MOLECULE 1;

ARTICLE; CONTROLLED STUDY; ENDOPLASMIC RETICULUM STRESS; ENDOTHELIUM CELL; ENZYME INHIBITION; HUMAN; HUMAN CELL; INFLAMMATION; MOLECULAR MECHANICS; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; UNFOLDED PROTEIN RESPONSE;

1-(5-ISOQUINOLINESULFONYL)-2-METHYLPIPERAZINE; CELLS, CULTURED; ENDOPLASMIC RETICULUM STRESS; ENDOTHELIAL CELLS; HUMANS; PROTEIN FOLDING; PROTEIN KINASE INHIBITORS; RHO-ASSOCIATED KINASES; STRESS, PHYSIOLOGICAL; UNFOLDED PROTEIN RESPONSE; VASCULAR CELL ADHESION MOLECULE-1;

EID: 84878425155     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2013.04.091     Document Type: Article

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