A KCNQ1 mutation causes a high penetrance for familial atrial fibrillation

Journal of Cardiovascular Electrophysiology

Volumn 24, Issue 5, 2013, Pages 562-569

  Bartos, Daniel C ^a   Anderson, Jeffrey B ^b   Bastiaenen, Rachel ^c   Johnson, Jonathan N ^d   Gollob, Michael H ^e   Tester, David J ^d   Burgess, Don E ^a   Homfray, Tessa ^c   Behr, Elijah R ^c   Ackerman, Michael J ^d   Guicheney, Pascale ^f,g   Delisle, Brian P ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b UNIVERSITY OF CINCINNATI   (United States)

^c ST GEORGE'S UNIVERSITY OF LONDON   (United Kingdom)

^d MAYO CLINIC   (United States)

^e UNIVERSITY OF OTTAWA HEART INSTITUTE   (Canada)

^f PITIÉ SALPÊTRIÈRE HOSPITAL   (France)

^g SORBONNE UNIVERSITÉ   (France)

Author keywords

arrhythmia; atrial fibrillation; ion channels; KCNQ1; long QT syndrome; potassium

Indexed keywords

POTASSIUM CHANNEL; POTASSIUM CHANNEL KCNQ1;

ADOLESCENT; ADULT; ARTICLE; BRADYCARDIA; ELECTROPHYSIOLOGY; FAINTNESS; FEMALE; GENE SEQUENCE; GENETIC PREDISPOSITION; GENETIC SCREENING; GENETIC VARIABILITY; GENOTYPE; HEART ARREST; HEART ATRIUM FIBRILLATION; HEART MUSCLE EXCITABILITY; HEART MUSCLE POTENTIAL; HEART REPOLARIZATION; HUMAN; HUMAN CELL; LONG QT SYNDROME; MALE; MISSENSE MUTATION; MUTAGENESIS; ONSET AGE; PENETRANCE; PRIORITY JOURNAL; SUDDEN DEATH;

ADOLESCENT; ATRIAL FIBRILLATION; CELLS, CULTURED; FEMALE; GENOTYPE; HUMANS; KCNQ1 POTASSIUM CHANNEL; LONG QT SYNDROME; MALE; MUTATION, MISSENSE; PENETRANCE;

EID: 84876933197     PISSN: 10453873     EISSN: 15408167     Source Type: Journal    
DOI: 10.1111/jce.12068     Document Type: Article

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