SIRT3 protects cardiomyocytes from oxidative stress-mediated cell death by activating NF-κB

Biochemical and Biophysical Research Communications

Volumn 430, Issue 2, 2013, Pages 798-803

  Chen, Chun Juan ^a   Fu, Yu Cai ^b   Yu, Wei ^c   Wang, Wei ^c  

^a THE FIRST AFFILIATED HOSPITAL OF SHANTOU UNIVERSITY MEDICAL COLLEGE   (China)

^b SHANTOU UNIVERSITY MEDICAL COLLEGE   (China)

^c THE SECOND AFFILIATED HOSPITAL OF SHANTOU UNIVERSITY MEDICAL COLLEGE   (China)

Author keywords

Apoptosis; Cardiomyocytes; NF B; Oxidative stress; SIRT3

Indexed keywords

HYDROGEN PEROXIDE; I KAPPA B; NICOTINAMIDE; PROTEIN BAX; PROTEIN BCL 2; REACTIVE OXYGEN METABOLITE; RESVERATROL; SIRTUIN 3; SUPEROXIDE DISMUTASE;

ANIMAL EXPERIMENT; ANTIOXIDANT ACTIVITY; APOPTOSIS; ARTICLE; CELL DAMAGE; CELL DEATH; CELL PROTECTION; CELL SURVIVAL; CONTROLLED STUDY; DOWN REGULATION; EMBRYO; HEART MUSCLE CELL; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT;

ANIMALS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CELL LINE; GENE EXPRESSION REGULATION; GENES, BCL-2; HYDROGEN PEROXIDE; MYOCYTES, CARDIAC; NF-KAPPA B; NIACINAMIDE; OXIDATIVE STRESS; RATS; REACTIVE OXYGEN SPECIES; SIRTUIN 3; SUPEROXIDE DISMUTASE;

EID: 84872308934     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2012.11.066     Document Type: Article

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