Salivary gland hypofunction induced by activation of innate immunity is dependent on type I interferon signaling

Journal of Oral Pathology and Medicine

Volumn 42, Issue 1, 2013, Pages 66-72

  Nandula, Seshagiri Rao ^a,c   Dey, Paromita ^a   Corbin, Kathryn L ^a   Nunemaker, Craig S ^a   Bagavant, Harini ^a,b   Deshmukh, Umesh S ^a,b  

^a UNIVERSITY OF VIRGINIA   (United States)

^b UNIVERSITY OF VIRGINIA   (United States)

^c UNIVERSITY OF MINNESOTA MEDICAL SCHOOL   (United States)

Author keywords

Innate immunity; Interferon; Sjogren's syndrome; Xerostomia

Indexed keywords

ALPHA INTERFERON RECEPTOR; INTERFERON; INTERLEUKIN 10; INTERLEUKIN 27; INTERLEUKIN 6; PILOCARPINE;

ACINAR CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CALCIUM MOBILIZATION; CALCIUM TRANSPORT; CONTROLLED STUDY; CYTOKINE PRODUCTION; FEMALE; GENE EXPRESSION; INNATE IMMUNITY; MOUSE; NONHUMAN; PRIORITY JOURNAL; REAL TIME POLYMERASE CHAIN REACTION; SALIVARY GLAND DISEASE; SALIVARY GLAND FUNCTION; SIGNAL TRANSDUCTION; SUBMANDIBULAR GLAND; WILD TYPE;

ANIMALS; CALCIUM SIGNALING; FEMALE; IMMUNITY, INNATE; INJECTIONS, INTRAPERITONEAL; INTERFERON TYPE I; INTERLEUKIN-10; INTERLEUKIN-17; INTERLEUKIN-6; MICE; MICE, MUTANT STRAINS; POLY I-C; RECEPTOR, INTERFERON ALPHA-BETA; SALIVA; SUBMANDIBULAR GLAND; XEROSTOMIA;

EID: 84871934788     PISSN: 09042512     EISSN: 16000714     Source Type: Journal    
DOI: 10.1111/j.1600-0714.2012.01181.x     Document Type: Article

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