-
1
-
-
0038708366
-
The Fn14 cytoplasmic tail binds tumour-necrosis-factor-receptor-associated factors 1, 2, 3 and 5 and mediates nuclear factor-kappaB activation
-
Brown S.A., Richards C.M., et al. The Fn14 cytoplasmic tail binds tumour-necrosis-factor-receptor-associated factors 1, 2, 3 and 5 and mediates nuclear factor-kappaB activation. Biochem J 2003, 371(Pt 2):395-403.
-
(2003)
Biochem J
, vol.371
, Issue.PART 2
, pp. 395-403
-
-
Brown, S.A.1
Richards, C.M.2
-
2
-
-
0030006198
-
Signaling through the lymphotoxin beta receptor induces the death of some adenocarcinoma tumor lines
-
Browning J.L., Miatkowski K., et al. Signaling through the lymphotoxin beta receptor induces the death of some adenocarcinoma tumor lines. J Exp Med 1996, 183(3):867-878.
-
(1996)
J Exp Med
, vol.183
, Issue.3
, pp. 867-878
-
-
Browning, J.L.1
Miatkowski, K.2
-
3
-
-
0031465594
-
TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis
-
Chicheportiche Y., Bourdon P.R., et al. TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis. J Biol Chem 1997, 272(51):32401-32410.
-
(1997)
J Biol Chem
, vol.272
, Issue.51
, pp. 32401-32410
-
-
Chicheportiche, Y.1
Bourdon, P.R.2
-
4
-
-
74549220324
-
Antibodies to TWEAK receptor inhibit human tumor growth through dual mechanisms
-
Culp P.A., Choi D., et al. Antibodies to TWEAK receptor inhibit human tumor growth through dual mechanisms. Clin Cancer Res: An Offic J Am Assoc Cancer Res 2010, 16(2):497-508.
-
(2010)
Clin Cancer Res: An Offic J Am Assoc Cancer Res
, vol.16
, Issue.2
, pp. 497-508
-
-
Culp, P.A.1
Choi, D.2
-
5
-
-
56949089974
-
IFNgamma signaling-does it mean JAK-STAT?
-
Gough D.J., Levy D.E., et al. IFNgamma signaling-does it mean JAK-STAT?. Cytokine Growth Factor Rev 2008, 19(5-6):383-394.
-
(2008)
Cytokine Growth Factor Rev
, vol.19
, Issue.5-6
, pp. 383-394
-
-
Gough, D.J.1
Levy, D.E.2
-
6
-
-
0038242506
-
TNF-related weak inducer of apoptosis receptor, a TNF receptor superfamily member, activates NF-kappa B through TNF receptor-associated factors
-
Han S., Yoon K., et al. TNF-related weak inducer of apoptosis receptor, a TNF receptor superfamily member, activates NF-kappa B through TNF receptor-associated factors. Biochem Biophys Res Commun 2003, 305(4):789-796.
-
(2003)
Biochem Biophys Res Commun
, vol.305
, Issue.4
, pp. 789-796
-
-
Han, S.1
Yoon, K.2
-
7
-
-
0030917141
-
Jak3 is associated with CD40 and is critical for CD40 induction of gene expression in B cells
-
Hanissian S.H., Geha R.S. Jak3 is associated with CD40 and is critical for CD40 induction of gene expression in B cells. Immunity 1997, 6(4):379-387.
-
(1997)
Immunity
, vol.6
, Issue.4
, pp. 379-387
-
-
Hanissian, S.H.1
Geha, R.S.2
-
8
-
-
33750582348
-
Cytokine cooperation in renal tubular cell injury: the role of TWEAK
-
Justo P., Sanz A.B., et al. Cytokine cooperation in renal tubular cell injury: the role of TWEAK. Kidney Int 2006, 70(10):1750-1758.
-
(2006)
Kidney Int
, vol.70
, Issue.10
, pp. 1750-1758
-
-
Justo, P.1
Sanz, A.B.2
-
9
-
-
0032752353
-
The mitogen-inducible Fn14 gene encodes a type I transmembrane protein that modulates fibroblast adhesion and migration
-
Meighan-Mantha R.L., Hsu D.K., et al. The mitogen-inducible Fn14 gene encodes a type I transmembrane protein that modulates fibroblast adhesion and migration. J Biol Chem 1999, 274(46):33166-33176.
-
(1999)
J Biol Chem
, vol.274
, Issue.46
, pp. 33166-33176
-
-
Meighan-Mantha, R.L.1
Hsu, D.K.2
-
10
-
-
84863454553
-
The anti-Fn14 antibody BIIB036 inhibits tumor growth in xenografts and patient derived primary tumor models and enhances efficacy of chemotherapeutic agents in multiple xenograft models
-
Michaeslon J.S., Kelly R., et al. The anti-Fn14 antibody BIIB036 inhibits tumor growth in xenografts and patient derived primary tumor models and enhances efficacy of chemotherapeutic agents in multiple xenograft models. Cancer Biol Ther 2012, 13(9):812-821.
-
(2012)
Cancer Biol Ther
, vol.13
, Issue.9
, pp. 812-821
-
-
Michaeslon, J.S.1
Kelly, R.2
-
11
-
-
79960091768
-
Development of an Fn14 agonistic antibody as an anti-tumor agent
-
Michaelson J.S., Amatucci A., et al. Development of an Fn14 agonistic antibody as an anti-tumor agent. mAbs 2011, 3(4):362-375.
-
(2011)
mAbs
, vol.3
, Issue.4
, pp. 362-375
-
-
Michaelson, J.S.1
Amatucci, A.2
-
12
-
-
77649315474
-
Therapeutic targeting of TWEAK/Fnl4 in cancer: exploiting the intrinsic tumor cell killing capacity of the pathway
-
Michaelson J.S., Burkly L.C. Therapeutic targeting of TWEAK/Fnl4 in cancer: exploiting the intrinsic tumor cell killing capacity of the pathway. Results Problems Cell Differ 2009, 49:145-160.
-
(2009)
Results Problems Cell Differ
, vol.49
, pp. 145-160
-
-
Michaelson, J.S.1
Burkly, L.C.2
-
13
-
-
17844381082
-
Tweak induces mammary epithelial branching morphogenesis
-
Michaelson J.S., Cho S., et al. Tweak induces mammary epithelial branching morphogenesis. Oncogene 2005, 24(16):2613-2624.
-
(2005)
Oncogene
, vol.24
, Issue.16
, pp. 2613-2624
-
-
Michaelson, J.S.1
Cho, S.2
-
14
-
-
0037080352
-
Multiple pathways of TWEAK-induced cell death
-
Nakayama M., Ishidoh K., et al. Multiple pathways of TWEAK-induced cell death. J Immunol 2002, 168(2):734-743.
-
(2002)
J Immunol
, vol.168
, Issue.2
, pp. 734-743
-
-
Nakayama, M.1
Ishidoh, K.2
-
15
-
-
0030985893
-
Interferon-gamma modulates a p53-independent apoptotic pathway and apoptosis-related gene expression
-
Ossina N.K., Cannas A., et al. Interferon-gamma modulates a p53-independent apoptotic pathway and apoptosis-related gene expression. J Biol Chem 1997, 272(26):16351-16357.
-
(1997)
J Biol Chem
, vol.272
, Issue.26
, pp. 16351-16357
-
-
Ossina, N.K.1
Cannas, A.2
-
16
-
-
0025371205
-
Tumor necrosis factor up-regulates gamma-interferon binding in a human carcinoma cell line
-
Raitano A.B., Korc M. Tumor necrosis factor up-regulates gamma-interferon binding in a human carcinoma cell line. J Biol Chem 1990, 265(18):10466-10472.
-
(1990)
J Biol Chem
, vol.265
, Issue.18
, pp. 10466-10472
-
-
Raitano, A.B.1
Korc, M.2
-
17
-
-
34547108380
-
JAK-STAT signaling: from interferons to cytokines
-
Schindler C., Levy D.E., et al. JAK-STAT signaling: from interferons to cytokines. J Biol Chem 2007, 282(28):20059-20063.
-
(2007)
J Biol Chem
, vol.282
, Issue.28
, pp. 20059-20063
-
-
Schindler, C.1
Levy, D.E.2
-
18
-
-
33947717604
-
Interferon-regulatory-factor 1 controls toll-like receptor 9-mediated IFN-beta production in myeloid dendritic cells
-
Schmitz F., Heit A., et al. Interferon-regulatory-factor 1 controls toll-like receptor 9-mediated IFN-beta production in myeloid dendritic cells. Eur J Immunol 2007, 37(2):315-327.
-
(2007)
Eur J Immunol
, vol.37
, Issue.2
, pp. 315-327
-
-
Schmitz, F.1
Heit, A.2
-
19
-
-
0842266786
-
Interferon-gamma: an overview of signals, mechanisms and functions
-
Schroder K., Hertzog P.J., et al. Interferon-gamma: an overview of signals, mechanisms and functions. J Leukocyte Biol 2004, 75(2):163-189.
-
(2004)
J Leukocyte Biol
, vol.75
, Issue.2
, pp. 163-189
-
-
Schroder, K.1
Hertzog, P.J.2
-
20
-
-
0035918171
-
Interferon gamma (IFNgamma) and tumor necrosis factor alpha synergism in ME-180 cervical cancer cell apoptosis and necrosis. IFNgamma inhibits cytoprotective NF-kappa B through STAT1/IRF-1 pathways
-
Suk K., Chang I., et al. Interferon gamma (IFNgamma) and tumor necrosis factor alpha synergism in ME-180 cervical cancer cell apoptosis and necrosis. IFNgamma inhibits cytoprotective NF-kappa B through STAT1/IRF-1 pathways. J Biol Chem 2001, 276(16):13153-13159.
-
(2001)
J Biol Chem
, vol.276
, Issue.16
, pp. 13153-13159
-
-
Suk, K.1
Chang, I.2
-
21
-
-
47549097367
-
TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1-TRAF2 complex to sensitize tumor cells to TNFalpha
-
Vince J.E., Chau D., et al. TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1-TRAF2 complex to sensitize tumor cells to TNFalpha. J Cell Biol 2008, 182(1):171-184.
-
(2008)
J Cell Biol
, vol.182
, Issue.1
, pp. 171-184
-
-
Vince, J.E.1
Chau, D.2
-
22
-
-
18244380611
-
A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis
-
Wiley S.R., Cassiano L., et al. A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis. Immunity 2001, 15(5):837-846.
-
(2001)
Immunity
, vol.15
, Issue.5
, pp. 837-846
-
-
Wiley, S.R.1
Cassiano, L.2
-
23
-
-
0036953306
-
Death of HT29 adenocarcinoma cells induced by TNF family receptor activation is caspase-independent and displays features of both apoptosis and necrosis
-
Wilson C.A., Browning J.L. Death of HT29 adenocarcinoma cells induced by TNF family receptor activation is caspase-independent and displays features of both apoptosis and necrosis. Cell Death Differ 2002, 9(12):1321-1333.
-
(2002)
Cell Death Differ
, vol.9
, Issue.12
, pp. 1321-1333
-
-
Wilson, C.A.1
Browning, J.L.2
-
24
-
-
0031788449
-
CD40 is functionally expressed on human breast carcinomas: variable inducibility by cytokines and enhancement of Fas-mediated apoptosis
-
Wingett D.G., Vestal R.E., et al. CD40 is functionally expressed on human breast carcinomas: variable inducibility by cytokines and enhancement of Fas-mediated apoptosis. Breast Cancer Res Treat 1998, 50(1):27-36.
-
(1998)
Breast Cancer Res Treat
, vol.50
, Issue.1
, pp. 27-36
-
-
Wingett, D.G.1
Vestal, R.E.2
-
26
-
-
40949123747
-
TNF activates an IRF1-dependent autocrine loop leading to sustained expression of chemokines and STAT1-dependent type I interferon-response genes
-
Yarilina A., Park-Min K.H., et al. TNF activates an IRF1-dependent autocrine loop leading to sustained expression of chemokines and STAT1-dependent type I interferon-response genes. Nat Immunol 2008, 9(4):378-387.
-
(2008)
Nat Immunol
, vol.9
, Issue.4
, pp. 378-387
-
-
Yarilina, A.1
Park-Min, K.H.2
-
27
-
-
0024596722
-
A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor
-
Yonehara S., Ishii A., et al. A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor. J Exp Med 1989, 169(5):1747-1756.
-
(1989)
J Exp Med
, vol.169
, Issue.5
, pp. 1747-1756
-
-
Yonehara, S.1
Ishii, A.2
|