High-dose sodium selenite toxicity cannot be prevented by the co-administration of pharmacological levels of epigallocatechin-3-gallate which in turn aggravates the toxicity

Food and Chemical Toxicology

Volumn 52, Issue , 2013, Pages 36-41

  Sun, Kang ^a   Wu, Shanshan ^a   Wang, Yijun ^a   Wan, Xiaochun ^a   Thompson, Henry J ^b   Zhang, Jinsong ^a  

^a ANHUI AGRICULTURAL UNIVERSITY   (China)

^b Department of Environmental and Radiological Health Sciences   (United States)

Author keywords

Epigallocatechin 3 gallate; Lethality; Sodium selenite; Toxicity

Indexed keywords

EPIGALLOCATECHIN GALLATE; GLUTATHIONE TRANSFERASE; SODIUM SELENITE; SUPEROXIDE DISMUTASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CAUSE OF DEATH; CONTROLLED STUDY; DISEASE EXACERBATION; DOSE TIME EFFECT RELATION; DRUG EFFECT; DRUG MEGADOSE; ENZYME ACTIVITY; GROWTH INHIBITION; LETHALITY; MALE; MOUSE; NONHUMAN; OXIDATIVE STRESS; SELENIUM TOXICITY; SINGLE DRUG DOSE; SURVIVAL TIME; TOXICITY; TREATMENT DURATION;

ANIMALS; CATECHIN; DOSE-RESPONSE RELATIONSHIP, DRUG; DRUG TOXICITY; GLUTATHIONE TRANSFERASE; GROWTH DISORDERS; LIVER; MALE; MICE; OXIDATIVE STRESS; PROTECTIVE AGENTS; SELENIUM; SODIUM SELENITE; TISSUE DISTRIBUTION;

ANIMALIA; MUS;

EID: 84869870686     PISSN: 02786915     EISSN: 18736351     Source Type: Journal    
DOI: 10.1016/j.fct.2012.10.038     Document Type: Article

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