Postnatal Ablation of Foxm1 from Cardiomyocytes Causes Late Onset Cardiac Hypertrophy and Fibrosis without Exacerbating Pressure Overload-Induced Cardiac Remodeling

PLoS ONE

Volumn 7, Issue 11, 2012, Pages

  Bolte, Craig ^a   Zhang, Yufang ^a   York, Allen ^a   Kalin, Tanya V ^a   Schultz, Jo El J ^b   Molkentin, Jeffery D ^a   Kalinichenko, Vladimir V ^a  

^a CINCINNATI CHILDREN S HOSPITAL RESEARCH FOUNDATION   (United States)

^b UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA SMOOTH MUSCLE ACTIN; CRE RECOMBINASE; FIBRONECTIN; FORKHEAD BOX M1 TRANSCRIPTION FACTOR; FORKHEAD TRANSCRIPTION FACTOR; GELATINASE B; UNCLASSIFIED DRUG; VIMENTIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CARDIAC REMODELING; CELL PROLIFERATION; CONTROLLED STUDY; DISEASE ASSOCIATION; GENE EXPRESSION; HEART DEVELOPMENT; HEART DISEASE; HEART FUNCTION; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE OVERLOAD; MOLECULAR PATHOLOGY; MOUSE; NONHUMAN; POSTNATAL DEVELOPMENT; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN HOMEOSTASIS; PROTEIN LOCALIZATION;

ACTINS; AGE FACTORS; ANIMALS; BASIC HELIX-LOOP-HELIX TRANSCRIPTION FACTORS; CARDIOMEGALY; CELL PROLIFERATION; FIBRONECTINS; FIBROSIS; FORKHEAD TRANSCRIPTION FACTORS; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HEART; MATRIX METALLOPROTEINASE 9; MICE; MICE, TRANSGENIC; MYOCARDIUM; MYOCYTES, CARDIAC; REPRESSOR PROTEINS; VIMENTIN;

MUS;

EID: 84869056795     PISSN: None     EISSN: 19326203     Source Type: Journal    
DOI: 10.1371/journal.pone.0048713     Document Type: Article

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